NSG 3850 EXAM 3 ACTUAL EXAM 2026 || MOST RECENT EXAM
2026|2027 ACTUAL COMPLETE REAL EXAM QUESTIONS AND
CORRECT ANSWERS (VERIFIED ANSWERS) ALREADY
GRADED A+ | GUARANTEED SUCCESS!! BRAND NEW
VERSION | JUST RELEASED!! GALEN COLLEGE OF
NURSING
clinical manifestations include paresthesia, muscle cramps,
tetany, fatigue,
neuromuscular irritability, seizures, anxiety prolongation
of Q-T intervals,
laryngospasms, depression - ANSWER-
hypoparathyroidism
Clinical manifestations are related to hypercalcemia. pt more
prone to kidney stones, polyuria, dehydration, anorexia, N/V,
and constipation. Causes cardiac issues like bradycardia, heart
blocks, and cardiac arrest - ANSWER hyperparathyroidism
primary: idiopathic, autoimmune, trauma. Secondary:
corticosteroid use,
damage to hypothalamus or pituitary gland. also called adrenal
insufficiency - ANSWER-Addison's disease
clinical manifestations: weakness, hypotension,
hyperpigmentation (bronze/tanned), hyperkalemia,
hyponatremia (salt cravings) – ANSWER-Addison’s disease
,complication of Addison’s that causes diminished vascular
tone, reduced cardiac output, and inadequate circulating blood
volume. these things can contribute to vascular collapse. pt will
not produce cortisol when tested – ANSWER-Addisonian crisis
a disease of the adrenal cortex resulting in conditions
characterized by
hypercortisolism - ANSWER-Cushing
syndrome
Primary: caused by disease of adrenal cortex (adrenal
adenoma) secondary: caused by ACTH secreting cells, Tertiary:
caused by hypothalamic dysfunction or injury - ANSWER-
Cushing syndrome
clinical manifestations: truncal obesity, moon face, flushed
cheeks, muscle wasting, petechia, risk for fracture, depression
and anxiety - ANSWER-Cushing syndrome
labs will show hyperglycemia, hypokalemia, elevated serum
cortisol - ANSWER-
cushing
syndrome
Type I hypersensitivity= - ANSWER-anaphylaxis
Type II hypersensitivity= - ANSWER-transfusion reaction
Type IIb hypersensitivity= - ANSWER-graves disease
,type III hypersensitivity complex glomerulonephritis can
be related to... -
ANSWER-dormant bacterial
infecton
type IV hypersensitivity= - ANSWER-TB test
abnormal immune system in which the phagocytes cant get
along and fight, instead they destroy themselves and each
other. kidney involvement is common
(nephritis and glomerulonephritis) - ANSWER-systemic lupus
erythematosus (SLE)
clinical manifestations: butterfly nose rash, rash on scalp, ears,
chest, and arms
- ANSWER-discoid
lupus
inflammatory response in joints leading to accumulation of
dense aggregates (clusters) of immune cells. This stimulates
edema and proliferation of a synovium (swelling in joints), and
well as fatigue, fever, and flu like symptoms - ANSWER-
rheumatoid arthritis
cause is unknown. Inflammation and immune cell
infiltration found in skin,
lungs, and other tissue. - ANSWER-
scleroderma
, vasculopathy and vascular in smooth muscle cells due to cell
proliferation that
leads to their dysfunction. Tissue ischemia followed by diffuse
tissue fibrosis. -
ANSWER-
scleroderma
clinical manifestations: Raynaud's phenomenon, swelling in
fingers/ hands/ and
feet, lesions on lips/ face, muscle atrophy,
inflammatory myositis, GI
involvement, pulmonary involvement - ANSWER-
scleroderma
slower, gradual onset. high numbers of mature WBC in
blood - ANSWER-
chronic myeloid leukemia
(CML)
s/s: high granulocyte count, splenomegaly, fatigue weight
loss. does not
respond well to chemo - ANSWER-chronic myeloid
leukemia (CML)
diagnosis: CBC with differential, physical exam, bone
marrow aspiration -
