ASCP RECALLS 2026 521 QUESTIONS WITH
COMPLETE SOLUTIONS VERIFIED
CONTENT GRADED A+
⩥ Hyperplasia.
Answer: Increased cell numbers in response to hormones and other
growth factors; occurs in tissues whose cells are able to divide
⩥ Atrophy.
Answer: Decreased cell and organ size, as a result of decreased nutrient
supply or disuse: associated with decreased synthesis and increased
proteolytic breakdown of cellular organelles
⩥ Metaplasia.
Answer: Change in phenotype of differentiated cells, often a response to
chronic irritation that makes cells better able to withstand the stress
⩥ Hypoxia.
Answer: Oxygen deficiency which interferes with aerobic oxidative
respirations and is an extremely important and common cause of cell
injury and death
⩥ Ischemic.
,Answer: Loss of blood supply in a tissue due to impeded arterial flow or
reduced venous drainage
⩥ Coagulative necrosis.
Answer: A form of tissue necrosis in which the component cells are dead
but the basic tissue architecture is preserved for at least several days
⩥ Liquefactive necrosis.
Answer: A form of necrosis seen in focal bacterial or occasionally fungal
infections because microbes stimulate the accumulation of inflammatory
cells and the enzymes of leukocytes digest the tissue
⩥ Caseous necrosis.
Answer: A form of necrosis encountered most often in foci of
tuberculous infections
⩥ Fat necrosis.
Answer: Term referring to focal areas of fat destruction, typically
resulting from release of activated pancreatic lipases into the peritoneal
cavity
⩥ Fibrinous necrosis.
Answer: A special form of necrosis usually seen in immune reactions
involving blood vessels
,⩥ Autophagy.
Answer: Lysosomal digestion of the cell's own components
⩥ Apoptosis.
Answer: A pathway of cell death that is induced by a tightly regulated
suicide program in which the cells destined to die activate enzymes
capable of degrading the cells own nuclear DNA
⩥ Steatosis (fatty change).
Answer: Refers to any abnormal accumulation of triglycerides within
parenchymal cells It is most often seen in the liver
⩥ Dystrophic calcification.
Answer: Depositions of calcium at sites of cell injury and necrosis
⩥ Metastatic calcification.
Answer: Deposition of calcium in normal tissues, caused by
hypercalcemia (usually a consequence of parathyroid hormone excess)
⩥ Inflammation.
Answer: A protective response intended to eliminate the initial cause of
cell injury as well as the necrotic cells and tissues resulting from the
original insult
, ⩥ Acute inflammation.
Answer: A rapid response to injury or microbes and other foreign
substance that is designed to deliver leukocytes and plasma proteins to
sites of injury
⩥ Serous inflammation.
Answer: Fluid in a serous cavity
Serous inflammation is marked by fluid transudates, reflecting
moderately increased vascular permeability. Such accumulations in the
peritoneal, pleural, and pericardial cavities are called effusions;
⩥ Effusion.
Answer: Fluid in a serous cavity ,
⩥ Fibrinous inflammation.
Answer: Inflammation occurring as a consequence of more severe
injuries, resulting in greater vascular permeability that allows large
molecules (such as fibrinogen) to pass the endothelial barrier
⩥ Abscess.
COMPLETE SOLUTIONS VERIFIED
CONTENT GRADED A+
⩥ Hyperplasia.
Answer: Increased cell numbers in response to hormones and other
growth factors; occurs in tissues whose cells are able to divide
⩥ Atrophy.
Answer: Decreased cell and organ size, as a result of decreased nutrient
supply or disuse: associated with decreased synthesis and increased
proteolytic breakdown of cellular organelles
⩥ Metaplasia.
Answer: Change in phenotype of differentiated cells, often a response to
chronic irritation that makes cells better able to withstand the stress
⩥ Hypoxia.
Answer: Oxygen deficiency which interferes with aerobic oxidative
respirations and is an extremely important and common cause of cell
injury and death
⩥ Ischemic.
,Answer: Loss of blood supply in a tissue due to impeded arterial flow or
reduced venous drainage
⩥ Coagulative necrosis.
Answer: A form of tissue necrosis in which the component cells are dead
but the basic tissue architecture is preserved for at least several days
⩥ Liquefactive necrosis.
Answer: A form of necrosis seen in focal bacterial or occasionally fungal
infections because microbes stimulate the accumulation of inflammatory
cells and the enzymes of leukocytes digest the tissue
⩥ Caseous necrosis.
Answer: A form of necrosis encountered most often in foci of
tuberculous infections
⩥ Fat necrosis.
Answer: Term referring to focal areas of fat destruction, typically
resulting from release of activated pancreatic lipases into the peritoneal
cavity
⩥ Fibrinous necrosis.
Answer: A special form of necrosis usually seen in immune reactions
involving blood vessels
,⩥ Autophagy.
Answer: Lysosomal digestion of the cell's own components
⩥ Apoptosis.
Answer: A pathway of cell death that is induced by a tightly regulated
suicide program in which the cells destined to die activate enzymes
capable of degrading the cells own nuclear DNA
⩥ Steatosis (fatty change).
Answer: Refers to any abnormal accumulation of triglycerides within
parenchymal cells It is most often seen in the liver
⩥ Dystrophic calcification.
Answer: Depositions of calcium at sites of cell injury and necrosis
⩥ Metastatic calcification.
Answer: Deposition of calcium in normal tissues, caused by
hypercalcemia (usually a consequence of parathyroid hormone excess)
⩥ Inflammation.
Answer: A protective response intended to eliminate the initial cause of
cell injury as well as the necrotic cells and tissues resulting from the
original insult
, ⩥ Acute inflammation.
Answer: A rapid response to injury or microbes and other foreign
substance that is designed to deliver leukocytes and plasma proteins to
sites of injury
⩥ Serous inflammation.
Answer: Fluid in a serous cavity
Serous inflammation is marked by fluid transudates, reflecting
moderately increased vascular permeability. Such accumulations in the
peritoneal, pleural, and pericardial cavities are called effusions;
⩥ Effusion.
Answer: Fluid in a serous cavity ,
⩥ Fibrinous inflammation.
Answer: Inflammation occurring as a consequence of more severe
injuries, resulting in greater vascular permeability that allows large
molecules (such as fibrinogen) to pass the endothelial barrier
⩥ Abscess.
