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CDCES Exam 2025 – Complete Study Guide with Practice Questions and Key Concepts for Certification

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This comprehensive document provides a complete overview of the CDCES (Certified Diabetes Care and Education Specialist) exam content for 2025. It includes quick-reference answers, practice questions, clinical criteria, medication mechanisms, insulin conversion guidelines, DSMES program steps, motivational interviewing frameworks, and management strategies for acute and chronic diabetes complications. The document is ideal for exam preparation, offering accurate, concise, and exam-aligned material with detailed medical, nutritional, and pharmacological insights.

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Instelling
Certified Diabetes Care And Education Specialist
Vak
Certified Diabetes Care and Education Specialist

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CDCES Exam 2025
how much of a plum would be 15 grams of carbs -
✅1 plum

Medicare DPP FBG Criteria -
✅110-125

CDC DPP FBG Criteria -
✅100-125

Which DPP program will NOT accept self reported blood sugars? -
✅Medicare DPP

Which DPP program will NOT accept patients with a history of ESRD or GDM? -
✅Medicare DPP

Which DPP program will NOT accept patients if they are currently pregnant? -
✅CDC DPP

Which medication can mask hypoglycemia symptoms? -
✅Beta Blockers

Simvastatin plus which medication can increase risk of myalgias? -
✅Gemfibrozil

What are the 8 risk factors for OSA? -
✅1. Age >60
2. African American (ONLY APPLIES if <35 yo)
3. Large neck
4. Heavy ETOH use
5. OTC decongestant use
6. Family history
7. Smoking
8. Male

Which lab value provides insight into short term glycemic outcomes and glucose excursions? -
✅1,5 AG

Which lab value will be inaccurate in advanced kidney or liver disease? -
✅1,5 AG

In patients with diabetes, who would qualify for ASA for primary prevention? -
✅age 50+ with 1+ additional risk factor

A1C goal in 2nd trimester -


1

, ✅<6%

For an emergency kit you should have: -
✅2 days of food + 3 days of water + 1 week of meds

Taking which med before bed (instead of before dinner) can help prevent nighttime
hypoglycemia and improve FBG? -
✅NPH

Low carb meal, increased activity, etoh, delayed/missed meals, insulin timing, too much meds
are all causes of: -
✅hypoglycemia

What are some ways to treat hypoglycemia? -
✅4-8 oz of sugary drink
handful of raisins
piece of fruit
4+ glucose tabs
glucose hel/honey
1 cup of milk
15+ skittles

Which hypoglycemia med is approved for all ages? -
✅Glucagon

Glucagon can be given via ___ -
✅SC or IM injection

Bolus insulin would lower -
✅Post prandial sugars

Basal insulin would lower -
✅FBG and in between meals

Name 2 types of bolus insulins -
✅Rapid LAG (lispro, aspart, glulisine)
Short (regular)

Name 2 types of basal insulins -
✅Intermediate (NPH)
Long "DDG" (degludec, detemir, glargine)

How to calculate estimated glucose? -
✅A1C 5 correlates to eGlucose of 97. For each additional A1C add +29.

What happens in Phase I (Fed State)? -
✅1. exogenous glucose is main source of glucose
2. increase insulin, decrease in glucagon


2

,3. insulin prevents glycogen breakdown
4. all tissues use glucose
5. excess glucose is stored as glycogen and TG

What happens in Phase II (post absorptive state)? -
✅1. insulin decrease, increase glucagon
2. glycogen breakdown is main source of glucose
3. hepatic gluconeogenesis also happens
4. adipose tissues break down TG into FFA
5. Glucose: used by all tissues except liver

What happens in Phase III (early starvation state)? -
✅1. some glycogen breakdown
2. but main source is hepatic gluconeogenesis
3. lactate provides 50% of gluconeogenesis substrate
4. AA, alanine, glycerol are other substrates
5. decrease insulin, increase counter-regulatory hormones (GH, cortisol, glucagon, epi)
6. Glucose: used by all tissues except liver

What happens in Phase IV (preliminary prolonged starvation)? -
✅1. renal + hepatic gluconeogenesis
2. brain: uses glucose > ketones
3. glucose: brain, RBC, renal medulla
4. increase counter-regulatory hormones
5. protein breakdown (catabolism) starts to happen because fat stores are depleted

What happens in Phase V (secondary prolonged starvation)? -
✅1. renal + hepatic gluconeogenesis
2. brain: uses ketones > glucose
3. glucose: used by brain, RBC, renal medulla

Phase I (Fed State) Timeline -
✅0-4 hours post meal

Phase II (post absorptive state) Timeline -
✅4-16 hours post meal

Phase III (early starvation state) timeline -
✅16-48 hours post meal

Phase IV (preliminary prolonged starvation) Timeline -
✅2-24 days

Phase V (secondary prolonged starvation) timeline -
✅24-40 days

Health and social conditions increase burden or disease on a community -
✅Syndemic


3

, #new cases/specific timeframe (Measures risk of people developing diabetes) -
✅Incidence

#people who already have diabetes -
✅Prevalence

focuses on clinical practice that integrates knowledge -
✅Clinical management and Integration

focuses on communication that is essential to optimize quality of care -
✅Communication and advocacy

identify ways to increase behavioral change, QOL, self management across lifespan -
✅person centered care and education across lifespan

identify research and quality improvement competencies essential to guide research and QI
activities -
✅research quality and improvement

systematic review of process and outcome data -
✅quality improvement

example: let's try to increase # of GDM patients in our program by 10% -
✅quality improvement

apply business principles, population health, systems practice to impact outcome of systems,
providers, populations -
✅systems wide practice

address competencies related to lifelong learning and professionalism -
✅professional practice

Help patients gain SKILLS, KNOWLEDGE, and BEHAVIORS to self manage their diabetes -
✅Goals of DSMES

When to refer to DSMES -
✅1. at diagnosis
2. annually
3. complicating factors (SDOH change)
4. life transition (new job/insurance/doctor)

Steps of DSMES program -
✅"AGPIE"
Assessment
Goal Setting
Planning
Implementation
Evaluation


4

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Certified Diabetes Care and Education Specialist
Vak
Certified Diabetes Care and Education Specialist

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