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IBHRE CEPS 2025: LATEST UPDATED GUARANTEED PASS!!

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IBHRE CEPS 2025: LATEST UPDATED GUARANTEED PASS!! 1.Class 1 Antiarrhythmics - ANSWER block the rapid sodium channel decrease speed of depolarization 2. Class 2 antiarrhythmics - ANSWER beta blockers decrease sympathetic tone 3. Class 3 antiarrhythmics - ANSWER K channel block, prolong refractory period; dofelitide, sotolol, amiodarone, ibutilide 4. Class 4 antiarrhythmics - ANSWER Calcium channel blockers (diltiazem, verapamil) mainly affect SA and AV nodes 5. Class 5 antiarrhythmics - ANSWER digitalis agents digitoxin and digoxin 6. Class 1A antiarrhythmics - ANSWER Quinidine Procainamide Disopyramide prolong action potential "After" 7. Class 1B antiarrhythmics - ANSWER lidocaine, phenytoin, tocainide, mexiletine shorten duration of AP decrease refractoriness "before" shorten action potential 8. Class 1C antiarrhythmics - ANSWER flecainide, encainide, propfenone, moricizine marked slowing of upstroke of AP 9. what drugs list bradycardia as a Side effect - ANSWER beta blockers calcium blockers amiodarone 10. Class 1B side effects - ANSWER CNS effects 11. normal SACT - ANSWER 50-125ms evaluates for SA node exit block 12. SA node is richly intervated by what - ANSWER both sympathetic and parasympathetic 13. CCW flutter - ANSWER inferior leads (-) V1 (+) V6(-) 14. CW flutter - ANSWER Inferior leads (+) V1 (-) V6 (+) 15. functions of the av node - ANSWER wenkebach cycle length refractory periods conduction curve dual av nodal physiology 16. anything that causes loss of AV synchrony can cause what - ANSWER PMT (fix by extending PVARP) 17. what is the crista terminalis - ANSWER prominate ridge dividing trabeculated anterior and smooth wall in the RA 18. what makes up the triangle of Koch - ANSWER Coronary os, tendon of tadero, tricuspid valve 19. normal AH interval - ANSWER 55-125 ms 20. normal HV interval - ANSWER 35-55 ms (not effected by sedation) 21. position 1 in NGB code - ANSWER chambers paced 0 A V D 22. position 2 in NGB code - ANSWER Chambers sensed o A V D 23. position 3 in NGB code - ANSWER response to sensing O-none I-inhibit T-Trigger

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IBHRE CEPS 2025: LATEST UPDATED
GUARANTEED PASS!!
1.Class 1 Antiarrhythmics - ANSWER block the rapid sodium channel
decrease speed of depolarization

2. Class 2 antiarrhythmics - ANSWER beta blockers
decrease sympathetic tone

3. Class 3 antiarrhythmics - ANSWER K channel block, prolong refractory period;
dofelitide, sotolol, amiodarone, ibutilide

4. Class 4 antiarrhythmics - ANSWER Calcium channel blockers (diltiazem, verapamil)
mainly affect SA and AV nodes

5. Class 5 antiarrhythmics - ANSWER digitalis agents
digitoxin and digoxin

6. Class 1A antiarrhythmics - ANSWER Quinidine
Procainamide
Disopyramide
prolong action potential
"After"

7. Class 1B antiarrhythmics - ANSWER lidocaine, phenytoin, tocainide, mexiletine
shorten duration of AP
decrease refractoriness
"before"
shorten action potential

8. Class 1C antiarrhythmics - ANSWER flecainide, encainide, propfenone, moricizine
marked slowing of upstroke of AP

9. what drugs list bradycardia as a Side effect - ANSWER beta blockers
calcium blockers
amiodarone

10. Class 1B side effects - ANSWER CNS effects

11. normal SACT - ANSWER 50-125ms
evaluates for SA node exit block

,12. SA node is richly intervated by what - ANSWER both sympathetic and
parasympathetic

13. CCW flutter - ANSWER inferior leads (-)
V1 (+)
V6(-)

14. CW flutter - ANSWER Inferior leads (+)
V1 (-)
V6 (+)

15. functions of the av node - ANSWER wenkebach cycle length
refractory periods
conduction curve
dual av nodal physiology

