①S-pa
d
of
-
↑ stretch
⑯ Receives O blood from
activity
lungs via L+ R petency Levels codicea fibres impulse
veirs
through dicuspid value
, get a
~
to LV
.
septem Mitral/bicuspid-LA + 1N,
in direction of I.
relaration phase
Thelleat Mrc/SV .
relate
↑
State when ventricles
Aortic-directi ↑
S
·
blood from#/SVC aurte
& !
~of body via
+
passes
to RV through The heat - es
LV + bod
,
&
I
&
tricuspid value
tricuspid-RA QU blood flor
, blood passes
regulate
Ceda
+
,
② blood prevent back artric-ventricl
⑳
from to words RV ID,
do Or blood through en
.
the
pul/semi-henor-RV ,
in
.
flor centr
pelmency value
to langs
Pulveins direction lungsthen
of . not all blood add
S
via pulmonary
artes ↑ into .
LA
wre-beat plmin
O
I contract
geited &
SenseTime
into
when ventricles
Bod
Vessels artey leaving pumped to lungs/
pumped under
⑤volklo
main
blood
S to get blood
VenerCure
outreduce
I -
& & Aorta-LV ,
On blood body .
per
heart is myog
delier de Oz
Pul arteries best I
&rest/afte
Max
contractive its ow
form bed resting WR is sleep, intentes
-
blood S
centrati
to RA de O
,
blood love in trained athletes
lungs value between
fr Ru to
of
.
↑=
series
RURMHR is a
D Greate HR
↑ calls electric
range e untrained
& He
S
.
range to central
S brain +
& spired
-
effects of
S
①Highe SV +
AWS
R
card
higher MR inturn Rejection
-
high
-
max &
puts enter streie
.
CO on CNS
range SV
:
performance fractiest
&
&
/
~
arteries +heart
.
3) Can cape
BP is the fere against on
& suggests the
Pretigwert
higher vessel wall
&
~ end diastolic vol
I Point where
a
highe the EDU the
intensity than
,
the cardiac muscl is amount of blood in
MighBP
WR so high that more
untrained
--
alleck etc
-
Stretch + greate the ventricles aftediasto
contraction
adversely
affected
SV when weekend
S
force of
insufficienttime
&
asthere blood vessel
supplying ↑
ventricles to -
brein
-
Preload
is pressive
d
of
-
↑ stretch
⑯ Receives O blood from
activity
lungs via L+ R petency Levels codicea fibres impulse
veirs
through dicuspid value
, get a
~
to LV
.
septem Mitral/bicuspid-LA + 1N,
in direction of I.
relaration phase
Thelleat Mrc/SV .
relate
↑
State when ventricles
Aortic-directi ↑
S
·
blood from#/SVC aurte
& !
~of body via
+
passes
to RV through The heat - es
LV + bod
,
&
I
&
tricuspid value
tricuspid-RA QU blood flor
, blood passes
regulate
Ceda
+
,
② blood prevent back artric-ventricl
⑳
from to words RV ID,
do Or blood through en
.
the
pul/semi-henor-RV ,
in
.
flor centr
pelmency value
to langs
Pulveins direction lungsthen
of . not all blood add
S
via pulmonary
artes ↑ into .
LA
wre-beat plmin
O
I contract
geited &
SenseTime
into
when ventricles
Bod
Vessels artey leaving pumped to lungs/
pumped under
⑤volklo
main
blood
S to get blood
VenerCure
outreduce
I -
& & Aorta-LV ,
On blood body .
per
heart is myog
delier de Oz
Pul arteries best I
&rest/afte
Max
contractive its ow
form bed resting WR is sleep, intentes
-
blood S
centrati
to RA de O
,
blood love in trained athletes
lungs value between
fr Ru to
of
.
↑=
series
RURMHR is a
D Greate HR
↑ calls electric
range e untrained
& He
S
.
range to central
S brain +
& spired
-
effects of
S
①Highe SV +
AWS
R
card
higher MR inturn Rejection
-
high
-
max &
puts enter streie
.
CO on CNS
range SV
:
performance fractiest
&
&
/
~
arteries +heart
.
3) Can cape
BP is the fere against on
& suggests the
Pretigwert
higher vessel wall
&
~ end diastolic vol
I Point where
a
highe the EDU the
intensity than
,
the cardiac muscl is amount of blood in
MighBP
WR so high that more
untrained
--
alleck etc
-
Stretch + greate the ventricles aftediasto
contraction
adversely
affected
SV when weekend
S
force of
insufficienttime
&
asthere blood vessel
supplying ↑
ventricles to -
brein
-
Preload
is pressive
