NSG552 Exam 3 Test Questions and Answers Graded A+
MOA: Pure opioid antagonist that competes and displaces
naloxone
opioids at opioid receptor sites.
methadone, buprenorphine, buprenorphine+naloxone Treatments for opioid use disorder.
buprenorphine+naloxone Treatment for opioid use disorder with comorbid pain.
Inappropriate use of what substance may be due to un-
opioids
controlled pain?
tablet, injectable, implant Naltrexone delivery methods.
implant Form of naltrexone limited to inpatient use.
buprenorphine Mu receptor partial agonist for opioid withdrawal.
Taking this medication too soon after last opioid use in-
buprenorphine creases the chances of intense withdrawal that comes on
very quickly (precipitated withdrawal).
Symptoms include nausea and vomiting, respiratory de-
opioid intoxication pression, constipation, itching, mioisis (small pupil). Pa-
tient will experience euphoria and sedation.
Symptoms include N/V/D and dehydration, irritability,
restlessness, yawning, and twitching, increased HR/BP,
opioid withdrawal
chills, increased temperature, rhinorrhea, lacrimation, di-
lated pupils.
Treatment for opioid intoxication during which cardiac or
naloxone
respiratory depression is a concern.
Symptoms include dilated pupils, HA, tremor, hyper-re-
flexia, twitching, seizures, or coma, increased HR/BP, ar-
cocaine intoxication
rhythmias, and MI, N/V, incontinence/ARF, or rhabdomy-
olysis
Treatment includes BZD, antipsychotics, and management
cocaine intoxication of medical problems including HTN, stroke, cardiac ar-
rhythmias, hyperthermia, and seizures.
1/6
, NSG552 Exam 3 Test Questions and Answers Graded A+
The use of beta blockers for treatment of chest pain and
cocaine MI during this intoxication is to be avoided due to unop-
posed a adrenergic stimulation.
Signs vary with blood levels, from decreased reaction time,
alcohol intoxication muscle incoordination, ataxia, dysarthria, to respiratory
failure and coma.
Treatment includes cardiopulmonary function mainte-
severe alcohol intoxication
nance, thiamine, and haloperidol PRN agitation.
Given IM/IV for 3 days to prevent Wernicke's en-
thiamine
cephalopathy, along with IV fluids and a banana bag.
benzodiazepines Class of drugs to avoid for acute alcohol intoxication.
Treatment includes BZD in either symptom triggered or
fixed dose; diazepam and chlordiazepoxide have a longer
uncomplicated alcohol withdrawal
half life, and oxazepam and lorazepam are suitable for
patients with hepatic dysfunction.
diazepam and chlordiazepoxide BZDs with a long half-life used to treat AUD.
BZDs with moderate half-life used in AUD patients with
oxazepam and lorazepam
liver disease.
Treatment includes diazepam IV or lorazepam IV/IM, thi-
alcohol withdrawal seizures
amine IV/IM, and addressing electrolyte imbalances.
Treatment includes acute care management, parenteral
DT diazepam or lorazepam, thiamine, and antipsychotics if
necessary.
MOA is via negative reinforcement, where drinking is
disulfiram
avoided due to unpleasant ettects.
NMDA receptor antagonist that is renally cleared, suitable
acamprosate
for AUD patients with hepatic dysfunction.
2/6
MOA: Pure opioid antagonist that competes and displaces
naloxone
opioids at opioid receptor sites.
methadone, buprenorphine, buprenorphine+naloxone Treatments for opioid use disorder.
buprenorphine+naloxone Treatment for opioid use disorder with comorbid pain.
Inappropriate use of what substance may be due to un-
opioids
controlled pain?
tablet, injectable, implant Naltrexone delivery methods.
implant Form of naltrexone limited to inpatient use.
buprenorphine Mu receptor partial agonist for opioid withdrawal.
Taking this medication too soon after last opioid use in-
buprenorphine creases the chances of intense withdrawal that comes on
very quickly (precipitated withdrawal).
Symptoms include nausea and vomiting, respiratory de-
opioid intoxication pression, constipation, itching, mioisis (small pupil). Pa-
tient will experience euphoria and sedation.
Symptoms include N/V/D and dehydration, irritability,
restlessness, yawning, and twitching, increased HR/BP,
opioid withdrawal
chills, increased temperature, rhinorrhea, lacrimation, di-
lated pupils.
Treatment for opioid intoxication during which cardiac or
naloxone
respiratory depression is a concern.
Symptoms include dilated pupils, HA, tremor, hyper-re-
flexia, twitching, seizures, or coma, increased HR/BP, ar-
cocaine intoxication
rhythmias, and MI, N/V, incontinence/ARF, or rhabdomy-
olysis
Treatment includes BZD, antipsychotics, and management
cocaine intoxication of medical problems including HTN, stroke, cardiac ar-
rhythmias, hyperthermia, and seizures.
1/6
, NSG552 Exam 3 Test Questions and Answers Graded A+
The use of beta blockers for treatment of chest pain and
cocaine MI during this intoxication is to be avoided due to unop-
posed a adrenergic stimulation.
Signs vary with blood levels, from decreased reaction time,
alcohol intoxication muscle incoordination, ataxia, dysarthria, to respiratory
failure and coma.
Treatment includes cardiopulmonary function mainte-
severe alcohol intoxication
nance, thiamine, and haloperidol PRN agitation.
Given IM/IV for 3 days to prevent Wernicke's en-
thiamine
cephalopathy, along with IV fluids and a banana bag.
benzodiazepines Class of drugs to avoid for acute alcohol intoxication.
Treatment includes BZD in either symptom triggered or
fixed dose; diazepam and chlordiazepoxide have a longer
uncomplicated alcohol withdrawal
half life, and oxazepam and lorazepam are suitable for
patients with hepatic dysfunction.
diazepam and chlordiazepoxide BZDs with a long half-life used to treat AUD.
BZDs with moderate half-life used in AUD patients with
oxazepam and lorazepam
liver disease.
Treatment includes diazepam IV or lorazepam IV/IM, thi-
alcohol withdrawal seizures
amine IV/IM, and addressing electrolyte imbalances.
Treatment includes acute care management, parenteral
DT diazepam or lorazepam, thiamine, and antipsychotics if
necessary.
MOA is via negative reinforcement, where drinking is
disulfiram
avoided due to unpleasant ettects.
NMDA receptor antagonist that is renally cleared, suitable
acamprosate
for AUD patients with hepatic dysfunction.
2/6
