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NURS245 Unit2 Questions and Answers
(Expert Solutions)
Q: First line of defense, 🗹🗹: Mechanical barriers like skin and mucous membranes.
Q: Second line of defense, 🗹🗹: Includes phagocytosis, inflammation, and interferon.
Q: Third line of defense, 🗹🗹: Production of specific antibodies during immune
response.
Q: Inflammation, 🗹🗹: Protective mechanism indicating underlying problems.
Q: Suffix -itis, 🗹🗹: Indicates inflammation in medical terminology.
Q: Signs of inflammation, 🗹🗹: Serve as warnings for potential health issues.
Q: Causes of inflammation, 🗹🗹: Includes physical damage, allergic reactions, and
infections.
Q: Bradykinin, 🗹🗹: Chemical released from injured cells activating pain receptors.
Q: Histamine, 🗹🗹: Released by mast cells, causing capillary dilation.
Q: Neutrophils, 🗹🗹: White blood cells that phagocytize bacteria.
Q: Macrophages, 🗹🗹: Leave bloodstream for phagocytosis of microbes.
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Q: Chemical mediators, 🗹🗹: Substances driving inflammation and immune response.
Q: Vascular phase, 🗹🗹: Involves vasoconstriction, vasodilation, and permeability
changes.
Q: Cellular phase, 🗹🗹: Involves chemotaxis, margination, and phagocytosis.
Q: Cardinal signs of inflammation, 🗹🗹: Rubor, tumor, calor, dolor, functio laesa.
Q: Rubor, 🗹🗹: Redness caused by increased blood flow.
Q: Tumor, 🗹🗹: Swelling due to fluid shift into tissues.
Q: Calor, 🗹🗹: Warmth from increased blood flow to injury.
Q: Dolor, 🗹🗹: Pain from pressure on nerves and mediators.
Q: Functio laesa, 🗹🗹: Loss of function due to nutrient lack and edema.
Q: Pyrexia, 🗹🗹: Fever caused by extensive inflammation.
Q: Interleukins, 🗹🗹: Type of pyrogen released by leukocytes.
Q: Fever patterns, 🗹🗹: Include intermittent, sustained, remittent, and relapsing.
Q: Malaise, 🗹🗹: General feeling of unwellness due to inflammation.
Q: Fever, 🗹🗹: Temporary increase in body temperature.
NURS245 Unit2 Questions and Answers
(Expert Solutions)
Q: First line of defense, 🗹🗹: Mechanical barriers like skin and mucous membranes.
Q: Second line of defense, 🗹🗹: Includes phagocytosis, inflammation, and interferon.
Q: Third line of defense, 🗹🗹: Production of specific antibodies during immune
response.
Q: Inflammation, 🗹🗹: Protective mechanism indicating underlying problems.
Q: Suffix -itis, 🗹🗹: Indicates inflammation in medical terminology.
Q: Signs of inflammation, 🗹🗹: Serve as warnings for potential health issues.
Q: Causes of inflammation, 🗹🗹: Includes physical damage, allergic reactions, and
infections.
Q: Bradykinin, 🗹🗹: Chemical released from injured cells activating pain receptors.
Q: Histamine, 🗹🗹: Released by mast cells, causing capillary dilation.
Q: Neutrophils, 🗹🗹: White blood cells that phagocytize bacteria.
Q: Macrophages, 🗹🗹: Leave bloodstream for phagocytosis of microbes.
, Page | 2
Q: Chemical mediators, 🗹🗹: Substances driving inflammation and immune response.
Q: Vascular phase, 🗹🗹: Involves vasoconstriction, vasodilation, and permeability
changes.
Q: Cellular phase, 🗹🗹: Involves chemotaxis, margination, and phagocytosis.
Q: Cardinal signs of inflammation, 🗹🗹: Rubor, tumor, calor, dolor, functio laesa.
Q: Rubor, 🗹🗹: Redness caused by increased blood flow.
Q: Tumor, 🗹🗹: Swelling due to fluid shift into tissues.
Q: Calor, 🗹🗹: Warmth from increased blood flow to injury.
Q: Dolor, 🗹🗹: Pain from pressure on nerves and mediators.
Q: Functio laesa, 🗹🗹: Loss of function due to nutrient lack and edema.
Q: Pyrexia, 🗹🗹: Fever caused by extensive inflammation.
Q: Interleukins, 🗹🗹: Type of pyrogen released by leukocytes.
Q: Fever patterns, 🗹🗹: Include intermittent, sustained, remittent, and relapsing.
Q: Malaise, 🗹🗹: General feeling of unwellness due to inflammation.
Q: Fever, 🗹🗹: Temporary increase in body temperature.
