Caseous necrosis
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cheese-like
fragmented or lysed cells seen
No visible tissue architecture
Common in granulomas (TB)
Describe mechanisms the phagocytic cell has for dealing with ingested material
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, It fuses with lysosome and degrades material with lysosomal enzymes and
ROS in the phagolysosome (neutrophils and macrophages)
NETs
Define pathogenesis and be able to use when discussing the cause of disease and
response of organism to disease
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Development/ MECHANISM of disease
List and describe the two main components of inflammation
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Vascular changes: Increased vasodilation- increased blood flow to area
leading to warmth and redness. Increased vascular permeability,
increased exudation, release of cells and proteins to surrounding area
leading to pain and swelling.
Cellular participation: WBC mainly Neutrophils (PMNs) at site first.
Macrophages arrive later, more chemical mediators involved
Cell Injury: Genetic Problems
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Congenital or later mutations
Possible causes:
, Inherited defects
Accumulation of damaged DNA or proteins (proteins are made improperly)
List and describe the function of important cytokines in inflammation
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Proteins
Describe NET
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Neutrophil extracellular traps-
List the major cells involved in inflammation and describe characteristics and
functions in the inflammatory process. State the timing of their involvement
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Leukocytes:
Neutrophils:
Monocytes/macrophages:
Describe ways normal cells or tissues can adapt to stress and the signals that induce
them: metaplasia
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Give this one a try later!
cheese-like
fragmented or lysed cells seen
No visible tissue architecture
Common in granulomas (TB)
Describe mechanisms the phagocytic cell has for dealing with ingested material
Give this one a try later!
, It fuses with lysosome and degrades material with lysosomal enzymes and
ROS in the phagolysosome (neutrophils and macrophages)
NETs
Define pathogenesis and be able to use when discussing the cause of disease and
response of organism to disease
Give this one a try later!
Development/ MECHANISM of disease
List and describe the two main components of inflammation
Give this one a try later!
Vascular changes: Increased vasodilation- increased blood flow to area
leading to warmth and redness. Increased vascular permeability,
increased exudation, release of cells and proteins to surrounding area
leading to pain and swelling.
Cellular participation: WBC mainly Neutrophils (PMNs) at site first.
Macrophages arrive later, more chemical mediators involved
Cell Injury: Genetic Problems
Give this one a try later!
Congenital or later mutations
Possible causes:
, Inherited defects
Accumulation of damaged DNA or proteins (proteins are made improperly)
List and describe the function of important cytokines in inflammation
Give this one a try later!
Proteins
Describe NET
Give this one a try later!
Neutrophil extracellular traps-
List the major cells involved in inflammation and describe characteristics and
functions in the inflammatory process. State the timing of their involvement
Give this one a try later!
Leukocytes:
Neutrophils:
Monocytes/macrophages:
Describe ways normal cells or tissues can adapt to stress and the signals that induce
them: metaplasia
Give this one a try later!
