TEST BANK FOR RUBIN’S PATHOLOGY: CL
v v v v vv
INICOPATHOLOGIC FOUNDATIONS OF ME v v vv
DICINE 7TH EDITION ( WITH ANSWER KEY)
v v v v v vv
LATEST UPDATED EXAMINATION STUDY G
v v v vv
UIDE 2023 v
GUARANTEED GRADE A+ v v
Chapterv 1:v Cellv Adaptation,v Injuryv and v Death
vIschemiav and v otherv toxicv injuriesv increasev thev accumulationv of v intracellularv calciumv asv av result
1. of:
A) releasev of v stored v calciumv fromv thev mitochondria.
B) improved v intracellularv volumevregulation.
C) decreased v influxv acrossv thev cellvmembrane.
D) attractionv of v calciumv tov fattyv infiltrates.
Thev patient v isv found v tov havev liverv disease,v resultingv inv thev removalv of v av lobev of v hisv liver.
2. Adaptationv tov thev reduced v sizev of vthev liverv leadsv tov _ of vthev remainingv liverv cells.
A) metaplasia
B) organv atrophy
C) compensatoryvhyperplasia
D) physiologicv hypertrophy
Av personv eatingv peanutsv startsv chokingv andvcollapses.v Hisv airwayv obstructionv isv partiallyv cleared
,v but v hev remainsv hypoxicv untilv hev reachesv thev hospital.v Thev prolonged v cellv hypoxia
3. caused v av cerebralv infarctionvand v resulting _v inv thev brain.
A) caspasev activation
B) coagulationv necrosis
C) rapid vphagocytosis
D) proteinv p53v deficiency
Bacteriavand v virusesv causev cellv damagev by ,v whichv isv uniquev fromv thev intracellular
4. damagev caused v byv otherv injuriousv agents.
A) disruptingvthev sodium/potassiumv ATPasev pump
B) interruptingv oxidativev metabolismv processes
C) replicatingv and v producingv continued v injury
,TEST BANK FOR RUBIN’S PATHOLOGY: CL
v v v v vv
INICOPATHOLOGIC FOUNDATIONS OF ME v v vv
DICINE 7TH EDITION ( WITH ANSWER KEY)
v v v v v vv
LATEST UPDATED EXAMINATION STUDY G
v v v vv
UIDE 2023
v
GUARANTEED GRADE A+ v v
D) decreasingv proteinvsynthesisv and v function
Thevpatient v hasv av prolonged v interruptionv invarterialvblood v flow v tov hisv left v kidney,v causing
5. hypoxicv cellv injuryvand v thev releasev of v freev radicals.v Freev radicalsv damagev cellsv by:
A) destroyingv phospholipidsv inv thev cellv membrane.
B) alteringv thev immunev responsev of v thev cell.
C) disruptingv calciumv storagev inv thev cell.
D) inactivationv of venzymesv and v mitochondria.
,TEST BANK FOR RUBIN’S PATHOLOGY: CL
v v v v vv
INICOPATHOLOGIC FOUNDATIONS OF ME v v vv
DICINE 7TH EDITION ( WITH ANSWER KEY)
v v v v v vv
LATEST UPDATED EXAMINATION STUDY G
v v v vv
UIDE 2023
v
GUARANTEED GRADE A+ v v
6. Injured v cellsv havev impaired v flow v of vsubstancesv throughv thev cellv membranev asv av result v of:
A) increased v fat v load.
B) altered vpermeability.
C) altered v glucosevutilization.
D) increased v surfacev receptors.
7. Reversiblev adaptivev intracellularv responsesv arev initiated v by:
A) stimulusv overload.
B) geneticvmutations.
C) chemicalvmessengers.
D) mitochondrialvDNA.
8. Injured v cellsv becomev veryvswollenv asv av result v of:
A) increased v cellv proteinvsynthesis.
B) altered v cellv volumevregulation.
