PATH 1017 W1 AND W2 WITH COMPLETE SOLUTIONS 100%
VERIFIED!!
Etiology - ANSWER cause of disease
Pathological process - ANSWER Mechanism of diseases/ illness, it is the
manifestations and symptoms that the body response, it shows what has gone wrong
due to the disease.
interventions - ANSWER actions taken to correct or manage a patient's problems, by
diagnostic test and therapeutic method
Shock/Circulatory Failure means there is a critical? - ANSWER decrease in tissue
perfusion, difficulty of the heart to push blood out throughout the body.
Cardiac Output is the amount? - ANSWER of blood pumped out of the heart per min
Normal cardiac output is? - ANSWER 4-8 liters/min
Stroke volume is the? - ANSWER blood being pushed out of the heart per beat or per
cardiac cycle.
average stroke volume is? - ANSWER 70-80 ml/beat
Mean Arterial Pressure (MAP) is the pressure that? - ANSWER propels blood to tissues
Decreased MAP causes a decrease ? - ANSWER tissue perfusion
,Systemic Vascular Resistance (SVR) is the resistance that blood must? - ANSWER
overcome to be able to move within the blood vessels. SVR is related to the amount of
dilation or constriction in the blood vessel.
peripheral vascular resistance is the force? - ANSWER exerted against the blood flow
and is determined by the diameter of the vessel.
the lower the vascular resistance the less? - ANSWER force is needed to eject the
blood out of the heart during systole.
Shock is when? - ANSWER tissue perfusion is inadequate to deliver O2 and nutrients to
support the vital organs and cellular functions
Inadequate tissue perfusion can caused? - ANSWER cells not to get enough nutritions
leading them to fail in function, get injured and die.
Pathophysiology of Shock in the cellular level includes changes in the? - ANSWER
metabolism due to hypoperfusion, that leads to ATP product being affected. and causes
cells to get injured and die.
What byproduct is produced when not enough ATP is produced? - ANSWER lactic acid
that causes muscle soreness and fatigue
Pathophysiology of Shock in the cellular level leads to? - ANSWER NA-K pump that
becomes impaired, causing water retention that results complications like edema,
pulmonary edema.
Aerobic Metabolism is how our body creates? - ANSWER energy through the
combustion of carbohydrates, amino acids and fat in the presence of oxygen.
Anaerobic Metabolism is the creation of? - ANSWER ATP by the combustion of
carbohydrates in the absence of o2.
, Our body engage in aerobic metabolism when we have? - ANSWER adequate o2
present that allows ATP tp be made.
Goals of Compensation Mechanism is to maintain? - ANSWER BP and CO by SNS and
RAA
SNS mechanism maintains? - ANSWER BP and perfusion, it is where epi is released
and alpha, beta 2 are activated that leads to rapid HR and vasoconstriction to maintain.
renin-angiotensin-aldosterone mechanism is where? - ANSWER angiotensin are
released, it causes vasoconstriction that helps maintain BP and releases aldosterone to
increase sodium to be reabsorbed.
Pathophysiology of Compensatory Shock? - ANSWER activated in response to low
perfusion.
SS of compensatory shock includes? - ANSWER increased HR, blood glucose,RR, Cool
clammy skin, decreased bowel sounds, urine output and metabolic acidosis and pt
feeling anxious or confused
Prognosis of Compensatory Shock is? - ANSWER good with early intervention and
correction of cause
Pathophysiology of Progressive shock is when? - ANSWER compensation mechanism
can't keep up, hypoperfusion worsens, cell damage.
SS of Progressive shock includes? - ANSWER decreased BP, urine output, LOC , HR
increases, cardiac schema dysrhythmias, hypoxia, pulmonary edema, and altered liver
GI, hematological.
Prognosis of Progressive shock worsens? - ANSWER depending on time to restore
VERIFIED!!
Etiology - ANSWER cause of disease
Pathological process - ANSWER Mechanism of diseases/ illness, it is the
manifestations and symptoms that the body response, it shows what has gone wrong
due to the disease.
interventions - ANSWER actions taken to correct or manage a patient's problems, by
diagnostic test and therapeutic method
Shock/Circulatory Failure means there is a critical? - ANSWER decrease in tissue
perfusion, difficulty of the heart to push blood out throughout the body.
Cardiac Output is the amount? - ANSWER of blood pumped out of the heart per min
Normal cardiac output is? - ANSWER 4-8 liters/min
Stroke volume is the? - ANSWER blood being pushed out of the heart per beat or per
cardiac cycle.
average stroke volume is? - ANSWER 70-80 ml/beat
Mean Arterial Pressure (MAP) is the pressure that? - ANSWER propels blood to tissues
Decreased MAP causes a decrease ? - ANSWER tissue perfusion
,Systemic Vascular Resistance (SVR) is the resistance that blood must? - ANSWER
overcome to be able to move within the blood vessels. SVR is related to the amount of
dilation or constriction in the blood vessel.
peripheral vascular resistance is the force? - ANSWER exerted against the blood flow
and is determined by the diameter of the vessel.
the lower the vascular resistance the less? - ANSWER force is needed to eject the
blood out of the heart during systole.
Shock is when? - ANSWER tissue perfusion is inadequate to deliver O2 and nutrients to
support the vital organs and cellular functions
Inadequate tissue perfusion can caused? - ANSWER cells not to get enough nutritions
leading them to fail in function, get injured and die.
Pathophysiology of Shock in the cellular level includes changes in the? - ANSWER
metabolism due to hypoperfusion, that leads to ATP product being affected. and causes
cells to get injured and die.
What byproduct is produced when not enough ATP is produced? - ANSWER lactic acid
that causes muscle soreness and fatigue
Pathophysiology of Shock in the cellular level leads to? - ANSWER NA-K pump that
becomes impaired, causing water retention that results complications like edema,
pulmonary edema.
Aerobic Metabolism is how our body creates? - ANSWER energy through the
combustion of carbohydrates, amino acids and fat in the presence of oxygen.
Anaerobic Metabolism is the creation of? - ANSWER ATP by the combustion of
carbohydrates in the absence of o2.
, Our body engage in aerobic metabolism when we have? - ANSWER adequate o2
present that allows ATP tp be made.
Goals of Compensation Mechanism is to maintain? - ANSWER BP and CO by SNS and
RAA
SNS mechanism maintains? - ANSWER BP and perfusion, it is where epi is released
and alpha, beta 2 are activated that leads to rapid HR and vasoconstriction to maintain.
renin-angiotensin-aldosterone mechanism is where? - ANSWER angiotensin are
released, it causes vasoconstriction that helps maintain BP and releases aldosterone to
increase sodium to be reabsorbed.
Pathophysiology of Compensatory Shock? - ANSWER activated in response to low
perfusion.
SS of compensatory shock includes? - ANSWER increased HR, blood glucose,RR, Cool
clammy skin, decreased bowel sounds, urine output and metabolic acidosis and pt
feeling anxious or confused
Prognosis of Compensatory Shock is? - ANSWER good with early intervention and
correction of cause
Pathophysiology of Progressive shock is when? - ANSWER compensation mechanism
can't keep up, hypoperfusion worsens, cell damage.
SS of Progressive shock includes? - ANSWER decreased BP, urine output, LOC , HR
increases, cardiac schema dysrhythmias, hypoxia, pulmonary edema, and altered liver
GI, hematological.
Prognosis of Progressive shock worsens? - ANSWER depending on time to restore