ANSWER-Chronic myeloid
leukemia (CML)
2026|2027 ACTUAL COMPLETE REAL EXAM QUESTIONS AND
CORRECT ANSWERS (VERIFIED ANSWERS) ALREADY
GRADED A+ | GUARANTEED SUCCESS!! BRAND NEW
VERSION | JUST RELEASED!! GALEN COLLEGE OF
NURSING
clinical manifestations include paresthesia, muscle cramps,
tetany, fatigue,
neuromuscular irritability, seizures, anxiety prolongation
of Q-T intervals,
laryngospasms, depression - ANSWER-
hypoparathyroidism
Clinical manifestations are related to hypercalcemia. pt more
prone to kidney stones, polyuria, dehydration, anorexia, N/V,
and constipation. Causes cardiac issues like bradycardia, heart
blocks, and cardiac arrest - ANSWER hyperparathyroidism
primary: idiopathic, autoimmune, trauma. Secondary:
corticosteroid use,
damage to hypothalamus or pituitary gland. also called adrenal
insufficiency - ANSWER-Addison's disease
clinical manifestations: weakness, hypotension,
hyperpigmentation (bronze/tanned), hyperkalemia,
hyponatremia (salt cravings) – ANSWER-Addison’s disease
,complication of Addison’s that causes diminished vascular
tone, reduced cardiac output, and inadequate circulating blood
volume. these things can contribute to vascular collapse. pt will
not produce cortisol when tested – ANSWER-Addisonian crisis
a disease of the adrenal cortex resulting in conditions
characterized by
hypercortisolism - ANSWER-Cushing
syndrome
Primary: caused by disease of adrenal cortex (adrenal
adenoma) secondary: caused by ACTH secreting cells, Tertiary:
caused by hypothalamic dysfunction or injury - ANSWER-
Cushing syndrome
clinical manifestations: truncal obesity, moon face, flushed
cheeks, muscle wasting, petechia, risk for fracture, depression
and anxiety - ANSWER-Cushing syndrome
labs will show hyperglycemia, hypokalemia, elevated serum
cortisol - ANSWER-
cushing
syndrome
Type I hypersensitivity= - ANSWER-anaphylaxis
Type II hypersensitivity= - ANSWER-transfusion reaction
Type IIb hypersensitivity= - ANSWER-graves disease
,type III hypersensitivity complex glomerulonephritis can
be related to... -
ANSWER-dormant bacterial
infecton
type IV hypersensitivity= - ANSWER-TB test
abnormal immune system in which the phagocytes cant get
along and fight, instead they destroy themselves and each
other. kidney involvement is common
(nephritis and glomerulonephritis) - ANSWER-systemic lupus
erythematosus (SLE)
clinical manifestations: butterfly nose rash, rash on scalp, ears,
chest, and arms
- ANSWER-discoid
lupus
inflammatory response in joints leading to accumulation of
dense aggregates (clusters) of immune cells. This stimulates
edema and proliferation of a synovium (swelling in joints), and
well as fatigue, fever, and flu like symptoms - ANSWER-
rheumatoid arthritis
cause is unknown. Inflammation and immune cell
infiltration found in skin,
lungs, and other tissue. - ANSWER-
scleroderma
, vasculopathy and vascular in smooth muscle cells due to cell
proliferation that
leads to their dysfunction. Tissue ischemia followed by diffuse
tissue fibrosis. -
ANSWER-
scleroderma
clinical manifestations: Raynaud's phenomenon, swelling in
fingers/ hands/ and
feet, lesions on lips/ face, muscle atrophy,
inflammatory myositis, GI
involvement, pulmonary involvement - ANSWER-
scleroderma
slower, gradual onset. high numbers of mature WBC in
blood - ANSWER-
chronic myeloid leukemia
(CML)
s/s: high granulocyte count, splenomegaly, fatigue weight
loss. does not
respond well to chemo - ANSWER-chronic myeloid
leukemia (CML)
diagnosis: CBC with differential, physical exam, bone
marrow aspiration -
ANSWER-Chronic myeloid
leukemia (CML)
