fpgee review questions with 100% correct answers

fpgee review questions with 100%
correct answers
InAFASTAresponseAfibersA-AElectrolyteAinvolvedAinAphaseA0A-
AAnswerANa+Ainflux


InAFASTAresponseAfibersA-AElectrolyteAinvolvedAinAphaseA2A-
AAnswerACa+Ainflux,AK+AoutfluxAplateau


InAFASTAresponseAfibersA-AElectrolyteAinvolvedAinAphaseA3A-
AAnswerAK+AoutfluxA(Ca+AdropsAoff)ArapidArepolarization


InASLOWAresponseAfibersA-AElectrolyteAinvolvedAinAphaseA0A-
AAnswerACa+(fewANa+AchannelsAinANodalAfibers)


InASLOWAresponseAfibersA-AElectrolyteAinvolvedAinAphaseA3A-
AAnswerAK+AoutfluxArepolarization


InASLOWAresponseAfibersA-AElectrolyteAinvolvedAinAphaseA4A-
AAnswerA"MainlyANa+A(ThisAisAtheApacemakerAcurrent)Aaarapim.A"


EctopicArhythmAcanAresultAfromA-
AAnswerAischemia,AelectrolyteAabnormalities,AorAheightenedAsympatheticAtone


IfAanAearlyAafterdepolarizationAisAsustainedA-
AAnswerAtoradesAdeApointesA(QRSAcomplexesAofAvaryingAamplitudes)


ECGAofAaApatientAwithAanAaccessoryApathwayA(bundleAofAKent)A-
AAnswerA"WideAQRSAcomplexes,AearlyAventricularAupstrokeA(alsoApredisposedAt
oAre-entryAloops)A(WolffAParkinsonAWhiteA(WPW)Asyndrome)Awolff-parkinson-
white-accessory-bundle-bundle-of-KentA"

,MOAAofAquinidineA-AAnswerABlocksA(open-
activated)ANa+Achannels:AInASAANodalAtissueA-
AShiftAtoAaAmoreApositiveAthresholdAandAdecreaseAtheApacemakerAslopeA(phase
A4),AInAVentricularAtissueA-
ADecreasesAtheAphaseA0AslopeA(depolarization)AandAblockAK+AchannelsAprolon
gsArepolarizationA(longerArefractoryAperiodAprotectedAfromAreentryAcurrents):AA
nticholanergicA(vagolyticAeffectAfasterAconductionAthroughAAVAnode)


IndicationsAofAquinidineA-
AAnswerA"atrialAflutterAorAfibrillationAmaintainAnormalAsinusArhythm,Paroxysm
alAsupraventricularAtachycardia,APrematureAatrialAorAventricularAcontractions,Ap
aroxysmalAAVAjunctionalArhythmA9"


WhenAtakingAthisAdrugApatientsAmustAbeAmonitoredAforAlupusA(withAANA)A-
AAnswerAProcainamide


TakeAwithAquinidineAtoApreventArapidAventricularAresponseAinApatientAwithAatr
ialAflutterA-AAnswerAβ-blockerAorACa2+AchannelAblocker


WhichA1AAantiarrhythmicAforAaApatientAonAdigoxinA-
AAnswerAProcainamideA(doesn'tAalterAdigoxinAlevels)


MOAAofALidocaineAandAPhenytoinA-AAnswerA(A1B)AblockAofAvoltage-
gatedANa+AinAventricularAmyocytesA(decreaseAphaseA0Aupstroke)


MOAAofAEncainide,AFlecainide,AMoricizine,APropafenoneA-
AAnswerA(A1C)AblockAofAvoltage-gatedANa+AchannelsinAventricularAmyocytes


MOAAofAPropranolol,AAtenolol,AMetoprolol...A-AAnswerA(A)AblockAβ1-
adrenergicAreceptorsASLOWASAAandAAVAnodalAactivity,(decreasesAphaseA4Aslope
)

,MOAAofAIbutilide,ADofetilide,ASotalol,AAmiodaroneA-
AAnswerA(AI)ABlockAK+AchannelsAlongerAactionApotentialAplateauAandAprolonge
dArepolarization


MOAAofAVerapamil,ADiltiazemA-
AAnswerA(AIV)ACa2+AchannelAblockerAslowsAactionApotentialAupstrokeAinASAAa
ndAAVAnodes


ThisA1AAdrugAisAassociatedAwithAtheAdevelopmentAofALupus,Ahematotoxicity,Ab
utAlessAlikelyAtoAcauseAtorsadesA-AAnswerAprocainamide


AntiarrhythmicsAofAchoiceAforApostAischemicAinsultsA-AAnswerA1B


DrugAusedAforASupraventricularAtachycardias,AandApost-MIAprophylaxisA-
AAnswerAantiarrhythmicsA(β-blockers)


WhatAdoesAQuinidineAdoAtoAAVAnodeA-
AAnswerAincreasesAconductionAvelocityA(viaAanticholinergicAeffects)


ThisA1AAdrugAisAmeolizedAtoAaAmeoliteAwithAaApureAIAeffectA(prolongingArefra
ctory,AlengtheningAQTAinterval)A-AAnswerAProcainamide


DOCAorAventricularAarrhythmiasAinAERAsituationsA(likeAinAMI)A-
AAnswerALidocaine


LidocaineMexiletineAshouldAbeA(increasedAorAdecreased)AinApatientsAwithAdrugA
inducibleAP0A-AAnswerAincreased


ModerateANa+AchannelAblockAdecreaseAphaseA0,AandAprolongedArepolarization
A(A1A,A1BAorA1C)A-AAnswerA1A

, MildANa+AchannelAblockAslightApahseA0AslopeAdecrease,AshortenedArepolarizat
ion(A1A,A1BAorA1C)A-AAnswerA1B


MarkedANa+AchannelAblockAsevereAdecreasedAinAphaseA0Aslope,ANoAchangeAi
nArepolarizationA(A1A,A1BAorA1C)A-AAnswerA1C


ThisAdrugAcanAworsenAtheAarrhythmiaAinApatientsAwithApre-
existingAventricularAtachyarrythmiasAandAthoseAwithAaAhistoryAofAMIA-
AAnswerAFlecainideA(aA1C)


DrugAusedAwhenAotherAmeasureAfailAtoAhelpAlifeAthreateningAparoxysmalAsupr
aventricularAorAvventricularAarrhythmiasA-AAnswerAFlecainideA(aA1C)


MOAAofAβ1-blockersA-
AAnswerAblockAsympatheticAinputAtoAAVA(andASAAnodes)AdecreaseAtheArateAof
AphaseA4AdepolarizationA(pacemakerAcurrent),AprolongArepolarization


Non-selectiveAβ1AandAβ2AblockersAthatADOANOTAprolongArepolarizationA-
AAnswerAFirstAgenerationAβ-
blockersA(Propranolol,ANadolol,ATimolol)AandAcarvedilolAandAlabetalol


Β-blockerAalsoAusedAforAglaucomaA-AAnswerATimolol


ThisAβ-blockerAcanAcauseAliverAdamageA-AAnswerAlabetalol


TheseAβ-blockersAcauseAvasodilationviaAα1-blockageA-
AAnswerACarvedilolAandAlabetalol


SelectiveAβ1-blockerAwithAaAveryAshortAhalf-lifeA-AAnswerAEsmolol


UsedAinAERAtreatmentAtoAblockAβAinAthyroidAstormA-AAnswerAesmolol