C) passivev entryv of vpotassiumv intov thev cell.
D) blebv formationv inv thev plasmav membrane.
Av diabeticv patient vhasv impaired v sensation,vcirculation,v and voxygenationvof vhisv feet.vHev stepsv onvav
piecev of v glass,v thev wound v doesv not v heal,v and v thev areav tissuev becomesv necrotic.v Thev necrotic
9. cellvdeathv isv characterized v by:
A) rapid vapoptosis.
B) cellularv rupture.
C) shrinkagev and v collapse.
D) chronicv inflammation.
Av 99-year-old v womanv hasv experienced v thev declinev of v cellv functionv associated v withv age.v A
10. groupv of v theoriesv of v cellularv agingv focusv onv programmed:
A) changesv withvgeneticv influences.
B) eliminationv of vcellv receptorv sites.
C) insufficient v telomerasev enzyme.
, TEST BANK FOR RUBIN’S PATHOLOGY: CL
v v v v vv
INICOPATHOLOGIC FOUNDATIONS OF ME v v vv
DICINE 7TH EDITION ( WITH ANSWER KEY)
v v v v v vv
LATEST UPDATED EXAMINATION STUDY G
v v v vv
UIDE 2023
v
GUARANTEED GRADE A+ v v
D) DNA vmutationv orv faultyvrepair.
Anv 89-year-
old v femalev patient v hasv experienced v significant v decreasesv inv herv mobilityv and vstaminav duringv av3
-
weekv hospitalv stayvforv thevtreatment vof vavfemoralv head vfracture.vWhichv ofv thev followingv phenom
enav most v likelyv accountsv forv thev patientsv decreasev inv musclev function
11. that vunderliesv herv reduced v mobility?
A) Impaired v musclev cellv metabolismv resultingv fromv metaplasia
B) Dysplasiav asv av consequencev of v inflammationv duringv bonev remodeling
C) Disusevatrophyv of v musclev cellsv duringv av prolonged v period v of v immobility
v v v v vv
INICOPATHOLOGIC FOUNDATIONS OF ME v v vv
DICINE 7TH EDITION ( WITH ANSWER KEY)
v v v v v vv
LATEST UPDATED EXAMINATION STUDY G
v v v vv
UIDE 2023 v
GUARANTEED GRADE A+ v v
Chapterv 1:v Cellv Adaptation,v Injuryv and v Death
vIschemiav and v otherv toxicv injuriesv increasev thev accumulationv of v intracellularv calciumv asv av result
1. of:
A) releasev of v stored v calciumv fromv thev mitochondria.
B) improved v intracellularv volumevregulation.
C) decreased v influxv acrossv thev cellvmembrane.
D) attractionv of v calciumv tov fattyv infiltrates.
Thev patient v isv found v tov havev liverv disease,v resultingv inv thev removalv of v av lobev of v hisv liver.
2. Adaptationv tov thev reduced v sizev of vthev liverv leadsv tov _ of vthev remainingv liverv cells.
A) metaplasia
B) organv atrophy
C) compensatoryvhyperplasia
D) physiologicv hypertrophy
Av personv eatingv peanutsv startsv chokingv andvcollapses.v Hisv airwayv obstructionv isv partiallyv cleared
,v but v hev remainsv hypoxicv untilv hev reachesv thev hospital.v Thev prolonged v cellv hypoxia
3. caused v av cerebralv infarctionvand v resulting _v inv thev brain.
A) caspasev activation
B) coagulationv necrosis
C) rapid vphagocytosis
D) proteinv p53v deficiency
Bacteriavand v virusesv causev cellv damagev by ,v whichv isv uniquev fromv thev intracellular
4. damagev caused v byv otherv injuriousv agents.
