test 1 term 3 pnvn 1631
1. what is hyperlipidemia: extreme levels of lipids (grease) in the blood
2. hyperlipidemia is a big cause of what ?: atherosclerosis
3. What is atherosclerosis?: amplify of grease, cholesterol, thickening of the channels
4. what puts you at extreme risk for atherosclerosis and coronary congestive heart failure: - depressed
levels of HDL
- extreme levels of LDL
5. basic situations for hyperlipidemia: - weigh decline
- depressed choledterol & fat
- exersice
6. what is the operation of hostility acid binding resins: bind hostility acids to promote raised absorption
of cholesterol
7. what drugs are hostility binding resins: - cholestyramine
-colestipol
-cocosevelam
8. what are the conduct of fibric acids: lower triglyceride levels
9. what drugs are fibric acids: -gemfibrozil (lopid)
-gnograte (tricor)
10. What is a dysrhythmia: Abnormal soul beat
11. what causes dysrhythmias: caused apiece atypical leader containers or a obstruction of the normal
road. stress, blood deficiency, and heart attack can too cause uneven brawny contraction
12. what are the 2 types of dysrhythmias and their delimiting characteris- tics: SUPRAVENTRICULAR,
expands above the truncus atrioventricularis, atrial flap and fibrillation, rash atrial contractions, cavity
heart attack and bradycardia
VENTRICULAR, expands beneath the truncus atrioventricularis, premature ventricular contrac- tions,
ventricular fibrillation, ventricular heart attack
13. class 1 dysrhythmia drugs (does what): myocardial sedative that restricts sodium ion drive
14. class 1a dysrhythmia drugs (does what): prolongs duration of elec- trical provocation
15. class 1b dysrhythmia drugs (does what): diminish event of electri- u.s. state stimulation
16. class 1c dysrhythmia drugs (does what): forceful myocardial depres- sants, slow broadcast rate
17. Class II dysrhythmia cure: being tested-adrenergic obstructing agents - block catecholamine from
SNS
18. Class III dysrhythmia cures: slow rate of energetic broadcast
19. class iv dysrhythmia cures: block calcium iomn flow
1/
1. what is hyperlipidemia: extreme levels of lipids (grease) in the blood
2. hyperlipidemia is a big cause of what ?: atherosclerosis
3. What is atherosclerosis?: amplify of grease, cholesterol, thickening of the channels
4. what puts you at extreme risk for atherosclerosis and coronary congestive heart failure: - depressed
levels of HDL
- extreme levels of LDL
5. basic situations for hyperlipidemia: - weigh decline
- depressed choledterol & fat
- exersice
6. what is the operation of hostility acid binding resins: bind hostility acids to promote raised absorption
of cholesterol
7. what drugs are hostility binding resins: - cholestyramine
-colestipol
-cocosevelam
8. what are the conduct of fibric acids: lower triglyceride levels
9. what drugs are fibric acids: -gemfibrozil (lopid)
-gnograte (tricor)
10. What is a dysrhythmia: Abnormal soul beat
11. what causes dysrhythmias: caused apiece atypical leader containers or a obstruction of the normal
road. stress, blood deficiency, and heart attack can too cause uneven brawny contraction
12. what are the 2 types of dysrhythmias and their delimiting characteris- tics: SUPRAVENTRICULAR,
expands above the truncus atrioventricularis, atrial flap and fibrillation, rash atrial contractions, cavity
heart attack and bradycardia
VENTRICULAR, expands beneath the truncus atrioventricularis, premature ventricular contrac- tions,
ventricular fibrillation, ventricular heart attack
13. class 1 dysrhythmia drugs (does what): myocardial sedative that restricts sodium ion drive
14. class 1a dysrhythmia drugs (does what): prolongs duration of elec- trical provocation
15. class 1b dysrhythmia drugs (does what): diminish event of electri- u.s. state stimulation
16. class 1c dysrhythmia drugs (does what): forceful myocardial depres- sants, slow broadcast rate
17. Class II dysrhythmia cure: being tested-adrenergic obstructing agents - block catecholamine from
SNS
18. Class III dysrhythmia cures: slow rate of energetic broadcast
19. class iv dysrhythmia cures: block calcium iomn flow
1/
