,Pathophysiology:nAnPracticalnApproach:nAnPracticalnApproachn5th Edition
StorynTestnBank
Chapter 1 Cellular Function
1. Ann increasen inn cardiacn sizen andn functionn duen ton increasedn workloadn isn termed
A) Atrophy.
B) Functional.
C) Hypertrophy.
D) Inflammation.
2. WhilenassessingnanpatientndiagnosednwithnMarfan'snSyndrome,nthennursenshouldninclud
enwhichnofnthenfollowingnthatnisnconsistentnwithnthensyndrome?
A) Cardiacn assessmentn forn coarctationn ofn then aorta.
B) Genitaln assessmentn forn smalln testicles.
C) Mentaln assessmentn forn impairment.
D) Oraln assessmentn forn cleftn palate.
3. ChildrennwithnPKUnmustnavoidnphenylalanineninnthendiet.nPhenylalaninenisnmostnlike
lyntonbenancomponentnof
A) Fat.
B) Sugar.
C) Protein.
D) Carbohydrate.
4. An17-year-oldncollege-
boundnstudentnreceivesnanvaccinenagainstnannorganismnthatncausesnmeningitis.nThisnisna
nnexamplenof
A) primaryn prevention.
B) secondaryn prevention.
C) tertiaryn prevention.
D) diseasen treatment.
5. Metaplasianis
A) Then disorganizationn ofn cellsn inton variousn sizes,n shapes,n andn arrangements.
B) Then replacementn ofn onen differentiatedn celln typen withn another.
C) Then transformationn ofn an celln typen ton malignancy.
D) Ann irreversiblen cellularn adaptation.
, 6. Duringnanroutinenultrasoundnofnan38nyearnoldnwomenn26nweeksngestationnrevealsnanfet
usnwithnansmallnsquarenhead,nupwardnslantnofntheneyes,nandnlownsetnears.nThennursenre
cognizesnthatnthesenfindingsnarenconsistentnwithnwhichnofnthenfollowing?
A) Fragilen Xn Syndrome.
B) Monosomyn Xn (Turner'sn Syndrome).
C) Trisomyn 21n (Down'sn Syndrome).
D) Trisomyn Xn (Klinefelter'sn Syndrome).
7. WhilendiscussingntreatmentnoptionsnwithnanparentnofnannewlyndiagnosednMonosomyn
Xn(Turner'snSyndrome)nchild,nthennursenshouldnincludenwhichnofnthenfollowing?
A) Theren isn nontreatmentn orn cure.
B) Symptomsn ofn then conditionn aren treatedn withn estrogen.
C) Symptomsn ofn then conditionn aren treatedn withn testosterone.
D) Institutionalizationn isn then preferredn methodn ofn managingn care.
8. Whichnofnthenfollowingnassessmentnfindingsnindicatesnannalterationninnhomeostaticncont
rolnmechanisms?
A) Fever
B) Throatn pain
C) Jointn stiffness
D) Positiven throatn culture
9. Whatninformationnshouldnparentsnbengivennaboutnthenconsequencesnofnphenylketonu
rian(PKU)?
A) Mentaln retardationn isn inevitable.
B) PKUn isn commonlyn associatedn withn othern congenitaln anomalies.
C) Highn dietaryn tyraminen mayn helpn inducen enzymen production.
D) Failuren ton treatn properlyn resultsn inn progressiven mentaln retardation.
10. Injurynthatn occursn whenn bloodn flownisn diminishedn tontissuen isncalled injury.
A) hypoxic
B) ischemic
C) hyperemic
D) neoplastic
11. Tayn sachsn isncausedn byn whichn ofnthen following?
A) Andeficiencyn orn absencen ofn hexosaminidasenA
B) Andefectn onn chromosomen 17n orn 22
C) Anmutationn onn chromosomen 15
, D) Ann errorn inn convertingn phenylalaninen ton tyrosine
12. Annobesenbutnotherwisenhealthynteenngoesntonanhealthnfairnandnhasnhernbloodnpres
surenchecked.nThisnisnannexamplenof
A) primaryn prevention.
B) secondaryn prevention.
C) tertiaryn prevention.
D) diseasen treatment.
13. Characteristicsn ofn X-linkedn recessiven disordersn includen whichn ofn then following?
A) Then sonn ofn an carriern mothern hasnan 25%n chancen ofn beingn affected.
B) Affectedn fathersn transmitn then genenton alln ofntheirn sons.
C) Alln daughtersn ofn affectedn fathersn aren carriers.
D) Boysn andn girlsn aren equallyn affected.
14. Anfactorn associatedn withn riskn ofn Downn syndromen is
A) Maternalnage.
B) Maternaln alcoholn intake.
C) Familyn historyn ofn heritablen diseases.
D) Exposuren ton TORCHn syndromen organisms.
15. Whichn typen ofn gangrenen isn usuallyn an resultn ofn arterialn occlusion?
A) Necrosis.
B) Dry.
C) Wet.
D) Gas.
16. Then cancern growthn continuumn isn dividedn inton then followingn stages.
A) Stagen 1,nStagen 2,nStagen 3
B) Initiation,n Progression,n Promotion
C) Preliminary,nEvolutionary,nMetastasis
D) Initiation,n Promotion,n Progression
17. Andiseaseninnwhichnthenprincipalnmanifestationnisnannabnormalngrowthnofncellsnle
adingntonformationnofntumorsnisncalledna disease.
