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Cardiovascular - Summary Saunders Comprehensive Review for the Nclex-Rn Examination Cardiovascular Nursing. Everything you need to know about cardiovascular nursing (excluding pharmacology) based on lecture notes and Saunders Comprehensive Review for the

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Cardiovascular - Summary Saunders Comprehensive Review for the Nclex-Rn Examination Cardiovascular Nursing Diagnostic Tests Cardiac Markers CK-MB (creatinine kinase, myocardial muscle) - an ele vation in value indicates myocardial damage - an elevation occurs within hours and peaks at 18 hours following an acute ischemic attack Normal value male: 2-6ng/mL Normal value female: 2 to 5ng/mL Troponin Values are low, any rise can indicate myocardial cell damage - troponin I: rises within 3 hours and persists for up to 7-10 days (this one is especially related to myocardial injury) … less than 0.3ng/mL - troponin T less than 0.2ng/mL - serum levels of troponin T and I increase 4-6 hours after the onset of the MI, peak at 10-24 hours, and return to baseline after 10-14 days Myoglobin - myoglonin is an oxygen-binding protein found in cardiac and skeletal muscle - the level rises within 2 hours after cell death, with a rapid decline in the level after 7 hours - may not be cardiac specific Complete Blood Count (CBC) Red Blood Cell Count - decreases in rheumatic heart disease and infective endocarditis - increases in conditions characterized by inadequate tissue oxygenation White Blood Cell Count - increases in infectious and inflammatory diseases of the heart - increases after an MI because large numbers of WBCs are needed to dispose of the necrotic tissue resulting from the infarction Hemocysteine - elevated levels may increase the risk of cardiovascular disease - normal value: 0.54 to 1.9 mg/L Highly Sensitive C-Reactive Protein - detects an inflammatory process (e.g. atherothrombosis) - less than 1mg/dL is considered low risk - greater than 3mg/dL Blood Coagulation Factors - an increase can occur during and after an MI, which places the patient at greater risk for thrombophlebitis and formation of clots in the coronary arteries Serum Lipids - the lipid profile measures serum cholesterol, triglyceride, and lipoprotein levels - lipid profile is used to assess the risk of developing coronary artery disease - lipoprotein-a or Lp(a) increases atherosclerotic plaques and increases clots…. Normal value should be less than 30mg/dl Electrolytes * electrolyte and mineral imbalances can cause cardiac electrical instability that can result in life-threatening dysrhythmias Potassium Hypokalemia - causes increased cardiac electrical instability, ventricular dysrhythmias, and increased risk of digoxin toxicity - ECG shows flattening and inversion of the T wave, the appearance of a U wave, and ST depression Hyperkalemia - causes asystole and ventricular dysrhythmias - ECG shows tall, peaked T waves, widened QRS, or flat P waves Sodium - the serum sodium level decreases with the use of diuretics - the serum sodium level decreases in heart failure, indicating water excess Calcium Hypocalcaemia - can cause ventricular dysrhythmias, prolonged ST and QT intervals, and cardiac arrest Hypercalcemia - can cause atrioventricular block, tachycardia or bradycardia, digitalis hypersensitivity, cardiac arrest Phosphorus - should be interpreted with calcium levels because the kidneys retain or excrete one electrolyte in an inverse relationship to the other Magnesium - low level can cause ventricular tachycardia and fibrillation Blood Urea Nitrogen - BUN is elevated in heart disorders that adversely affect renal circulation, such as heart failure and cardiogenic shock - range: B-type Natriuretic peptide (BNP) - BNP is released in response to atrial and ventricular stretch; it serves as a marker for heart failure - should be less than 100pg/mL… the higher the level, the more severe the heart failure Diagnostic Proce

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Subido en
16 de noviembre de 2023
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2023/2024
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lOMoAR cPSD| 784381




Cardiovascular - Summary Saunders Comprehensive Review for the
Nclex-Rn Examination
Cardiovascular Nursing


Diagnostic Tests
Cardiac Markers
CK-MB - an elevation in value indicates myocardial
(creatinine damage
kinase, - an elevation occurs within hours and peaks at 18
myocardial hours following an acute ischemic attack
muscle) Normal value male: 2-6ng/mL
Normal value female: 2 to 5ng/mL
Troponin Values are low, any rise can indicate myocardial cell
damage
- troponin I: rises within 3 hours and persists for
up to 7-10 days (this one is especially related to
myocardial injury) … less than 0.3ng/mL
- troponin T less than 0.2ng/mL
- serum levels of troponin T and I increase 4-6
hours after the onset of the MI, peak at 10-24
hours, and return to baseline after 10-14 days

