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NR507/ NR 507 Midterm Exam (Latest 2025/ 2026 Update) Advanced Pathophysiology Study Guide with Complete Solutions | 100% Correct- Chamberlain.

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Most diagnosed interstitial lung disease -radiation pnemonitis -pneumonconioses -hypersensitivity pneumonitis Microcytic-hypochromic iron deficiency sideroblastic thalassemia Microcytic normochromic Anemia of inflammation and chronic disease Microcytic hyperchromic Spherocytosis Hypertension directly effects which load? Afterload Which condition can lower preload? Hemorrhage left side of heart Aorta, pulmonary vein, left atrium, mitral valve, aortic valve, left ventricle Right sided of the heart: Super vena cava (preload), pulmonary artery, right atrium, pulmonary valve, tricuspid valve, inferior vena cava, right ventricle Hypersensitivity: Type 1 -Mediated IgE -inflammation due to mast cell degranultion -anaphylactic, asthma, hay fever treatment: epinephrine Hypersensitivity: Type 2 -Cytotoxic reaction: tissue specific -binds to the antigen ON THE CELL SURFACE -macrophages are primary effectors cells involved cause tissue damage or alter function Hypersensitivity 2 -Graves Disease (hyperthyroidism) -alters function not destroy -blood transfusions reaction- transfused erythrocytes are destroyed by agglutination or lysis -drug allergies -hemolytic anemia Hypersensitivity: type 3 -Not organ specific -antibody binds to soluble antigen OUTSIDE THE CELL SURFACE that was released into the blood or bodily fluids, and the complex is then deposited in the tissues Hypersensitivity: Type 3 -rheumatoid arthritis -Systemic Lupus Erythematosus (SLE) -Raynaud's Hives (urticaria) Hypersensitivity Type 1 NR507/ NR 507 Midterm Exam (Latest 2025/ 2026 Update) Advanced Pathophysiology Study Guide with Complete Solutions | 100% Correct- Chamberlain.

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NR507/ NR 507 Midterm Exam (Latest 2025/
i,- i,- i,- i,- i,- i,- i,-




2026 Update) Advanced Pathophysiology
i,- i,- i,- i,-




Study Guide with Complete Solutions | 100%
i,- i,- i,- i,- i,- i,- i,-




Correct- Chamberlain. i,-




Most diagnosed interstitial lung disease
i,- i,- i,- i,- i,-i,- i,- -radiation pnemonitis
i,-




-pneumonconioses i,-




-hypersensitivity pneumonitis i,-




Microcytic-hypochromic i,-i,- i,- iron deficiencyi,-




sideroblastic
thalassemia


Microcytic normochromic i,- i,-i,- i,- Anemia of inflammation and i,- i,- i,- i,-



chronic disease i,-




Microcytic hyperchromic i,- i,-i,- i,- Spherocytosis


Hypertension directly effects which load? i,- i,- i,- i,- i,-i,- i,- Afterload


Which condition can lower preload?
i,- i,- i,- i,- i,-i,- i,- Hemorrhage

,left side of heart
i,- Aorta, pulmonary vein, left atrium, mitral
i,- i,- i,-i,- i,- i,- i,- i,- i,- i,- i,-



valve, aortic valve, left ventricle
i,- i,- i,- i,-




Right sided of the heart:
i,- Super vena cava (preload),
i,- i,- i,- i,-i,- i,- i,- i,- i,- i,-



pulmonary artery, right atrium, pulmonary valve, tricuspid valve,
i,- i,- i,- i,- i,- i,- i,- i,-



inferior vena cava, right ventricle
i,- i,- i,- i,-




Hypersensitivity: Type 1 i,- i,- i,-i,- i,- -Mediated IgE i,-




-inflammation due to mast cell degranultion i,- i,- i,- i,- i,- i,-




-anaphylactic, asthma, hay fever i,- i,- i,- i,-




treatment: epinephrine i,-




Hypersensitivity: Type 2 i,- i,- i,-i,- i,- -Cytotoxic reaction: tissue specific i,- i,- i,-




-binds to the antigen ON THE CELL SURFACE
i,- i,- i,- i,- i,- i,- i,- i,-




-macrophages are primary effectors cells involved i,- i,- i,- i,- i,- i,-




cause tissue damage or alter function
i,- i,- i,- i,- i,-




Hypersensitivity 2 -Graves Disease (hyperthyroidism) -alters i,- i,-i,- i,- i,- i,- i,- i,-



function not destroy i,- i,- i,-




-blood transfusions reaction- transfused erythrocytes are
i,- i,- i,- i,- i,- i,-



destroyed by agglutination or lysis i,- i,- i,- i,- i,-

,-drug allergies i,- i,-




-hemolytic anemia i,-




Hypersensitivity: type 3 i,- i,- i,-i,- i,- -Not organ specific i,- i,- i,-




-antibody binds to soluble antigen OUTSIDE THE CELL SURFACE
i,- i,- i,- i,- i,- i,- i,- i,- i,-



that was released into the blood or bodily fluids, and the complex
i,- i,- i,- i,- i,- i,- i,- i,- i,- i,- i,- i,-



is then deposited in the tissues
i,- i,- i,- i,- i,-




Hypersensitivity: Type 3 i,- i,- i,-i,- i,- -rheumatoid arthritis i,- i,-




-Systemic Lupus Erythematosus (SLE) i,- i,- i,- i,-




-Raynaud's


Hives (urticaria) i,- i,-i,- i,- Hypersensitivity Type 1 i,- i,-




First responders to innate the immune system
i,- i,- i,- i,- i,- i,- i,-i,- i,- Neutrophils


Allergic contact dermatitis i,- i,- i,-i,- i,- Hypersensitivity Type IV i,- i,-




Type 2 cytotoxic hypersensitivity mediated by:
i,- i,- i,- i,- i,- i,-i,- i,- -IgG IgM i,-




-macrophages are primary effort cells i,- i,- i,- i,- i,-




-

, Serum Sickness i,- i,-i,- i,- type 3 hypersensitivity i,- i,-




Hypersensitivity IV, i,- i,-i,- i,- -is more of a delayed immune response.
i,- i,- i,- i,- i,- i,-




-mediated T-cells attack tissue directly (no antibodies)
i,- i,- i,- i,- i,- i,-




Autoimmune -diseases in which the body makes antibodies
i,-i,- i,- i,- i,- i,- i,- i,- i,- i,-



directed against its own tissues
i,- i,- i,- i,-




-


Primary immunodeficiency
i,- i,-i,- i,- -Genetic; inherited i,- i,-




-result of single gene defects
i,- i,- i,- i,- i,-




-B and T cell deficiencies
i,- i,- i,- i,-




secondary immunodeficiency i,- i,-i,- i,- -acquired


Common cause of secondary immunodeficiency
i,- i,- i,- i,- i,-i,- i,- Malnutrition


Most patients diagnosed with SLE will have a positive ?
i,- i,- i,- i,- i,- i,- i,- i,- i,- i,-i,- i,-



antinuclear antibody (ANA) i,- i,-
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