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Midterm Exam: NR507 / NR 507 (Latest 2025 / 2026) Advanced Pathophysiology | Questions & Answers | 100% Correct | Grade A - Chamberlain

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Midterm Exam: NR507 / NR 507 (Latest 2025 / 2026) Advanced Pathophysiology | Questions & Answers | 100% Correct | Grade A - Chamberlain

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Midterm Exam: NR507 / NR 507 (Latest
) Advanced Pathophysiology
| Questions & Answers | 100% Correct |
Grade A - Chamberlain

_____________________________________________________________________________________

what are the 3 aspects of pulmonary ventilation must be considered to understand pathophysiology
of restrictive lung disease

-compliance
-lung volumes and capacity
-work of breathing

what are components of compliance in restrictive lung disease

what are components of lung volume and capacity in restrictive lung disease

what are components of work of breathing in restrictive lung disease

what are the 6 classic signs of restrictive lung disease

-Tachypnea-inspiratory muscles work hard to overcome decreased pulmonary compliance
-Hypoxemia-low O2 saturation-mismatch of ventilation-perfusion-caused by collagenous framework of
lung, scarring of capillary channels, distortion of small airways, compression of tissue from tumors.
-Decreased breath sounds with dry inspiratory crackles (heard a lung base)
-Decreased lung volume and capacity-as seen with pulmonary function tests (PFTs)
-Decreased diffusing capacity of lung for carbon monoxide-consequence of increased interstitial space
due to scar tissue, fibrosis of capillaries
-Cor pulmonale-right sided heart failure caused by fibrosis, hypoxemia and compression of pulmonary
capillaries, leading to pulmonary hypertension, increased right ventricle work, decreased chest wall
expansion, cyanosis, clubbing of nails

what are 3 symptoms of restrictive lung disease

-Dyspnea-shortness of breath
-Dry, non-productive cough
-Emaciated appearance-due to increased work of breathing

what are basic treatments for restrictive lung disease

, -if causes are permanent (SCI, progressive diseases)-supportive measures (O2, antibiotics)
-If causes are acute/reversable-specific corrective interventions (chest tube, vent)

what is atelectasis

Incomplete expansion of alveoli, lung parenchyma is collapsed and non-aerated

what is PT treatment for atelectasis

-deep breathing techniques
-mobility out of bed

what are the 5 subtypes of atelectasis

-resorptive/obstructive
-passive loss of lung volume
-adhesive
-compressive
-cicatrization

what is resorptive/obstructive atelectasis

IE-tumor, foreign body, mucus plug

what is passive loss of lung volume atelectasis

caused by pneumothorax or diaphragmatic dysfunction-caused by anesthesia, lack of deep breathing,
bed rest, sedatives

what is adhesive atelectasis

surfactant deficiency causing alveoli to collapse, walls stick together, hard to aerate-see with smoke
inhalation, ARDS, PE, cardiac bypass, pneumonia

what is compressive atelectasis

-lung tissue compressed by space occupying lesion, squeezing air out of alveoli-pleural effusion (fluid
collection in pleural space between lung and chest wall), pleural tumor, empyema (pus collection in
pleural space between lung and chest wall)

what is cicatrization atelectasis

volume loss caused by decreased pulmonary compliance-fibrosis

what are infectious causes of restrictive lung disease

Pneumonia -inflammatory process of lung parenchyma beginning with infection in lower respiratory
tract due to bacteria, virus, fungi or mycoplasms

what are the 4 subtypes of infectious causes of RLD

-Community acquired (CAP)-acute and chronic, typically from contact in community
-Hospital acquired(HAP)-usually following NG tube placement, intubation, dysphagia, tracheostomy,
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