Myoglobin - myoglonin is an oxygen-binding protein found in
cardiac and skeletal muscle
- the level rises within 2 hours after cell death,
with a rapid decline in the level after 7 hours
- may not be cardiac specific



Complete Blood Count (CBC)

Red Blood Cell Count
- decreases in rheumatic heart disease and infective endocarditis
- increases in conditions characterized by inadequate tissue oxygenation

White Blood Cell Count
- increases in infectious and inflammatory diseases of the heart
- increases after an MI because large numbers of WBCs are needed to dispose of the
necrotic tissue resulting from the infarction

, lOMoAR cPSD| 784381




Hemocysteine
- elevated levels may increase the risk of cardiovascular disease
- normal value: 0.54 to 1.9 mg/L

Highly Sensitive C-Reactive Protein
- detects an inflammatory process (e.g. atherothrombosis)
- less than 1mg/dL is considered low risk
- greater than 3mg/dL

Blood Coagulation Factors
- an increase can occur during and after an MI, which places the patient at greater risk
for thrombophlebitis and formation of clots in the coronary arteries

Serum Lipids
- the lipid profile measures serum cholesterol, triglyceride, and lipoprotein levels
- lipid profile is used to assess the risk of developing coronary artery disease
- lipoprotein-a or Lp(a) increases atherosclerotic plaques and increases clots…. Normal
value should be less than 30mg/dl

Electrolytes

* electrolyte and mineral imbalances can cause cardiac electrical instability that
can result in life-threatening dysrhythmias
Potassium Hypokalemia
- causes increased cardiac electrical instability,
ventricular dysrhythmias, and increased risk of
digoxin toxicity
- ECG shows flattening and inversion of the T wave,
the appearance of a U wave, and ST depression

Hyperkalemia
- causes asystole and ventricular dysrhythmias
- ECG shows tall, peaked T waves, widened QRS,
or flat P waves
Sodium - the serum sodium level decreases with the use of
diuretics
- the serum sodium level decreases in heart failure,
indicating water excess
Calcium Hypocalcaemia
- can cause ventricular dysrhythmias, prolonged ST
and QT intervals, and cardiac arrest

Hypercalcemia
- can cause atrioventricular block, tachycardia or

, lOMoAR cPSD| 784381




bradycardia, digitalis hypersensitivity, cardiac arrest




Phosphorus - should be interpreted with calcium levels because
the kidneys retain or excrete one electrolyte in an
inverse relationship to the other
Magnesium - low level can cause ventricular tachycardia and
fibrillation
- ECG will show tall T waves and depressed
ST segments (low level)
- high level can cause muscle
weakness, hypotension, and
bradycardia
Blood Urea Nitrogen
- BUN is elevated in heart disorders that adversely affect renal circulation, such as heart
failure and cardiogenic shock
- range:

B-type Natriuretic peptide (BNP)
- BNP is released in response to atrial and ventricular stretch; it serves as a marker for
heart failure
- should be less than 100pg/mL… the higher the level, the more severe the heart failure

Diagnostic Procedures

Chest X-Ray
- done to determine anatomical changes such as size, silhouette, and position of the
heart

Holter Monitoring
- noninvasive test where the client wears a monitor and an electrocardiographic tracing
is recorded over 24 or more hours while the client performs their activities of daily living
- the monitor identifies dysrhythmias and evaluates the effectiveness of anti-dysrhythmic
or pacemaker therapy

Echocardiography
- non-invasive procedure that evaluates structural and functional changes in the heart
- used to detect vulvar abnormalities, congenital heart defects, cardiac function

Exercise Electrocardiography Testing (stress test)
- studies the heart during activity and detects and evaluates coronary artery disease

, lOMoAR cPSD| 784381




- treadmill testing is the most common
- if the client is unable to tolerate exercise, an IV infusion of dipyridamole or dobutamine
hydrochloride is given to dilate the coronary arteries and stimulate the effects of
exercise (NPO for 3-6 hours before)

Cardiac Catheterization
- an invasive test involving the insertion of a catheter into the heart and surrounding
vessels
- obtains information about the structure and performance of the heart chambers and
valves and the coronary circulation

 Pre-procedure interventions:
- obtain informed consent
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