16. anything that causes loss of AV synchrony can cause what - ANSWER PMT

(fix by extending PVARP)

17. what is the crista terminalis - ANSWER prominate ridge dividing trabeculated
anterior and smooth wall in the RA

18. what makes up the triangle of Koch - ANSWER Coronary os, tendon of tadero,
tricuspid valve

19. normal AH interval - ANSWER 55-125 ms

20. normal HV interval - ANSWER 35-55 ms
(not effected by sedation)

21. position 1 in NGB code - ANSWER chambers paced
0
A
V
D

22. position 2 in NGB code - ANSWER Chambers sensed
o
A
V
D

23. position 3 in NGB code - ANSWER response to sensing
O-none
I-inhibit

,T-Trigger
D- inhibit and trigger

24. position 4 in NGB code - ANSWER rate modulation
O-none
R- Rate modulation

25. what percentage of people have retrograde conduction - ANSWER 66%

26. what is the purpose of safety pacing? - ANSWER ensure ventricular pacing in spite
of crosstalk

27. what parameters would one most likely program to correct the problem of cross talk
? - ANSWER V blanking
A output
V sensitivity

28. what type of tachycardia typically occurs within 48 hrs of an acute MI and accounts
for about 20% of cardiac deaths - ANSWER automatic

29. which types of tachycardia is typically non-inducible , younger patients with heart
disease and often provoked by exercise - ANSWER outflow tract

30. cross talk Is most commonly what - ANSWER atrial output sensed on the ventricular
channel


31.midazolam - ANSWER what is the common name for Versed

32. .04 mg/kg - ANSWER standard adult dose of atropine

33. half dose - ANSWER how much NPH insulin should a patient be administered prior
to an EP study? (they are fasting think about blood sugar)

34. 4-8 weeks - ANSWER What is the elimination half life of amiodarone?

35. 4 half lives - ANSWER how long should an anti arrhythmic drug be discontinued
prior to EP study?

36. sodium channel - ANSWER what do class I drugs primarily affect?

37. slope of phase 0 (depolarization speed) - ANSWER what do class 1 drugs effect on
sodium channels primarily do?

38. Class 1A drugs - ANSWER slow conduction velocity and increase refractoriness

, 39. Class 1B drugs - ANSWER drugs that shorten refractory periods

40. Class 1C drugs - ANSWER pronounced depressants on conduction velocity

41. class II drugs - ANSWER What class antiarrhythmic are beta blockers

42. Class II Drugs (Beta Blockers) - ANSWER Prevent catecholamine-mediated action
on the heart, blocks sympathetic nervous system cardiac effects (no effect on AP,
decreases sympathetic tone)

43. `class III drugs - ANSWER increase AP duration by increasing refractoriness but do
not affect conduction velocity

44. class IV drugs - ANSWER calcium channel blockers (primarily affecting SA and AV
nodes)

45. Class V drugs - ANSWER digitalis agents that are used to increase parasympathetic
activity

46. Class IA drug examples - ANSWER procainamide, disopyrimidine, quinidine

47. Class IB drug examples - ANSWER Lidocaine Mexiletine

48. Class IC drug examples - ANSWER Flecainide, Propafenone, Encainide

49. Class II antiarrhythmic drug examples (beta blockers) - ANSWER propanolol,
metoprolol, atenolol, esmolol

50. class III drug examples - ANSWER amiodarone, ibutilide, sotolol, dofetilide

51. Class IV drug examples - ANSWER Verapamil, Diltiazem

52. 1:1 (10 mg/1000 units) - ANSWER what is the ratio that heparin and protamine
counteract eachother?

53. 2.0-3.0 - ANSWER INR needed for heparinizing a left sided case

54. prothrombin to thrombin - ANSWER what part of the coagulation cascade does
heparin block?

55. after gaining venous access - ANSWER when is heparin usually administered
during a case requiring a transeptal puncture?

56. hypotension - ANSWER what can happen if procainamide is administered too
quickly?

Información del documento

Subido en
4 de febrero de 2025
Número de páginas
67
Escrito en
2024/2025
Tipo
Examen
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