Lippincott Illustrated Reviews:
Pharmacology, 8th Edition - Whalen, Test
Bank - Chapters 1 – 48 Latest 2026
,Table Of Contents
1. Chapter 1: pharmacokinetics
2. Chapter 2: drug–receptor interactions and pharmacodynamics
3. Chapter 3: the autonomic nervous system
4. Chapter 4: cholinergic agonists
5. Chapter 5: cholinergic antagonists
6. Chapter 6: adrenergic agonists
7. Chapter 7: adrenergic antagonists
8. Chapter 8: drugs for neurodegenerative diseases
9. Chapter 9: anxiolytic and hypnotic drugs
10. Chapter 10: antidepressants
11. Chapter 11: antipsychotic drugs
12. Chapter 12: drugs for epilepsy
13. Chapter 13: anesthetics
14. Chapter 14: opioids
15. Chapter 15: drugs of abuse
16. Chapter 16: cns stimulants
17. Chapter 17: antihypertensives
18. Chapter 18: diuretics
19. Chapter 19: heart failure
20. Chapter 20: antiarrhythmics
21. Chapter 21: antianginal drugs
22. Chapter 22: anticoagulants and antiplatelet agents
23. Chapter 23: drugs for hyperlipidemia
24. Chapter 24: pituitary and thyroid
25. Chapter 25: drugs for diabetes
26. Chapter 26: estrogens and androgens
27. Chapter 27: adrenal hormones
28. Chapter 28: drugs for obesity
29. Chapter 29: drugs for disorders of the respiratory system
30. Chapter 30: antihistamines
31. Chapter 31: gastrointestinal and antiemetic drugs
32. Chapter 32: drugs for urologic disorders
33. Chapter 33: drugs for anemia
34. Chapter 34: drugs for dermatologic disorders
35. Chapter 35: drugs for bone disorders
36. Chapter 36: anti-inflammatory, antipyretic, and analgesic agents
37. Chapter 37: principles of antimicrobial therapy
38. Chapter 38: cell wall inhibitors
39. Chapter 39: protein synthesis inhibitors
40. Chapter 40: quinolones, folic acid antagonists, and urinary tract antiseptics
41. Chapter 41: antimycobacterial drugs
42. Chapter 42: antifungal drugs
43. Chapter 43: antiprotozoal drugs
44. Chapter 44: anthelmintic drugs
45. Chapter 45: antiviral drugs
46. Chapter 46: anticancer drugs
47. Chapter 47: immunosuppressants
48. Chapter 48: clinical toxicology
,3. The nurse is teaching a patient who will be discharged home with a prescription for an enteric-
coated tablet. Which statement by the patient indicates understanding of the teaching?
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, a. I may crush the tablet and put it in applesauce to improve absorption.
b. I should consume acidic foods to enhance absorption of this medication.
c. I should expect a delay in onset of the drugs effects after taking the tablet.
d. I should take this medication with high-fat foods to improve its action.
Answer: c
Enteric-coated tablets resist disintegration in the acidic environment of the stomach and
disintegrate when they reach the small intestine. There is usually some delay in onset of actions
after taking these medications. Enteric-coated tablets should not be crushed or chewed, which
would alter the time and location of absorption. Acidic foods will not enhance the absorption of
the medication. The patient should not to eat high-fat food before ingesting an enteric-coated
tablet, because high-fat foods decrease the absorption rate.
Dif: cognitive level: applying (application) ref: dm 3 top: nursing
process: nursing intervention
Msc: nclex: physiological integrity: pharmacological and parenteral therapies
4. A patient who is newly diagnosed with type 1 diabetes mellitus asks why insulin must be
given by subcutaneous injection instead of by mouth. The nurse will explain that this is because
a. Absorption is diminished by the first-pass effects in the liver.
b. Absorption is faster when insulin is given subcutaneously.
c. Digestive enzymes in the gastrointestinal tract prevent absorption.
d. The oral form is less predictable with more adverse
effects. Answer: c
Insulin, growth hormones, and other protein-based drugs are destroyed in the small intestine by
digestive enzymes and must be given parenterally. Because insulin is destroyed by digestive
enzymes, it would not make it to the liver for metabolism with a first-pass effect. Subcutaneous
tissue has fewer blood vessels, so absorption is slower in such tissue. Insulin is given
subcutaneously because it is desirable to have it absorb slowly.
