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Examen

NSG 3000 Exam 1 Questions and Answers 2026

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1 Where is the site of O2 & CO2 exhange? Pores of Kohn 2 What is the point of surfactant? To keep the aviolli open 3 How much fluid should be in the pleural space? 20-25ml 4 To simply movement of air in and out of the lungs Ventalation 5 Shortness of breath Dyspnea 6 Elastin fibers in the alveolar walls and bronchioles allow the lungs to recoil back after inspiration is over, in essence creating a passive expiration. This is called... Elastic Recoil 7 What two diseases are linked to the lost or decreased ability to have elastic recoil? COPD and Asthma 8 The term used to describe the ease of the lungs to expand with inspiration. Compliance 9 What happens when we have decreased compliance in the lungs? Lungs are more difficult to inflate. Seen with an increase in fluid such as ARDs, PNA, pulmonary edema, or lost elasticity such as in pulmonary fibrosis or sarcoidosis, restricted lung movement such as pleural effusions 10 What happens when we have increased compliance in the lungs? Lungs inflate to easily/there is not enough elasticity. Seen in destroyed alveolar walls such as in COPD

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NSG 3000



NSG 3000 Exam 1 Questions and Answers 2026
1
Where is the site of O2 & CO2 exhange?
Pores of Kohn

2
What is the point of surfactant?
To keep the aviolli open

3
How much fluid should be in the pleural space?
20-25ml

4
To simply movement of air in and out of the lungs
Ventalation

5
Shortness of breath
Dyspnea

6
Elastin fibers in the alveolar walls and bronchioles allow the lungs to recoil back after inspiration
is over, in essence creating a passive expiration. This is called...
Elastic Recoil

7
What two diseases are linked to the lost or decreased ability to have elastic recoil?
COPD and Asthma

8
The term used to describe the ease of the lungs to expand with inspiration.
Compliance

9
What happens when we have decreased compliance in the lungs?
Lungs are more difficult to inflate.
Seen with an increase in fluid such as ARDs, PNA, pulmonary edema, or lost elasticity such as
in pulmonary fibrosis or sarcoidosis, restricted lung movement such as pleural effusions

10
What happens when we have increased compliance in the lungs?
Lungs inflate to easily/there is not enough elasticity. Seen in destroyed alveolar walls such as in
COPD



NSG 3000

, NSG 3000



11
How O2 and CO2 moves across the alveolar-capillary membrane.
Diffusion (high to low concentration)

12
Used to measure arterial oxygenation.
Oximetry

13
Respond to changes in pH, Hydrogen, CO2. Depending on the changes sensed by these
receptors, the medulla will change the respiratory rate.
Chemoreceptors

14
Found in lungs, upper airway, chest wall, and diaphragm and stimulated by various factors to
control respiration. Stimulants can be irritants, muscle stretching, or alveolar wall distortion.
Mechanical Receptors

15
The process where particles and bacteria get stopped as they enter the upper respiratory system
by nasal hairs and mucous.
Filtration

16
Small particles sometimes make it pass filtration and make it down to the aveoli. An example is
coal dust which leads to...
pneumoconiosis (black lung)

17
Mucous secreted -> particles stuck to mucous -> cilia that covers areas below the trachea to
bronchioles (so not in alveoli) beat and move mucous to mouth -> swallowed
This process is called...
Mucociliary Clearance System

18
This is the bodies response to clear large and main airways. It is also considered a backup to the
Mucociliary Clearance System.
Coughing

19
When the bronchi constrict in response to stimuli/particles trying to enter airway, it is called...
Reflex Bronchoconstriction

20
These are located in our alveoli and seek to phagocytize inhaled foreign particles.



NSG 3000

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