w = e e . A wv-, - R —— W‘m’”flfl?wfi*ww
ROOOOORORO0NRRSRRSARRRRNS.
26
Week 2: Glucose Regulation
non; Chapler 50; Silvestritri 590-
Weekly Reading: Giddens: Chapter 15; Lin& tMao
ttes
597; 609-613; Willihnganz/Clayton: 561-575; 579-582; 586-593
Metabolic Syndrome (Linton & Matteson p. 1001, Silvestri p 590)
Define Metabolic syndrome: o Qronip of cpnddAons MIAMMW %M’
Patients with metabolic syndrome often have
* Impaired _{gsng amoose
¢ High serum fl‘@)—wf exides
* Wicreased
walst pixeumfrence
e Low HODL cholesterol
* Elevated B9p
Metabolic syndrome represents a Chroniv inflammatory process affecting
endothelial tissues.
and metabolic: syndrome: 2
What lifestyle changes are recommended for managing prediabetes
eu SAVAYY, 1 coot o balaveed duet, exercige
M
r btvod
ot deosk Mwwné/ otau,. AOAD SMOV—M. Redune gtress, Movito
Yoot regrlonly,
0000000000
of symptoms, lab values, and treatment
How is prediabetes different from type 2 diabetes in terms
\
Boals: (testring glucose)
thAMv\té O‘me 0 Symptoms, 61007125 Alc -- 58 -bH2%, o
oau/l e Aovped WIM,%NYW amdl WW)HLOS‘S VS Ty dlpdoctes
2 pe
cumiptots P, Fo= 2120, Ale= = b.5%, cannet e stopped, JUsT
P\r'eve‘r\-r further c,ompwccum\s.
0 R0
© Hondros College of Nursing FALL25 Initials and Date:% 10=7-25
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PP
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|
(
{
27
-.4
B
/
D " 3¢3 Process Map
R
Diseas;
A A AN AR R R R R R PR R RN
T
°ase Name: Diabetes mellitus type 2~
e R
g
S I
A
ee el
What am | going to see duringm
¢ \
{ What is h 2T
o s happening in the body? \ [ ass:ss?nent? -(-wfiw,,,wca,&cfuéc.’ p
%’S':Lln veastame,lea,(” to | ) PONUYiO.wlqd}pfiaaPO\thMla.
lflvnolm it FWaoge ; plurved vistan, reourvert infections,
e Cauigeg Cellwla v ' slow wound healing , weight Gain . |
S"‘W\/qj—‘\bn
"""""" l What tests and labs will be
What trends and findings are expected
? ordered?
Wiih bluod olucose Fusting blood glucose
/kulo levele = 12 mg|dL
Wish fiiWouldcs ,
Hypertensipn i Low HOL, lnigh LpL HbAle> 115%
O6TT =200 mgldL@2h
51'«1‘\1,
eandom bloed glucose
urivialysis
$
i Lipid panel
What medications and nursing interventions/treatments will you anticipate?
? & How will you know they are
[ 1 . g improving?
Ry MMMM“PV Bl:nu;d%juwsie/kl(/@%u 1
Moty bioodl e ' v
0aGlUCose
duiek (XENLLEE) | cducate ned,
teach s o4 Wpo/ns ndlovence, | deereased symptoms,
. ' iStable weigiht
S
inspect feet cadly, s < o
POl peratycevnio,
; - Normal wound healing
o o®
What are the long-term complications?
Z8
What are the risk factors for diagnosis?
Nerve Cl(:lmaqc, Cidney £ ilure , Vision loss,
A A
O\oefifus,, %C\-&n“mrla t(@eg%
Poor cureulation | infection.
E wndy He ) Sucyle T8p,
by pe dipidemia, gestatio nal dicdrief‘ts.
d
L edhnic backyvound
K
0
A
FALL25 Initials and Date:%
© Hondros College of Nursing \0~7-29
U
A
ROOOOORORO0NRRSRRSARRRRNS.
26
Week 2: Glucose Regulation
non; Chapler 50; Silvestritri 590-
Weekly Reading: Giddens: Chapter 15; Lin& tMao
ttes
597; 609-613; Willihnganz/Clayton: 561-575; 579-582; 586-593
Metabolic Syndrome (Linton & Matteson p. 1001, Silvestri p 590)
Define Metabolic syndrome: o Qronip of cpnddAons MIAMMW %M’
Patients with metabolic syndrome often have
* Impaired _{gsng amoose
¢ High serum fl‘@)—wf exides
* Wicreased
walst pixeumfrence
e Low HODL cholesterol
* Elevated B9p
Metabolic syndrome represents a Chroniv inflammatory process affecting
endothelial tissues.
and metabolic: syndrome: 2
What lifestyle changes are recommended for managing prediabetes
eu SAVAYY, 1 coot o balaveed duet, exercige
M
r btvod
ot deosk Mwwné/ otau,. AOAD SMOV—M. Redune gtress, Movito
Yoot regrlonly,
0000000000
of symptoms, lab values, and treatment
How is prediabetes different from type 2 diabetes in terms
\
Boals: (testring glucose)
thAMv\té O‘me 0 Symptoms, 61007125 Alc -- 58 -bH2%, o
oau/l e Aovped WIM,%NYW amdl WW)HLOS‘S VS Ty dlpdoctes
2 pe
cumiptots P, Fo= 2120, Ale= = b.5%, cannet e stopped, JUsT
P\r'eve‘r\-r further c,ompwccum\s.
0 R0
© Hondros College of Nursing FALL25 Initials and Date:% 10=7-25
@)
PP
,U T TS s WY S N D —
|
(
{
27
-.4
B
/
D " 3¢3 Process Map
R
Diseas;
A A AN AR R R R R R PR R RN
T
°ase Name: Diabetes mellitus type 2~
e R
g
S I
A
ee el
What am | going to see duringm
¢ \
{ What is h 2T
o s happening in the body? \ [ ass:ss?nent? -(-wfiw,,,wca,&cfuéc.’ p
%’S':Lln veastame,lea,(” to | ) PONUYiO.wlqd}pfiaaPO\thMla.
lflvnolm it FWaoge ; plurved vistan, reourvert infections,
e Cauigeg Cellwla v ' slow wound healing , weight Gain . |
S"‘W\/qj—‘\bn
"""""" l What tests and labs will be
What trends and findings are expected
? ordered?
Wiih bluod olucose Fusting blood glucose
/kulo levele = 12 mg|dL
Wish fiiWouldcs ,
Hypertensipn i Low HOL, lnigh LpL HbAle> 115%
O6TT =200 mgldL@2h
51'«1‘\1,
eandom bloed glucose
urivialysis
$
i Lipid panel
What medications and nursing interventions/treatments will you anticipate?
? & How will you know they are
[ 1 . g improving?
Ry MMMM“PV Bl:nu;d%juwsie/kl(/@%u 1
Moty bioodl e ' v
0aGlUCose
duiek (XENLLEE) | cducate ned,
teach s o4 Wpo/ns ndlovence, | deereased symptoms,
. ' iStable weigiht
S
inspect feet cadly, s < o
POl peratycevnio,
; - Normal wound healing
o o®
What are the long-term complications?
Z8
What are the risk factors for diagnosis?
Nerve Cl(:lmaqc, Cidney £ ilure , Vision loss,
A A
O\oefifus,, %C\-&n“mrla t(@eg%
Poor cureulation | infection.
E wndy He ) Sucyle T8p,
by pe dipidemia, gestatio nal dicdrief‘ts.
d
L edhnic backyvound
K
0
A
FALL25 Initials and Date:%
© Hondros College of Nursing \0~7-29
U
A
