RELIAS ADVANCED
DYSRHYTHMIA EXAM A
2025/2026 COMPLETE 52
QUESTIONS WITH
STEP-BY-STEP VERIFIED
ANSWERS AND CLINICAL
ASSESSMENT EXPLANATIONS
Comprehensive Guide for Exam Preparation September 19, 2025
,1 Introduction
This refactored guide provides a comprehensive review of advanced dysrhythmias, de-
signed for clarity and practical application in preparation for the Relias Advanced Dys-
rhythmia Exam A 2025/2026. The original 52 questions have been revised for conciseness,
eliminating redundancy while maintaining accuracy, and are paired with step-by-step ex-
planations and clinical insights. An additional 100 relevant questions have been crafted
to cover a broader range of cardiac rhythms, ensuring thorough preparation. Questions
are highlighted in a very dark red color for emphasis, as requested, to enhance readability
and focus during study.
The document is organized by rhythm type for logical flow, with each question numbered,
answered concisely, and supported by clinical assessments to bridge theory and practice.
This version improves upon previous iterations by streamlining explanations, reducing
jargon, and expanding practice scenarios.
2 Revised Original 52 Questions and Answers
1 What defines atrial fibrillation on an ECG?
Answer: Wavy baseline, no P waves, irregular R-R intervals, narrow QRS.
Step-by-Step: Confirm absent P waves (atrial chaos), check irregular ventricular
rhythm, verify QRS width.
Clinical Assessment: Monitor for palpitations or fatigue; assess stroke risk and
consider anticoagulation.
2 Which dysrhythmias result from fluid volume deficit?
Answer: Tachycardias (e.g., sinus tachycardia, SVT).
Step-by-Step: Reduced volume lowers cardiac output, prompting faster heart rate.
Clinical Assessment: Look for dehydration signs (e.g., dry mucous membranes);
administer IV fluids.
3 What causes fluid volume deficit?
Answer: Hemorrhage, vomiting, diarrhea, fever, excessive diuretics.
Step-by-Step: Identify sources of fluid loss reducing preload.
Clinical Assessment: Check vitals for hypotension; correct with fluid replacement.
4 What dysrhythmias are associated with fluid volume overload?
Answer: Premature atrial or ventricular beats, variable rates.
Step-by-Step: Excess fluid stretches cardiac tissue, triggering ectopic beats.
Clinical Assessment: Assess for edema or dyspnea; use diuretics to reduce volume.
5 What medications are administered before cardioversion?
Answer: Sedatives (e.g., midazolam) for comfort.
Step-by-Step: Cardioversion is painful; sedation ensures patient tolerance.
Clinical Assessment: Confirm stability; monitor respiratory status post-procedure.
6 Where do junctional rhythms originate?
Answer: AV node.
Step-by-Step: SA node failure prompts AV node pacing.
Clinical Assessment: Rate 40-60 bpm; check for dizziness.
1
, 7 What are the ECG features of a junctional rhythm?
Answer: Absent or inverted P waves, rate 40-60, narrow QRS.
Step-by-Step: Look for missing or retrograde P waves, regular rhythm.
Clinical Assessment: Evaluate for symptoms like weakness; consider pacing if per-
sistent.
8 What triggers junctional rhythms?
Answer: Digoxin toxicity, sick sinus syndrome, beta blockers, post-cardiac surgery.
Step-by-Step: Identify factors suppressing SA node.
Clinical Assessment: Review medication history; discontinue causative agents.
9 How is a junctional rhythm treated?
Answer: Stop offending drugs, administer atropine, or use pacemaker.
Step-by-Step: Atropine increases rate if symptomatic; pacing for chronic issues.
Clinical Assessment: Monitor heart rate; prepare for transcutaneous pacing.
10 What characterizes a PVC on ECG?
Answer: Early, wide, bizarre QRS without P wave.
Step-by-Step: Identify premature wide QRS followed by compensatory pause.
Clinical Assessment: Common in healthy hearts; frequent PVCs may indicate
pathology.
11 What is bigeminy in the context of PVCs?
Answer: Every other beat is a PVC.
Step-by-Step: Observe alternating normal-PVC pattern.
Clinical Assessment: May reduce cardiac output; address underlying cause.
12 What defines trigeminy with PVCs?
Answer: Every third beat is a PVC.
Step-by-Step: Pattern of two normal beats, one PVC.
Clinical Assessment: Less concerning; check electrolytes.
13 How are PVCs managed?
Answer: Correct low potassium or magnesium, provide oxygen, reduce stimulants.
Step-by-Step: Review labs, eliminate triggers like caffeine.
Clinical Assessment: Consider beta blockers for symptomatic cases.
14 When are PVCs considered frequent?
Answer: More than 6 per minute.
Step-by-Step: Count PVCs over 60 seconds; high frequency risks VT.
Clinical Assessment: Prepare antiarrhythmics for escalation.
15 What is ventricular tachycardia?
Answer: Rapid ventricular rhythm, pulseless if sustained.
Step-by-Step: Identify wide QRS, rate >100 bpm, regular rhythm.
Clinical Assessment: Emergency; defibrillate if no pulse.
16 How is ventricular fibrillation treated?
Answer: Immediate CPR and defibrillation.
Step-by-Step: Deliver shock to restore rhythm, continue CPR.
Clinical Assessment: Activate code; administer epinephrine.
