Dysrhythmia Worksheet; normal sinus
and PVC/VTach answers
Rate: 60-100
Rhythm: Regular
P wave: before every QRS
PR interval; 0.12-0.20
QRS: 0.06-0.10✔✔ECG characteristics
Normal Sinus Rhythm
Rate: variable
Rhythm: irregular;
P wave; absent before PVC
PR interval; absent before PVC
QRS; more than 0.12 sec; bizarre in appearance, differs from normal QRS complex.
Couplet; 2 in a row
Bigeminy; PVC following ea. normal beat
Triplet; 3 in a row or Salvo; short run of vent. tachycardia.
Unifocal; all look same, org. from one ectopic site (monomorphic)
Multifocal; arise diff. ectopic sites and appear diff from one another on ECG
(polymorphic)
Ventricular tachycardia; 3 or more PVC's in a row, sustained or non-
sustained✔✔ECG characteristics; PVC
Premature Ventricular Contractions/Ventricular tachycardia
PVC; increased automaticity or re-entry
Vtach; re-entry
PVC; usually no S & S, if frequent; decreased CO
Vtach-decrease CO to no CO; pulse or pulseless
Warning signs of PVCs: (client with acute heart disease)
*PVC with 4 hours after MI
*Frequent PVC (6 or more per min)
*Couplet/triplets
*R on T phenomenon (PVC falling on the T wave)
*Multifocal PVC✔✔Clinical Manifestations of PVC
*The frequency and patterns of PVC's can be indicative of myocardial irritability and
the risk for a lethal dysrhythmia.
Anxiety, Stress, tobacco, alcohol, caffeine, hypoxia, acidosis, hypokalemia,
hypocalcemia, hypomagnesium, drug toxicity (digoxin), CAD, heart failure, invasive
lines, reperfusion after fibrolytic therapy. Incidence and significance greatest after
MI✔✔Causes of PVC
O2, electrolytes, cardiac enzymes; ie. -CPK, creatine phosphokinase or CK. creatine
kinase; CK-MB more specific form -Myoglobin and Troponin (proteins; when heart
muscle is damaged these substances are released from the heart muscle cells into
and PVC/VTach answers
Rate: 60-100
Rhythm: Regular
P wave: before every QRS
PR interval; 0.12-0.20
QRS: 0.06-0.10✔✔ECG characteristics
Normal Sinus Rhythm
Rate: variable
Rhythm: irregular;
P wave; absent before PVC
PR interval; absent before PVC
QRS; more than 0.12 sec; bizarre in appearance, differs from normal QRS complex.
Couplet; 2 in a row
Bigeminy; PVC following ea. normal beat
Triplet; 3 in a row or Salvo; short run of vent. tachycardia.
Unifocal; all look same, org. from one ectopic site (monomorphic)
Multifocal; arise diff. ectopic sites and appear diff from one another on ECG
(polymorphic)
Ventricular tachycardia; 3 or more PVC's in a row, sustained or non-
sustained✔✔ECG characteristics; PVC
Premature Ventricular Contractions/Ventricular tachycardia
PVC; increased automaticity or re-entry
Vtach; re-entry
PVC; usually no S & S, if frequent; decreased CO
Vtach-decrease CO to no CO; pulse or pulseless
Warning signs of PVCs: (client with acute heart disease)
*PVC with 4 hours after MI
*Frequent PVC (6 or more per min)
*Couplet/triplets
*R on T phenomenon (PVC falling on the T wave)
*Multifocal PVC✔✔Clinical Manifestations of PVC
*The frequency and patterns of PVC's can be indicative of myocardial irritability and
the risk for a lethal dysrhythmia.
Anxiety, Stress, tobacco, alcohol, caffeine, hypoxia, acidosis, hypokalemia,
hypocalcemia, hypomagnesium, drug toxicity (digoxin), CAD, heart failure, invasive
lines, reperfusion after fibrolytic therapy. Incidence and significance greatest after
MI✔✔Causes of PVC
O2, electrolytes, cardiac enzymes; ie. -CPK, creatine phosphokinase or CK. creatine
kinase; CK-MB more specific form -Myoglobin and Troponin (proteins; when heart
muscle is damaged these substances are released from the heart muscle cells into