A) congenital
B) degenerative
C) metabolic
StorynTestnBank
Chapter 1 Cellular Function
1. Ann increasen inn cardiacn sizen andn functionn duen ton increasedn workloadn isn termed
A) Atrophy.
B) Functional.
C) Hypertrophy.
D) Inflammation.
2. WhilenassessingnanpatientndiagnosednwithnMarfan'snSyndrome,nthennursenshouldninclud
enwhichnofnthenfollowingnthatnisnconsistentnwithnthensyndrome?
A) Cardiacn assessmentn forn coarctationn ofn then aorta.
B) Genitaln assessmentn forn smalln testicles.
C) Mentaln assessmentn forn impairment.
D) Oraln assessmentn forn cleftn palate.
3. ChildrennwithnPKUnmustnavoidnphenylalanineninnthendiet.nPhenylalaninenisnmostnlike
lyntonbenancomponentnof
A) Fat.
B) Sugar.
C) Protein.
D) Carbohydrate.
4. An17-year-oldncollege-
boundnstudentnreceivesnanvaccinenagainstnannorganismnthatncausesnmeningitis.nThisnisna
nnexamplenof
A) primaryn prevention.
B) secondaryn prevention.
C) tertiaryn prevention.
D) diseasen treatment.
5. Metaplasianis
A) Then disorganizationn ofn cellsn inton variousn sizes,n shapes,n andn arrangements.
B) Then replacementn ofn onen differentiatedn celln typen withn another.
C) Then transformationn ofn an celln typen ton malignancy.
D) Ann irreversiblen cellularn adaptation.
, 6. Duringnanroutinenultrasoundnofnan38nyearnoldnwomenn26nweeksngestationnrevealsnanfet
usnwithnansmallnsquarenhead,nupwardnslantnofntheneyes,nandnlownsetnears.nThennursenre
cognizesnthatnthesenfindingsnarenconsistentnwithnwhichnofnthenfollowing?
A) Fragilen Xn Syndrome.
B) Monosomyn Xn (Turner'sn Syndrome).
C) Trisomyn 21n (Down'sn Syndrome).
D) Trisomyn Xn (Klinefelter'sn Syndrome).
7. WhilendiscussingntreatmentnoptionsnwithnanparentnofnannewlyndiagnosednMonosomyn
Xn(Turner'snSyndrome)nchild,nthennursenshouldnincludenwhichnofnthenfollowing?
A) Theren isn nontreatmentn orn cure.
B) Symptomsn ofn then conditionn aren treatedn withn estrogen.
C) Symptomsn ofn then conditionn aren treatedn withn testosterone.
D) Institutionalizationn isn then preferredn methodn ofn managingn care.
8. Whichnofnthenfollowingnassessmentnfindingsnindicatesnannalterationninnhomeostaticncont
rolnmechanisms?
A) Fever
B) Throatn pain
C) Jointn stiffness
D) Positiven throatn culture
9. Whatninformationnshouldnparentsnbengivennaboutnthenconsequencesnofnphenylketonu
rian(PKU)?
A) Mentaln retardationn isn inevitable.
B) PKUn isn commonlyn associatedn withn othern congenitaln anomalies.
C) Highn dietaryn tyraminen mayn helpn inducen enzymen production.
D) Failuren ton treatn properlyn resultsn inn progressiven mentaln retardation.
10. Injurynthatn occursn whenn bloodn flownisn diminishedn tontissuen isncalled injury.
A) hypoxic
B) ischemic
C) hyperemic
D) neoplastic
11. Tayn sachsn isncausedn byn whichn ofnthen following?
A) Andeficiencyn orn absencen ofn hexosaminidasenA
B) Andefectn onn chromosomen 17n orn 22
C) Anmutationn onn chromosomen 15
, D) Ann errorn inn convertingn phenylalaninen ton tyrosine
12. Annobesenbutnotherwisenhealthynteenngoesntonanhealthnfairnandnhasnhernbloodnpres
surenchecked.nThisnisnannexamplenof
A) primaryn prevention.
B) secondaryn prevention.
C) tertiaryn prevention.
D) diseasen treatment.
13. Characteristicsn ofn X-linkedn recessiven disordersn includen whichn ofn then following?
A) Then sonn ofn an carriern mothern hasnan 25%n chancen ofn beingn affected.
B) Affectedn fathersn transmitn then genenton alln ofntheirn sons.
C) Alln daughtersn ofn affectedn fathersn aren carriers.
D) Boysn andn girlsn aren equallyn affected.
14. Anfactorn associatedn withn riskn ofn Downn syndromen is
A) Maternalnage.
B) Maternaln alcoholn intake.
C) Familyn historyn ofn heritablen diseases.
D) Exposuren ton TORCHn syndromen organisms.
15. Whichn typen ofn gangrenen isn usuallyn an resultn ofn arterialn occlusion?
A) Necrosis.
B) Dry.
C) Wet.
D) Gas.
16. Then cancern growthn continuumn isn dividedn inton then followingn stages.
A) Stagen 1,nStagen 2,nStagen 3
B) Initiation,n Progression,n Promotion
C) Preliminary,nEvolutionary,nMetastasis
D) Initiation,n Promotion,n Progression
17. Andiseaseninnwhichnthenprincipalnmanifestationnisnannabnormalngrowthnofncellsnle
adingntonformationnofntumorsnisncalledna disease.
A) congenital
B) degenerative
C) metabolic