A) disruptingvthev sodium/potassiumv ATPasev pump
B) interruptingv oxidativev metabolismv processes
C) replicatingv and v producingv continued v injury
,TEST BANK FOR RUBIN’S PATHOLOGY: CL
v v v v vv
INICOPATHOLOGIC FOUNDATIONS OF ME v v vv
DICINE 7TH EDITION ( WITH ANSWER KEY)
v v v v v vv
LATEST UPDATED EXAMINATION STUDY G
v v v vv
UIDE 2023
v
GUARANTEED GRADE A+ v v
D) decreasingv proteinvsynthesisv and v function
Thevpatient v hasv av prolonged v interruptionv invarterialvblood v flow v tov hisv left v kidney,v causing
5. hypoxicv cellv injuryvand v thev releasev of v freev radicals.v Freev radicalsv damagev cellsv by:
A) destroyingv phospholipidsv inv thev cellv membrane.
B) alteringv thev immunev responsev of v thev cell.
C) disruptingv calciumv storagev inv thev cell.
D) inactivationv of venzymesv and v mitochondria.
,TEST BANK FOR RUBIN’S PATHOLOGY: CL
v v v v vv
INICOPATHOLOGIC FOUNDATIONS OF ME v v vv
DICINE 7TH EDITION ( WITH ANSWER KEY)
v v v v v vv
LATEST UPDATED EXAMINATION STUDY G
v v v vv
UIDE 2023
v
GUARANTEED GRADE A+ v v
6. Injured v cellsv havev impaired v flow v of vsubstancesv throughv thev cellv membranev asv av result v of:
A) increased v fat v load.
B) altered vpermeability.
C) altered v glucosevutilization.
D) increased v surfacev receptors.
7. Reversiblev adaptivev intracellularv responsesv arev initiated v by:
A) stimulusv overload.
B) geneticvmutations.
C) chemicalvmessengers.
D) mitochondrialvDNA.
8. Injured v cellsv becomev veryvswollenv asv av result v of:
A) increased v cellv proteinvsynthesis.
B) altered v cellv volumevregulation.
C) passivev entryv of vpotassiumv intov thev cell.
D) blebv formationv inv thev plasmav membrane.
Av diabeticv patient vhasv impaired v sensation,vcirculation,v and voxygenationvof vhisv feet.vHev stepsv onvav
piecev of v glass,v thev wound v doesv not v heal,v and v thev areav tissuev becomesv necrotic.v Thev necrotic
9. cellvdeathv isv characterized v by:
A) rapid vapoptosis.
B) cellularv rupture.
C) shrinkagev and v collapse.
D) chronicv inflammation.
Av 99-year-old v womanv hasv experienced v thev declinev of v cellv functionv associated v withv age.v A
10. groupv of v theoriesv of v cellularv agingv focusv onv programmed:
A) changesv withvgeneticv influences.
B) eliminationv of vcellv receptorv sites.
C) insufficient v telomerasev enzyme.
, TEST BANK FOR RUBIN’S PATHOLOGY: CL
v v v v vv
INICOPATHOLOGIC FOUNDATIONS OF ME v v vv
DICINE 7TH EDITION ( WITH ANSWER KEY)
v v v v v vv
LATEST UPDATED EXAMINATION STUDY G
v v v vv
UIDE 2023
v
GUARANTEED GRADE A+ v v
D) DNA vmutationv orv faultyvrepair.
Anv 89-year-
old v femalev patient v hasv experienced v significant v decreasesv inv herv mobilityv and vstaminav duringv av3
-
weekv hospitalv stayvforv thevtreatment vof vavfemoralv head vfracture.vWhichv ofv thev followingv phenom
enav most v likelyv accountsv forv thev patientsv decreasev inv musclev function
11. that vunderliesv herv reduced v mobility?
A) Impaired v musclev cellv metabolismv resultingv fromv metaplasia
B) Dysplasiav asv av consequencev of v inflammationv duringv bonev remodeling
C) Disusevatrophyv of v musclev cellsv duringv av prolonged v period v of v immobility
