Exam 1 Blueprint NUR 529 Questions With
Complete Solutions
Liquefaction necrosis- example
softening of the center of an abscess with
discharge of its contents
Coagulation necrosis
acidosis devleops and dentaures enzymatic and
structural proteins of the cell
characteristic of hypoxic injury; seen in infarcted
areas
Caseous necrosis
dead cells persist indefinitely
ex: center of tuberculous granulomnas/tubercles
cheese-like substance
Gangrene types
wet
dry
gas (clostridium perfringens)
Ischemia
decreased blood flow leading to decreased O2 and
nutrients
ex: blockage of a vessel (blood clot),
,atherosclerosis, arterial spasm
can be reversible if blood flow is restored in time
infarction
necrosis due to prolonged ischemia
ex: thrombus/ embolus, dz of arterial wall, pressure
from outside the vessel
Fatty necrosis
breakdown of fatty tissue into fatty acids and
glycerol
formation of chalky white areas due to fatty acids
and calcium that form sops (saponification)
examples of fatty necrosis
trauma to breast, pancreas, subq tissue
acute pancreattitis due to release of enzymes like
lipase
Intracellular waste build up
can be normal or abnormal
normal causes of intrcellular waste buildup
lipids, proteins, carbs, melanin, bilirubin in large
amounts
Tay-Sachs disease
genetic
abnormal lipids build up in the brain and other
, tissues
motor/ mental deterioration
Pathologic calcificatons
dystrophic/ metastatic
abnormal tissues deposition of calcium salts,
together with smaller amounts of iron, mag, and
other minerals
Dystrophic calcifications
pathologic
in DEAD/DYING tissues
macroscopic )visible to the naked eye)
gritty grains to rock material
intracellular/ extracellular formation of crystalline
calcium phosphate
most prevalent mechanism of calcific aortic valve
disease
examples of dysrtrophic calcifications
atheromatous lesions of advanced atherosclerosis
areas of injury in the aorta and large blood vessels
necrotic tissues
Metastatic calcifications
pathologic
in NORMAL tissues
results from increased serum calcium
Complete Solutions
Liquefaction necrosis- example
softening of the center of an abscess with
discharge of its contents
Coagulation necrosis
acidosis devleops and dentaures enzymatic and
structural proteins of the cell
characteristic of hypoxic injury; seen in infarcted
areas
Caseous necrosis
dead cells persist indefinitely
ex: center of tuberculous granulomnas/tubercles
cheese-like substance
Gangrene types
wet
dry
gas (clostridium perfringens)
Ischemia
decreased blood flow leading to decreased O2 and
nutrients
ex: blockage of a vessel (blood clot),
,atherosclerosis, arterial spasm
can be reversible if blood flow is restored in time
infarction
necrosis due to prolonged ischemia
ex: thrombus/ embolus, dz of arterial wall, pressure
from outside the vessel
Fatty necrosis
breakdown of fatty tissue into fatty acids and
glycerol
formation of chalky white areas due to fatty acids
and calcium that form sops (saponification)
examples of fatty necrosis
trauma to breast, pancreas, subq tissue
acute pancreattitis due to release of enzymes like
lipase
Intracellular waste build up
can be normal or abnormal
normal causes of intrcellular waste buildup
lipids, proteins, carbs, melanin, bilirubin in large
amounts
Tay-Sachs disease
genetic
abnormal lipids build up in the brain and other
, tissues
motor/ mental deterioration
Pathologic calcificatons
dystrophic/ metastatic
abnormal tissues deposition of calcium salts,
together with smaller amounts of iron, mag, and
other minerals
Dystrophic calcifications
pathologic
in DEAD/DYING tissues
macroscopic )visible to the naked eye)
gritty grains to rock material
intracellular/ extracellular formation of crystalline
calcium phosphate
most prevalent mechanism of calcific aortic valve
disease
examples of dysrtrophic calcifications
atheromatous lesions of advanced atherosclerosis
areas of injury in the aorta and large blood vessels
necrotic tissues
Metastatic calcifications
pathologic
in NORMAL tissues
results from increased serum calcium
