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NSG 233 Exam 4 Study Guide 2025: Complex Health Alterations Practice Questions & Review

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Prepare for your NSG 233 Complex Health Alterations Exam 4. Our 2025 study guide covers advanced pathophysiology, critical care concepts, complex disease management, and clinical decision-making with practice questions for comprehensive patient care.

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2025/2026
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Page 1 of 77


NSG233 / NSG 233 Exam 4 (Latest 2025 Update):

Medical-Surgical Nursing III | Questions and Verified

Answers | 100% Correct | Grade A - Herzing

asphyxia .....ANSWER.....Insufficient oxygen and excess carbon

dioxide in the blood and tissues.

bilirubin encephalopathy .....ANSWER.....Acute manifestation of

bilirubin toxicity occurring in the first weeks after birth.

erythroblastosis fetalis .....ANSWER.....Agglutination and

hemolysis of fetal erythrocytes caused by incompatibility

between the maternal and fetal blood types, such as when the

fetus is Rh-positive and the mother is Rh-negative.

esophageal atresia .....ANSWER.....Condition in which the

esophagus is separated from the stomach and ends in a blind

pouch.

,Page 2 of 77


gastroschisis .....ANSWER.....Protrusion of the intestines through a

defect in the abdominal wall. The intestines are not covered by a

peritoneal sac or skin.

hydrops fetalis .....ANSWER.....Heart failure and generalized

edema in the fetus secondary to severe anemia resulting from

destruction of erythrocytes.

kernicterus .....ANSWER.....Chronic and permanent result of

bilirubin toxicity.

meningocele .....ANSWER.....Protrusion of the meninges through a

defect in the vertebrae; a form of neural tube defect.

myelomeningocele .....ANSWER.....Protrusion of the meninges and

spinal cord through a defect in the vertebrae; a form of neural

tube defect.

neonatal abstinence syndrome .....ANSWER.....A cluster of

physical signs exhibited by newborns exposed in utero to

maternal use of substances such as heroin.

,Page 3 of 77


omphalocele .....ANSWER.....Protrusion of the intestines into the

base of the umbilical cord. The intestines are covered by a

peritoneal sac.

persistent pulmonary hypertension .....ANSWER.....Vasoconstriction

of the infant's pulmonary vessels after birth; may result in right-

to-left shunting of blood flow through the ductus arteriosus, the

foramen ovale, or both.

spina bifida .....ANSWER.....Defective closure of the bony spine

that encloses the spinal cord; a type of neural tube defect.

tracheoesophageal fistula .....ANSWER.....Abnormal connection

between the esophagus and trachea.

transient tachypnea of the newborn .....ANSWER.....Condition of

rapid respirations caused by inadequate absorption of fetal lung

fluid.

How is TTN different from RDS? .....ANSWER.....Transient

tachypnea of the newborn is thought to be caused by failure of

, Page 4 of 77


fetal lung fluid to be absorbed completely in late preterm, full-

term, or preterm infants. Respiratory distress syndrome (RDS)

occurs in preterm infants as a result of inadequate surfactant. It is

less serious than RDS but is the most common respiratory cause of

NICU admission.

Why is there resistance of blood flow into the lungs in PPHN?

.....ANSWER.....Infants with PPHN have constriction of the

pulmonary blood vessels from inadequate oxygen levels. This

increases resistance to blood flow into the lungs and causes

blood to flow through the foramen ovale and patent ductus

arteriosus.

How can kernicterus be prevented? .....ANSWER.....Kernicterus

can be prevented by identifying women whose infants are at risk

for blood incompatibilities, giving Rh-negative mothers Rh

immune globulin, recognizing infants with bilirubin levels that are

not normal, and instituting phototherapy when it is needed.
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