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ADVANCED PHARMACOLOGY MIDTERM EXAM NEWEST ACTUAL EXAM COMPLETE 180 QUESTIONS AND CORRECT DETAILED ANSWERS (VERIFIED ANSWERS) |ALREADY GRADED A+||BRAND NEW!!(2025)

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ADVANCED PHARMACOLOGY MIDTERM EXAM NEWEST ACTUAL EXAM COMPLETE 180 QUESTIONS AND CORRECT DETAILED ANSWERS (VERIFIED ANSWERS) |ALREADY GRADED A+||BRAND NEW!!(2025)

Institution
Advanced Pharmacology
Module
Advanced pharmacology

Content preview

ADVANCED PHARMACOLOGY MIDTERM EXAM NEWEST ACTUAL

EXAM COMPLETE 180 QUESTIONS AND CORRECT DETAILED

ANSWERS (VERIFIED ANSWERS) |ALREADY GRADED A+||BRAND

NEW!!(2025)



Lispro - (answers)Class: Rapid acting insulin for hyperglycemia

MOA: Onset 5 minutes; peaks 5 minutes; duration 4-5 hours



Aspart - (answers)Class: Rapid acting insulin for hyperglycemia

MOA: Onset 5 minutes; peaks 5 minutes; duration 4-5 hours



Regular - (answers)Class: Short acting insulin for hyperglycemia

MOA: Onset 30-45 minutes; peaks 3-4 hours; duration 4-10 hours



NPH - (answers)Class: Intermediate insulin for hyperglycemia

MOA: Onset 30 minutes-1 hour; peaks 4-10 hours; duration 12-24 hours



Glargine - (answers)Class: Long acting insulin for hyperglycemia

MOA: Onset 2-4 hours; does not have a peak, duration 24 hours



Detemir - (answers)Class: Long acting insulin for hyperglycemia

MOA: Onset 2-4 hours; does not have a peak, duration 24 hours



Glipizide - (answers)Class: Short acting Sulfonylurea for an insufficient production of insulin in DM2

MOA: Stimulates insulin release form beta cells, all potentiate effects of ADH.



Glyburide - (answers)Class: Sulfonylurea for an insufficient production of insulin in DM2

MOA: Stimulates insulin release form beta cells, all potentiate effects of ADH.

, ADVANCED PHARMACOLOGY MIDTERM EXAM NEWEST ACTUAL

EXAM COMPLETE 180 QUESTIONS AND CORRECT DETAILED

ANSWERS (VERIFIED ANSWERS) |ALREADY GRADED A+||BRAND

NEW!!(2025)




Chlorpropamide - (answers)Class: Sulfonylurea for an insufficient production of insulin in DM2; off label
for neurogenic DI

MOA: Stimulates insulin release form beta cells, all potentiate effects of ADH.



Metformin - (answers)Class: Biguanide, first line drug for DM2

MOA: Decreases glucose production in liver, decreases GI glucose absorption and improves insulin
sensitivity by increasing peripheral glucose uptake and utilization; does not stimulate insulin release for
beta cells; inhibits platelet aggregation and reduces blood viscosity.



Acarbose - (answers)Class: Alpha Glucosidase Inhibitor; third-line for DM2 in insulin resistant patients

MOA: Inhibits absorption of CHO in GI, lowers BG levels after meals



Miglitol - (answers)Class: Alpha Glucosidase Inhibitor; third-line for DM2 in insulin resistant patients

MOA: Inhibits absorption of CHO in GI, lowers BG levels after meals



Sitagliptin - (answers)Class: Dipeptidyl Peptidase 4 Inhibitor for DM2

MOA: Inhibits DPP4, breaks down GLP1 and GIP which are released in response to a meal; leads to
increase in secretion of insulin and suppresses the release of glucagon by the pancreas; promotes pre
and postprandial glucose levels.



Saxaglipin - (answers)Class: Dipeptidyl Peptidase 4 Inhibitor for DM2

MOA: Inhibits DPP4, breaks down GLP1 and GIP which are released in response to a meal; leads to
increase in secretion of insulin and suppresses the release of glucagon by the pancreas; promotes pre
and postprandial glucose levels.

, ADVANCED PHARMACOLOGY MIDTERM EXAM NEWEST ACTUAL

EXAM COMPLETE 180 QUESTIONS AND CORRECT DETAILED

ANSWERS (VERIFIED ANSWERS) |ALREADY GRADED A+||BRAND

NEW!!(2025)



Exenatide - (answers)Class: Glucagon Like Peptide 1 Agonist for DM2

MOA: Promotes insulin release from pancreatic beta cells in the presence of elevated glucose
concentrations.



Glucagon - (answers)Class: Antidote to hypoglycemia or insulin overdose

MOA: Stimulates hepatic gluconeogenesis and glycogenolysis, raised BG levels; BG concentration rises
within 10 minutes of injection and maximal concentrations are attained at approximately 30 minutes
after injection.



Levothyroxine - (answers)Class: T4 thyroid hormone for hypothyroidism; first line drug of choice for
thyroid replacement and suppression therapy due to longer half-life

MOA: Produce the same effects in the body as endogenous thyroid hormones.



Liothyronine - (answers)Class: T3 thyroid hormone for hypothyroidism

MOA: Produce the same effects in the body as endogenous thyroid hormones.



Liotrix - (answers)Class: Mix ratio (4:1) T4:T3 thyroid hormone for hypothyroidism

MOA: Produce the same effects in the body as endogenous thyroid hormones.



Propylthiouracil - (answers)Class: Antithyroid agent

MOA: Blocks synthesis of thyroxine and triiodothyronine.



Methimazole - (answers)Class: Antithyroid agent; first line drug

MOA: Blocks synthesis of thyroxine and triiodothyronine.

, ADVANCED PHARMACOLOGY MIDTERM EXAM NEWEST ACTUAL

EXAM COMPLETE 180 QUESTIONS AND CORRECT DETAILED

ANSWERS (VERIFIED ANSWERS) |ALREADY GRADED A+||BRAND

NEW!!(2025)



What is the first line drug for a patient with diabetes? - (answers)Metformin, a biguanide



What is the first line drug for a patient with hypothyroidism? - (answers)Levothyroxine, a thyroid
hormone



What is the first line drug for a patient with hyperthryoidism? - (answers)Methimazole, an antithyroid
agent



What is the first line drug for a pregnant patient with diabetes? - (answers)Insulins that are rapid or short
acting that are hum- derivatives.



What should patients HbA1C levels be below? - (answers)6.5%



Alendronate - (answers)Class: Bisphosphanate used to treat osteoporosis and Paget's dx

MOA: Inhibits osteoclastic activity, increases bone density.



Tiludronate - (answers)Class: Bisphosphanate used to treat osteoporosis and Paget's dx

MOA: Inhibits osteoclastic activity, increases bone density.



T or F: Amitriptylline will increase the concentration of norepinephrine in the synapse - (answers)True



T or F: Haloperidol is a high potency antipsychotic which blocks dopamine receptors? - (answers)True



T or F: Glipizide inhibits the dipeptidyl peptidase 4 enzyme? - (answers)False

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Institution
Advanced pharmacology
Module
Advanced pharmacology

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