MSCP Exam – Questions With Fully Explained
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Terms in this set (65)
What does long life increase risk of? osteoporosis, cardiovascular disease, cognitive
decline
What % of women live past age 80? 60%
leading cause of death for women cardiovascular disease (1 in 5)
, what physiologic change initiates the ovarian follicular depletion - (ovaries are reaching
menopause transition in the their retirement, low eggs causing low estrogen
hypothalamic-pituitary-ovarian axis which then causes less LH secretion)
physiology of the menopause DECREASED sensitivity to gonadotropins causes
transition decreases estradiol/HP feedback, increasedFSH and
LH as compensation, causing common anovulatory
cycles and erratic estrogen output
hypothalamic pituitary ADRENAL axis adrenals are back up for HPO axis - increased DHEA,
in the menopause transition androstenedione, testosterone and cortisol. cortisol
rise associated w mood, sleep,VMS.
when do adrenal androgens return to 1-2 years post final menstrual period (this is why
premenopausal levels in perimenopause can be so difficult and cause
perimenopause/menopause transition heterogenous symptoms)
what is the relationship between LH LH may stimulate adrenal cortisol and DHEAS
and adrenal hormone secretion in secretion
midlife women?
what hormones does the adrenal cortisol (glucocorticoid), androstenedione
cortex produce? (mineralcorticoid), androgens (DHEA, DHEAS,
testosterone)
what hormones does the adrenal catecholamines (epinephrine and norepinephrine)
medulla produce
DHEA-S and the menopause transition DHEA-S decline with age demonstrated by SWAN
study
What study looked at DHEA-S in the SWAN study
menopause transition?
Solutions
Save Groups
Play your way to mastery with fun games
Blast
Terms in this set (65)
What does long life increase risk of? osteoporosis, cardiovascular disease, cognitive
decline
What % of women live past age 80? 60%
leading cause of death for women cardiovascular disease (1 in 5)
, what physiologic change initiates the ovarian follicular depletion - (ovaries are reaching
menopause transition in the their retirement, low eggs causing low estrogen
hypothalamic-pituitary-ovarian axis which then causes less LH secretion)
physiology of the menopause DECREASED sensitivity to gonadotropins causes
transition decreases estradiol/HP feedback, increasedFSH and
LH as compensation, causing common anovulatory
cycles and erratic estrogen output
hypothalamic pituitary ADRENAL axis adrenals are back up for HPO axis - increased DHEA,
in the menopause transition androstenedione, testosterone and cortisol. cortisol
rise associated w mood, sleep,VMS.
when do adrenal androgens return to 1-2 years post final menstrual period (this is why
premenopausal levels in perimenopause can be so difficult and cause
perimenopause/menopause transition heterogenous symptoms)
what is the relationship between LH LH may stimulate adrenal cortisol and DHEAS
and adrenal hormone secretion in secretion
midlife women?
what hormones does the adrenal cortisol (glucocorticoid), androstenedione
cortex produce? (mineralcorticoid), androgens (DHEA, DHEAS,
testosterone)
what hormones does the adrenal catecholamines (epinephrine and norepinephrine)
medulla produce
DHEA-S and the menopause transition DHEA-S decline with age demonstrated by SWAN
study
What study looked at DHEA-S in the SWAN study
menopause transition?
