WCU Patho 370 CHYU: Week 3: Questions &
Answers(Solved)
Tertiary Prevention Examples - ANSWERCounseling on medication; rehabilitation;
supportive care; reducing disability
Latent Period - ANSWERtime between exposure of tissue to injurious agent and first
appearance of signs and/or symptoms
Endemic - ANSWERNative to local region
Subclinical Disease - ANSWERno noticeable signs or symptoms (inapparent infection)
Etiology - ANSWERcause of disease
Idiopathic - ANSWERunknown cause
latrogenic - ANSWERCause results from unintended or unwanted medical treatment
Primary prevention - ANSWERaltering susceptibility or reducing exposure for
susceptible persons ex: vaccinations
Allostasis - ANSWERprocess by which the body achieves stability through
homeostatis
Selye's 3 Phases of Stress - ANSWERAlarm reaction; resistance; exhaustian
sympathetic nervous system: Norepinephrine - ANSWERcauses vasoconstriction &
increases BP; Reduces gastric secretions; increases night and far vision
sympathetic nervous system: epinephrine - ANSWERenhances myocardial
contractility; increases HR & CO; causes bronchiodilation; increases glucose release
from liver
Hormones released during the stress response - ANSWERCortisol
Antidiuretic Hormone (ADH)
Norepinephrine
Epinephrine
Endorphines
Oxytocin
RAA pathway (renin-angiotensin-aldosterone)
Types of intracellular accumulation - ANSWER-Excessive amounts of normal
intracellular substance
-Abnormal substances from faulty matabolism synthesis
-particles that the cell is unable to degrade
, Cellular adaptation types - ANSWER-Atrophy: Decrease cell size
-Hypertrophy: increase cell size
-Hyperplasia: increase cell number
-Metaplasia: conversion of one cell type to another
-Dysplasia: disorderly growth
Coagulative necrosis - ANSWERthis process begins with ischemia, ends with
degradation of plasma membrane (heart)
Liquefactive necrosis - ANSWERliquification of lysosomal enzymes, formation of
abscess or cyst from dissolved dead tissue (brain)
Fat necrosis - ANSWERdeath of adipose tissue, appears as chalk white area ,usually
due to trauma or pancreatitis (pancreas)
Caseous Necrosis - ANSWERcharacteristic of lung damage secondary to tuberculosis
(bacterial infection; resembles clumpy cheese (lung)
Apoptosis - ANSWERprogrammed cell death
Side effects of chemotherapy - ANSWERanemia
nausea
bleeding
infections
-oma - ANSWERtumor
carcinoma - ANSWERcancerous tumor (begins in kin or tissue that lines or covers
body organs)
sarcoma - ANSWERmalignant tumor (begins in bone or in soft tissue of the body)
staging - ANSWERthe process of classifying tumors with respect to how far the
disease has progressed, the potential for its responding to therapy, and the patient's
prognosis (stage 0 to 4)
grading - ANSWERa way to classify cancer cells & is done by a pathologist
Degree of malignancy & differentiation
Low Grade (Grade I, II)
High Grade (Grade III, IV)
Deficits in immune system function in cancer - ANSWERchemotherapy
cancer cells
cancer metastasis to bone marrow
malnutrition
Answers(Solved)
Tertiary Prevention Examples - ANSWERCounseling on medication; rehabilitation;
supportive care; reducing disability
Latent Period - ANSWERtime between exposure of tissue to injurious agent and first
appearance of signs and/or symptoms
Endemic - ANSWERNative to local region
Subclinical Disease - ANSWERno noticeable signs or symptoms (inapparent infection)
Etiology - ANSWERcause of disease
Idiopathic - ANSWERunknown cause
latrogenic - ANSWERCause results from unintended or unwanted medical treatment
Primary prevention - ANSWERaltering susceptibility or reducing exposure for
susceptible persons ex: vaccinations
Allostasis - ANSWERprocess by which the body achieves stability through
homeostatis
Selye's 3 Phases of Stress - ANSWERAlarm reaction; resistance; exhaustian
sympathetic nervous system: Norepinephrine - ANSWERcauses vasoconstriction &
increases BP; Reduces gastric secretions; increases night and far vision
sympathetic nervous system: epinephrine - ANSWERenhances myocardial
contractility; increases HR & CO; causes bronchiodilation; increases glucose release
from liver
Hormones released during the stress response - ANSWERCortisol
Antidiuretic Hormone (ADH)
Norepinephrine
Epinephrine
Endorphines
Oxytocin
RAA pathway (renin-angiotensin-aldosterone)
Types of intracellular accumulation - ANSWER-Excessive amounts of normal
intracellular substance
-Abnormal substances from faulty matabolism synthesis
-particles that the cell is unable to degrade
, Cellular adaptation types - ANSWER-Atrophy: Decrease cell size
-Hypertrophy: increase cell size
-Hyperplasia: increase cell number
-Metaplasia: conversion of one cell type to another
-Dysplasia: disorderly growth
Coagulative necrosis - ANSWERthis process begins with ischemia, ends with
degradation of plasma membrane (heart)
Liquefactive necrosis - ANSWERliquification of lysosomal enzymes, formation of
abscess or cyst from dissolved dead tissue (brain)
Fat necrosis - ANSWERdeath of adipose tissue, appears as chalk white area ,usually
due to trauma or pancreatitis (pancreas)
Caseous Necrosis - ANSWERcharacteristic of lung damage secondary to tuberculosis
(bacterial infection; resembles clumpy cheese (lung)
Apoptosis - ANSWERprogrammed cell death
Side effects of chemotherapy - ANSWERanemia
nausea
bleeding
infections
-oma - ANSWERtumor
carcinoma - ANSWERcancerous tumor (begins in kin or tissue that lines or covers
body organs)
sarcoma - ANSWERmalignant tumor (begins in bone or in soft tissue of the body)
staging - ANSWERthe process of classifying tumors with respect to how far the
disease has progressed, the potential for its responding to therapy, and the patient's
prognosis (stage 0 to 4)
grading - ANSWERa way to classify cancer cells & is done by a pathologist
Degree of malignancy & differentiation
Low Grade (Grade I, II)
High Grade (Grade III, IV)
Deficits in immune system function in cancer - ANSWERchemotherapy
cancer cells
cancer metastasis to bone marrow
malnutrition
