TEST BANK $vb
Advanced Physiology and Pathophysiology: Essentials for Clinical
$vb $vb $vb $vb $vb $vb
Practice by Nancy Tkacs, Linda Herrmann
$vb $vb $vb $vb $vb
2nd Edition
$vb
,Test Bank for Advanced Physiology and Pathophysiology: Essentials for
Clinical Practice 2nd Edition
Chapter 1. THE FOUNDATIONAL CONCEPTS OF CLINICALPRACTICE
1. The characteristic, localized cardinal signs of acute inflammation include:
A) fever.
B) fatigue.
C) redness.
D) granuloma.
2. The vascular, hemodynamic stage of acute inflammation is initiated by
momentary vasoconstriction followed by vasodilation that causes localized:
A) bleeding.
B) congestion.
C) pale skin.
D) coolness.
3. The cellular stage of acute inflammation is marked by the movement of
leukocytes into the area. Which of the following cells arrives early in great
numbers?
A) Basophils
B) Lymphocytes
C) Neutrophils
D) Platelets
,4. The $vbphagocytosis $vbprocess $vbinvolves $vbthree $vbdistinct $vbsteps. $vbWhat $vbis
$vbthe $vbinitial $vbstep $vbin $vbthe $vbprocess?
A) Engulfment
B) Intracellular $vbkilling
C) Antigen $vbmargination
D) Recognition $vband $vb adherence
5. Which $vbof $vbthe $vbfollowing $vbmediators $vbof $vbinflammation $vbcauses $vbincreased
$vbcapillary $vbpermeability $vband $vbpain?
A) Serotonin
B) Histamine
C) Bradykinin
D) Nitric $vboxide
6. Inflammatory $vbexudates $vbare $vba $vbcombination $vbof $vbseveral $vbtypes.
$vbWhich $vbof $vbthe $vbfollowing $vbexudates $vbis $vbcomposed $vbof $vbenmeshed
$vbnecrotic $vbcells?
A) Serous
B) Fibrinous
C) Suppurative
D) Membranous
7. The $vbacute-phase $vbsystemic $vbresponse $vbusually $vbbegins $vbwithin $vbhours $vbof
$vbthe $vbonset $vbof $vbinflammation $vband $vbincludes:
A) fever $vband $vb lethargy.
B) decreased $vbC-reactive $vbprotein.
C) positive $vbnitrogen $vbbalance.
D) low $vberythrocyte $vbsedimentation $vbrate.
, 8. In $vbcontrast $vbto $vbacute $vbinflammation, $vbchronic $vbinflammation $vbis
$vbcharacterized $vbby $vbwhich $vbof $vbthe $vbfollowing $vbphenomena?
A) Profuse $vbfibrinous $vbexudation
B) A $vbshift $vb to $vbthe $vbleft $ v b of $vbgranulocytes
C) Metabolic $vband $vbrespiratory $vbalkalosis
D) Lymphocytosis $vband $vbactivated $vb macrophages
9. Exogenous $vbpyrogens $vb(interleukin-1) $vband $vbthe $vbpresence $vbof $vbbacteria $vbin
$vbthe $vbblood $vblead $vbto $vbthe $vbrelease $vbof $vbendogenous $vbpyrogens $vbthat:
A) stabilize $vbthermal $vbcontrol $vbin $vbthe $vb brain.
B) produce $vbleukocytosis $vb and $vbanorexia.
C) block $vbviral $vbreplication $vbin $vbcells.
D) inhibit $vbprostaglandin $vbrelease.
10 An $vbolder $vbadult $vbpatient $vb has $vbjust $vbsheared $vbthe $vbskin $vbon $vbher $vbelbow
$vb while $vb attempted
. to $vbboost $vbherself $vbup $vbin $vbbed, $vban $vbevent $vbthat $vbhas $vbprecipitated $vbacute
$vbinflammation $vbin $vbthe $vbregion $vbsurrounding $vbthe $vbwound. $vbWhich $vbof
$vbthe $vbfollowing $vbevents $vbwill $vboccur $vbduring $vbthe $vbvascular $vbstage $vbof
$vbthe $vbpatients $vbinflammation?
A) Outpouring $vbof $vbexudate $vbinto $vbinterstitial $vbspaces
B) Chemotaxis
C) Accumulation $vbof $vbleukocytes $vbalong $vbthe $vbepithelium
D) Phagocytosis $vbof $vbcellular $vbdebris
11 Which $vbof $vbthe $vb following $vbindividuals $vbmost $vb likely $vbhas $vbthe $ vb highest $vbrisk
$vbof
. experiencing $vbchronic $vb inflammation?
