GCU 631 Final exam Advanced pathophysiology Full
Study online at https://quizlet.com/_ch59za
1. Where do we hold the DNA inside of a cell? The nucleus
2. What is cell suicide? apoptosis
3. What are some things we see clinically in a patient -Normal SpO2
presenting with carbon monoxide poisoning? -Tissue damage
4. What do we have: ABG w/ pH 7.25, bicarb 28, CO2 is Respiratory acidosis
60?
5. How do we get molecules to between intracellular & Hydrostatic pressure
extra cellular? (If it's water: osmotic pres-
sure)
6. If patient has large volume of vomiting, what would Metabolic Alkalosis
we see?
7. Why does metabolic alkalosis develop from vomiting? Losing acid, retaining Bi-
carb
8. What do we look for on an amnio to see neural tube Alpha-fetal protein
defects?
9. Clinical characteristics of Trisomy 21 Wide eyes
Wide neck
Short stature
Low IQ
Low nasal bridge
Low set ears
Cardiac defects
10. How could a newborn have type 1 diabetes Autoimmune/ Cannot be
prevented
, GCU 631 Final exam Advanced pathophysiology Full
Study online at https://quizlet.com/_ch59za
11. We have a certain gene that takes care of mainte- Housekeeping genes
nance of other cells
12. Patient comes in with chronic wounds that aren't heal- -Diabetes
ing. What are you thinking? -Something else is going
on with those patient/oth-
er comorbidities
13. If a newborn doesn't have enough collectin-like pro- -Pneumonia
tein what kind of infection might they develop? -Anything RESPIRATORY
related
14. Where do B lymphocytes grow up and develop Bone marrow
15. I go over to a friend's house to get chickenpox. What Active acquired immuni-
type of immunity is that? ty/Body will make anti-
bodies
16. What is happening at a cellular level with a type two -Antibodies are attaching
sensitivity reaction? to the cellular surface of
the antigen.
-release of histamines and
IgE
17. What does Rhogram do? -Stops hemolytic anemia
-Give to Rh - moms with a
Rh + baby
18. If we have a patient that went under some organ Start to see TH1 cells re-
transplant, why would we have tissue damage? lease too many cytokines
so we will see the cytotoxic
effects. Cytokines will ac-
, GCU 631 Final exam Advanced pathophysiology Full
Study online at https://quizlet.com/_ch59za
tually attack the endothe-
lial cells.
19. What is an exotoxin? When bacteria is growing
they release exotoxins.
20. What do we have that helps us fight fungal infections? -Phagocytes
-T-Lymphocytes
21. Why, if I'm really stressed, am I more likely to get sick? Stress releases cortisol,
cortisol increases, helper T
cells are suppressed.
22. Stressed patient (long period of time), what can we -Hypoglycemia
see develop with lab results? -Cortisol increase caus-
es hyperglycemia initial-
ly, but with chronic stress
cortisol levels become
depleted causing hypo-
glycemia. (Adrenal insuffi-
ciency)
23. What happens when we have cellular metabolism that Build up of waste
just isn't working right? What will develop?
24. What is the least likely IL to cause endothelial cells to IL4
go into that proinflammatory state?
25. If you had a patient that developed MODS, what sub- Interleukins
stances stimulate the normal endothelial cells to go tumor necrosis factor
into a proinflammatory state? IL-6
26. How dehydrated does a child have to be before we see 10%
them have low BP
Study online at https://quizlet.com/_ch59za
1. Where do we hold the DNA inside of a cell? The nucleus
2. What is cell suicide? apoptosis
3. What are some things we see clinically in a patient -Normal SpO2
presenting with carbon monoxide poisoning? -Tissue damage
4. What do we have: ABG w/ pH 7.25, bicarb 28, CO2 is Respiratory acidosis
60?
5. How do we get molecules to between intracellular & Hydrostatic pressure
extra cellular? (If it's water: osmotic pres-
sure)
6. If patient has large volume of vomiting, what would Metabolic Alkalosis
we see?
7. Why does metabolic alkalosis develop from vomiting? Losing acid, retaining Bi-
carb
8. What do we look for on an amnio to see neural tube Alpha-fetal protein
defects?
9. Clinical characteristics of Trisomy 21 Wide eyes
Wide neck
Short stature
Low IQ
Low nasal bridge
Low set ears
Cardiac defects
10. How could a newborn have type 1 diabetes Autoimmune/ Cannot be
prevented
, GCU 631 Final exam Advanced pathophysiology Full
Study online at https://quizlet.com/_ch59za
11. We have a certain gene that takes care of mainte- Housekeeping genes
nance of other cells
12. Patient comes in with chronic wounds that aren't heal- -Diabetes
ing. What are you thinking? -Something else is going
on with those patient/oth-
er comorbidities
13. If a newborn doesn't have enough collectin-like pro- -Pneumonia
tein what kind of infection might they develop? -Anything RESPIRATORY
related
14. Where do B lymphocytes grow up and develop Bone marrow
15. I go over to a friend's house to get chickenpox. What Active acquired immuni-
type of immunity is that? ty/Body will make anti-
bodies
16. What is happening at a cellular level with a type two -Antibodies are attaching
sensitivity reaction? to the cellular surface of
the antigen.
-release of histamines and
IgE
17. What does Rhogram do? -Stops hemolytic anemia
-Give to Rh - moms with a
Rh + baby
18. If we have a patient that went under some organ Start to see TH1 cells re-
transplant, why would we have tissue damage? lease too many cytokines
so we will see the cytotoxic
effects. Cytokines will ac-
, GCU 631 Final exam Advanced pathophysiology Full
Study online at https://quizlet.com/_ch59za
tually attack the endothe-
lial cells.
19. What is an exotoxin? When bacteria is growing
they release exotoxins.
20. What do we have that helps us fight fungal infections? -Phagocytes
-T-Lymphocytes
21. Why, if I'm really stressed, am I more likely to get sick? Stress releases cortisol,
cortisol increases, helper T
cells are suppressed.
22. Stressed patient (long period of time), what can we -Hypoglycemia
see develop with lab results? -Cortisol increase caus-
es hyperglycemia initial-
ly, but with chronic stress
cortisol levels become
depleted causing hypo-
glycemia. (Adrenal insuffi-
ciency)
23. What happens when we have cellular metabolism that Build up of waste
just isn't working right? What will develop?
24. What is the least likely IL to cause endothelial cells to IL4
go into that proinflammatory state?
25. If you had a patient that developed MODS, what sub- Interleukins
stances stimulate the normal endothelial cells to go tumor necrosis factor
into a proinflammatory state? IL-6
26. How dehydrated does a child have to be before we see 10%
them have low BP