17 What defines asystole on ECG?
2
DYSRHYTHMIA EXAM A
2025/2026 COMPLETE 52
QUESTIONS WITH
STEP-BY-STEP VERIFIED
ANSWERS AND CLINICAL
ASSESSMENT EXPLANATIONS
Comprehensive Guide for Exam Preparation September 19, 2025
,1 Introduction
This refactored guide provides a comprehensive review of advanced dysrhythmias, de-
signed for clarity and practical application in preparation for the Relias Advanced Dys-
rhythmia Exam A 2025/2026. The original 52 questions have been revised for conciseness,
eliminating redundancy while maintaining accuracy, and are paired with step-by-step ex-
planations and clinical insights. An additional 100 relevant questions have been crafted
to cover a broader range of cardiac rhythms, ensuring thorough preparation. Questions
are highlighted in a very dark red color for emphasis, as requested, to enhance readability
and focus during study.
The document is organized by rhythm type for logical flow, with each question numbered,
answered concisely, and supported by clinical assessments to bridge theory and practice.
This version improves upon previous iterations by streamlining explanations, reducing
jargon, and expanding practice scenarios.
2 Revised Original 52 Questions and Answers
1 What defines atrial fibrillation on an ECG?
Answer: Wavy baseline, no P waves, irregular R-R intervals, narrow QRS.
Step-by-Step: Confirm absent P waves (atrial chaos), check irregular ventricular
rhythm, verify QRS width.
Clinical Assessment: Monitor for palpitations or fatigue; assess stroke risk and
consider anticoagulation.
2 Which dysrhythmias result from fluid volume deficit?
Answer: Tachycardias (e.g., sinus tachycardia, SVT).
Step-by-Step: Reduced volume lowers cardiac output, prompting faster heart rate.
Clinical Assessment: Look for dehydration signs (e.g., dry mucous membranes);
administer IV fluids.
3 What causes fluid volume deficit?
Answer: Hemorrhage, vomiting, diarrhea, fever, excessive diuretics.
Step-by-Step: Identify sources of fluid loss reducing preload.
Clinical Assessment: Check vitals for hypotension; correct with fluid replacement.
4 What dysrhythmias are associated with fluid volume overload?
Answer: Premature atrial or ventricular beats, variable rates.
Step-by-Step: Excess fluid stretches cardiac tissue, triggering ectopic beats.
Clinical Assessment: Assess for edema or dyspnea; use diuretics to reduce volume.
5 What medications are administered before cardioversion?
Answer: Sedatives (e.g., midazolam) for comfort.
Step-by-Step: Cardioversion is painful; sedation ensures patient tolerance.
Clinical Assessment: Confirm stability; monitor respiratory status post-procedure.
6 Where do junctional rhythms originate?
Answer: AV node.
Step-by-Step: SA node failure prompts AV node pacing.
Clinical Assessment: Rate 40-60 bpm; check for dizziness.
1
, 7 What are the ECG features of a junctional rhythm?
Answer: Absent or inverted P waves, rate 40-60, narrow QRS.
Step-by-Step: Look for missing or retrograde P waves, regular rhythm.
Clinical Assessment: Evaluate for symptoms like weakness; consider pacing if per-
sistent.
8 What triggers junctional rhythms?
Answer: Digoxin toxicity, sick sinus syndrome, beta blockers, post-cardiac surgery.
Step-by-Step: Identify factors suppressing SA node.
Clinical Assessment: Review medication history; discontinue causative agents.
9 How is a junctional rhythm treated?
Answer: Stop offending drugs, administer atropine, or use pacemaker.
Step-by-Step: Atropine increases rate if symptomatic; pacing for chronic issues.
Clinical Assessment: Monitor heart rate; prepare for transcutaneous pacing.
10 What characterizes a PVC on ECG?
Answer: Early, wide, bizarre QRS without P wave.
Step-by-Step: Identify premature wide QRS followed by compensatory pause.
Clinical Assessment: Common in healthy hearts; frequent PVCs may indicate
pathology.
11 What is bigeminy in the context of PVCs?
Answer: Every other beat is a PVC.
Step-by-Step: Observe alternating normal-PVC pattern.
Clinical Assessment: May reduce cardiac output; address underlying cause.
12 What defines trigeminy with PVCs?
Answer: Every third beat is a PVC.
Step-by-Step: Pattern of two normal beats, one PVC.
Clinical Assessment: Less concerning; check electrolytes.
13 How are PVCs managed?
Answer: Correct low potassium or magnesium, provide oxygen, reduce stimulants.
Step-by-Step: Review labs, eliminate triggers like caffeine.
Clinical Assessment: Consider beta blockers for symptomatic cases.
14 When are PVCs considered frequent?
Answer: More than 6 per minute.
Step-by-Step: Count PVCs over 60 seconds; high frequency risks VT.
Clinical Assessment: Prepare antiarrhythmics for escalation.
15 What is ventricular tachycardia?
Answer: Rapid ventricular rhythm, pulseless if sustained.
Step-by-Step: Identify wide QRS, rate >100 bpm, regular rhythm.
Clinical Assessment: Emergency; defibrillate if no pulse.
16 How is ventricular fibrillation treated?
Answer: Immediate CPR and defibrillation.
Step-by-Step: Deliver shock to restore rhythm, continue CPR.
Clinical Assessment: Activate code; administer epinephrine.
17 What defines asystole on ECG?
2