A) A $vbpatient $vb who $vbhas $vbrecently $vbbeen $vbdiagnosed $vb with $vbtype $vb 2 $vbdiabetes
B) A $vbpatient $vbwho $vbis $vba $vbcarrier $vbof $vban $vbantibiotic-resistant $vb organism
C) A $vbpatient $vbwho $vb is $vbtaking $vb oral $vbantibiotics $v b for $vb an $vbupper $vbrespiratory
$vbinfection
Advanced Physiology and Pathophysiology: Essentials for Clinical
$vb $vb $vb $vb $vb $vb
Practice by Nancy Tkacs, Linda Herrmann
$vb $vb $vb $vb $vb
2nd Edition
$vb
,Test Bank for Advanced Physiology and Pathophysiology: Essentials for
Clinical Practice 2nd Edition
Chapter 1. THE FOUNDATIONAL CONCEPTS OF CLINICALPRACTICE
1. The characteristic, localized cardinal signs of acute inflammation include:
A) fever.
B) fatigue.
C) redness.
D) granuloma.
2. The vascular, hemodynamic stage of acute inflammation is initiated by
momentary vasoconstriction followed by vasodilation that causes localized:
A) bleeding.
B) congestion.
C) pale skin.
D) coolness.
3. The cellular stage of acute inflammation is marked by the movement of
leukocytes into the area. Which of the following cells arrives early in great
numbers?
A) Basophils
B) Lymphocytes
C) Neutrophils
D) Platelets
,4. The $vbphagocytosis $vbprocess $vbinvolves $vbthree $vbdistinct $vbsteps. $vbWhat $vbis
$vbthe $vbinitial $vbstep $vbin $vbthe $vbprocess?
A) Engulfment
B) Intracellular $vbkilling
C) Antigen $vbmargination
D) Recognition $vband $vb adherence
5. Which $vbof $vbthe $vbfollowing $vbmediators $vbof $vbinflammation $vbcauses $vbincreased
$vbcapillary $vbpermeability $vband $vbpain?
A) Serotonin
B) Histamine
C) Bradykinin
D) Nitric $vboxide
6. Inflammatory $vbexudates $vbare $vba $vbcombination $vbof $vbseveral $vbtypes.
$vbWhich $vbof $vbthe $vbfollowing $vbexudates $vbis $vbcomposed $vbof $vbenmeshed
$vbnecrotic $vbcells?
A) Serous
B) Fibrinous
C) Suppurative
D) Membranous
7. The $vbacute-phase $vbsystemic $vbresponse $vbusually $vbbegins $vbwithin $vbhours $vbof
$vbthe $vbonset $vbof $vbinflammation $vband $vbincludes:
A) fever $vband $vb lethargy.
B) decreased $vbC-reactive $vbprotein.
C) positive $vbnitrogen $vbbalance.
D) low $vberythrocyte $vbsedimentation $vbrate.
, 8. In $vbcontrast $vbto $vbacute $vbinflammation, $vbchronic $vbinflammation $vbis
$vbcharacterized $vbby $vbwhich $vbof $vbthe $vbfollowing $vbphenomena?
A) Profuse $vbfibrinous $vbexudation
B) A $vbshift $vb to $vbthe $vbleft $ v b of $vbgranulocytes
C) Metabolic $vband $vbrespiratory $vbalkalosis
D) Lymphocytosis $vband $vbactivated $vb macrophages
9. Exogenous $vbpyrogens $vb(interleukin-1) $vband $vbthe $vbpresence $vbof $vbbacteria $vbin
$vbthe $vbblood $vblead $vbto $vbthe $vbrelease $vbof $vbendogenous $vbpyrogens $vbthat:
A) stabilize $vbthermal $vbcontrol $vbin $vbthe $vb brain.
B) produce $vbleukocytosis $vb and $vbanorexia.
C) block $vbviral $vbreplication $vbin $vbcells.
D) inhibit $vbprostaglandin $vbrelease.
10 An $vbolder $vbadult $vbpatient $vb has $vbjust $vbsheared $vbthe $vbskin $vbon $vbher $vbelbow
$vb while $vb attempted
. to $vbboost $vbherself $vbup $vbin $vbbed, $vban $vbevent $vbthat $vbhas $vbprecipitated $vbacute
$vbinflammation $vbin $vbthe $vbregion $vbsurrounding $vbthe $vbwound. $vbWhich $vbof
$vbthe $vbfollowing $vbevents $vbwill $vboccur $vbduring $vbthe $vbvascular $vbstage $vbof
$vbthe $vbpatients $vbinflammation?
A) Outpouring $vbof $vbexudate $vbinto $vbinterstitial $vbspaces
B) Chemotaxis
C) Accumulation $vbof $vbleukocytes $vbalong $vbthe $vbepithelium
D) Phagocytosis $vbof $vbcellular $vbdebris
11 Which $vbof $vbthe $vb following $vbindividuals $vbmost $vb likely $vbhas $vbthe $ vb highest $vbrisk
$vbof
. experiencing $vbchronic $vb inflammation?
A) A $vbpatient $vb who $vbhas $vbrecently $vbbeen $vbdiagnosed $vb with $vbtype $vb 2 $vbdiabetes
B) A $vbpatient $vbwho $vbis $vba $vbcarrier $vbof $vban $vbantibiotic-resistant $vb organism
C) A $vbpatient $vbwho $vb is $vbtaking $vb oral $vbantibiotics $v b for $vb an $vbupper $vbrespiratory
$vbinfection
