Anaphylactic shock treatment
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-Drug of choice is epinephrine
-Maintaining airway is critical
-Aggressive fluid replacement
,Obstructive shock
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Physical obstruction to blood flow occurs with a decreased CO
Phases of AKI
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Oliguric, diuretic, recovery
Compensatory stage of shock
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-clinically apparent and involves neural, hormonal, and biochemical
compensatory mechanisms in an attempt to overcome the increasing
consequences of anaerobic metabolism and to maintain homeostasis
- decreased urine output
-increased HR
-Increased glucose
Urinary Incontinence management
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, -Adequate volume of fluids and reduction or elimination of bladder irritants
from the diet
-Voiding regimens, bladder retraining, pelvic floor muscle training
-Drug therapy, devices, surgery
Electrolyte changes in CKD
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High: Sodium, potassium, phosphorus
Low: Calcium, Vitamin D
hemodialysis (HD)
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-treatment for renal failure using artificial kidney machine to filter waste
from blood
- Complications: hypotension, muscle cramps, blood loss
Manifestations of Addison's disease
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-slow onset
-weakness, weight loss, anorexia
-skin hyperpigmentation
- hypoglycemia
, Cushing's disease manifestations
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-Central obesity, moon face, purple striae, buffalo hump, hirsutism in
women, hyperglycemia, hypertension, hypokalemia, and osteoporosis
Neurogenic shock clinical manifestations
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Hypotension, bradycardia, temperature dysregulation, dry skin,
poikilothermia
Cystitis
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inflammation of the bladder
-Sx: urgency, frequency, pain in the bladder/pelvis
- Stress management techniques, tricyclic antidepressants and OTCs help
with burning pain and urinary frequency; avoid clothing that creates
suprapubic pressure
Obstructive shock treatment
Give this one a try later!
Give this one a try later!
-Drug of choice is epinephrine
-Maintaining airway is critical
-Aggressive fluid replacement
,Obstructive shock
Give this one a try later!
Physical obstruction to blood flow occurs with a decreased CO
Phases of AKI
Give this one a try later!
Oliguric, diuretic, recovery
Compensatory stage of shock
Give this one a try later!
-clinically apparent and involves neural, hormonal, and biochemical
compensatory mechanisms in an attempt to overcome the increasing
consequences of anaerobic metabolism and to maintain homeostasis
- decreased urine output
-increased HR
-Increased glucose
Urinary Incontinence management
Give this one a try later!
, -Adequate volume of fluids and reduction or elimination of bladder irritants
from the diet
-Voiding regimens, bladder retraining, pelvic floor muscle training
-Drug therapy, devices, surgery
Electrolyte changes in CKD
Give this one a try later!
High: Sodium, potassium, phosphorus
Low: Calcium, Vitamin D
hemodialysis (HD)
Give this one a try later!
-treatment for renal failure using artificial kidney machine to filter waste
from blood
- Complications: hypotension, muscle cramps, blood loss
Manifestations of Addison's disease
Give this one a try later!
-slow onset
-weakness, weight loss, anorexia
-skin hyperpigmentation
- hypoglycemia
, Cushing's disease manifestations
Give this one a try later!
-Central obesity, moon face, purple striae, buffalo hump, hirsutism in
women, hyperglycemia, hypertension, hypokalemia, and osteoporosis
Neurogenic shock clinical manifestations
Give this one a try later!
Hypotension, bradycardia, temperature dysregulation, dry skin,
poikilothermia
Cystitis
Give this one a try later!
inflammation of the bladder
-Sx: urgency, frequency, pain in the bladder/pelvis
- Stress management techniques, tricyclic antidepressants and OTCs help
with burning pain and urinary frequency; avoid clothing that creates
suprapubic pressure
Obstructive shock treatment
Give this one a try later!