Dif: cognitive level: understanding (comprehension) ref: dm 3 top: nursing
process: nursing intervention: patient teaching
Msc: nclex: physiological integrity: pharmacological and parenteral therapies
Page 3 of 507
Plusbay.Plus
Pharmacology, 8th Edition - Whalen, Test
Bank - Chapters 1 – 48 Latest 2026
,Table Of Contents
1. Chapter 1: pharmacokinetics
2. Chapter 2: drug–receptor interactions and pharmacodynamics
3. Chapter 3: the autonomic nervous system
4. Chapter 4: cholinergic agonists
5. Chapter 5: cholinergic antagonists
6. Chapter 6: adrenergic agonists
7. Chapter 7: adrenergic antagonists
8. Chapter 8: drugs for neurodegenerative diseases
9. Chapter 9: anxiolytic and hypnotic drugs
10. Chapter 10: antidepressants
11. Chapter 11: antipsychotic drugs
12. Chapter 12: drugs for epilepsy
13. Chapter 13: anesthetics
14. Chapter 14: opioids
15. Chapter 15: drugs of abuse
16. Chapter 16: cns stimulants
17. Chapter 17: antihypertensives
18. Chapter 18: diuretics
19. Chapter 19: heart failure
20. Chapter 20: antiarrhythmics
21. Chapter 21: antianginal drugs
22. Chapter 22: anticoagulants and antiplatelet agents
23. Chapter 23: drugs for hyperlipidemia
24. Chapter 24: pituitary and thyroid
25. Chapter 25: drugs for diabetes
26. Chapter 26: estrogens and androgens
27. Chapter 27: adrenal hormones
28. Chapter 28: drugs for obesity
29. Chapter 29: drugs for disorders of the respiratory system
30. Chapter 30: antihistamines
31. Chapter 31: gastrointestinal and antiemetic drugs
32. Chapter 32: drugs for urologic disorders
33. Chapter 33: drugs for anemia
34. Chapter 34: drugs for dermatologic disorders
35. Chapter 35: drugs for bone disorders
36. Chapter 36: anti-inflammatory, antipyretic, and analgesic agents
37. Chapter 37: principles of antimicrobial therapy
38. Chapter 38: cell wall inhibitors
39. Chapter 39: protein synthesis inhibitors
40. Chapter 40: quinolones, folic acid antagonists, and urinary tract antiseptics
41. Chapter 41: antimycobacterial drugs
42. Chapter 42: antifungal drugs
43. Chapter 43: antiprotozoal drugs
44. Chapter 44: anthelmintic drugs
45. Chapter 45: antiviral drugs
46. Chapter 46: anticancer drugs
47. Chapter 47: immunosuppressants
48. Chapter 48: clinical toxicology
,3. The nurse is teaching a patient who will be discharged home with a prescription for an enteric-
coated tablet. Which statement by the patient indicates understanding of the teaching?
Page 2 of 507
Plusbay.Plus
, a. I may crush the tablet and put it in applesauce to improve absorption.
b. I should consume acidic foods to enhance absorption of this medication.
c. I should expect a delay in onset of the drugs effects after taking the tablet.
d. I should take this medication with high-fat foods to improve its action.
Answer: c
Enteric-coated tablets resist disintegration in the acidic environment of the stomach and
disintegrate when they reach the small intestine. There is usually some delay in onset of actions
after taking these medications. Enteric-coated tablets should not be crushed or chewed, which
would alter the time and location of absorption. Acidic foods will not enhance the absorption of
the medication. The patient should not to eat high-fat food before ingesting an enteric-coated
tablet, because high-fat foods decrease the absorption rate.
Dif: cognitive level: applying (application) ref: dm 3 top: nursing
process: nursing intervention
Msc: nclex: physiological integrity: pharmacological and parenteral therapies
4. A patient who is newly diagnosed with type 1 diabetes mellitus asks why insulin must be
given by subcutaneous injection instead of by mouth. The nurse will explain that this is because
a. Absorption is diminished by the first-pass effects in the liver.
b. Absorption is faster when insulin is given subcutaneously.
c. Digestive enzymes in the gastrointestinal tract prevent absorption.
d. The oral form is less predictable with more adverse
effects. Answer: c
Insulin, growth hormones, and other protein-based drugs are destroyed in the small intestine by
digestive enzymes and must be given parenterally. Because insulin is destroyed by digestive
enzymes, it would not make it to the liver for metabolism with a first-pass effect. Subcutaneous
tissue has fewer blood vessels, so absorption is slower in such tissue. Insulin is given
subcutaneously because it is desirable to have it absorb slowly.
Dif: cognitive level: understanding (comprehension) ref: dm 3 top: nursing
process: nursing intervention: patient teaching
Msc: nclex: physiological integrity: pharmacological and parenteral therapies
Page 3 of 507
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