CVICU Exam Questions and Verified Answers/Accurate Solutions| New Update Already
Graded A+
SVO2 gives us info about balance between oxygen delivery and oxygen consumption in
the body
SVO2 normal values 60-80%
SVO2 <60% considered low
SVO2 >80% considered high
SVO2 <60% decreased oxygen delivery (oxygen in body is low) increased oxygen
consumption
reasons for decreased O2 delivery (when SVO2 is low) -- anemia, hemorrhage (pt could
be bleeding), hypoxia, heart failure (decreased CO)
reasons for increased O2 consumption (when SVO2 is low) -- agitation, pain, fever
(infection), shivering, respiratory failure, stress/anxiety, bleeding --taking all that it can to
survive
reasons for increased O2 delivery (when SVO2) is high) -- too much oxygen delivery,
blood transfusions, IV fluids, inotropics, increased CO
reasons for decreased O2 consumption (when SVO2 is high) -- sedation/anesthesia,
analgesia (pain medicine), hypothermia, mechanical ventilation, tissues aren't using enough of
the over oxygenation they are getting
SVO2 significance least reliable to be accurate, if all over numbers are WDL except SVO2,
PA/surgeon may be less concerned about it
,mixed venous blood gas this would be drawn from the yellow port on the swan, this is
the only time you would access this port
mixed venous blood gas measures SVO2, respiratory runs this after you draw it from
YELLOW port
preload the amount of blood that fills the ventricle during diastole
CVP central venous pressure
deals with right sided preload CVP
good indicator of right heart function and right sided preload CVP
if the right ventricle is failing, what will happen to CVP? it will rise
normal CVP values 2-6 mmHg
high CVP d/t over-hydration (^ venous return), cardiac tamponade, <3 failure, pulmonary
stenosis (limits venous outflow and leads to venous congestion) positive pressure breathing,
straining, RV failure
what is the goal with high CVP? decreasing patient's preload
How do we treat high CVP? pt's need diuresis, vasodilation (widens vessels), or
treatment for tamponade
, high CVP hypervolemia, RV failure, tamponade, tricuspid insufficiency/stenosis, PE - goal
is to reduce preload
low CVP d/t hypovolemic shock from bleed/hemorrhage, fluid shift, dehydration (dry
patient), negative pressure breathing from when pt demonstrates retractions or mechanical
negative pressure - sometimes used for high spinal cord injury
what is our goal with low CVP? to increase pt's preload
how do we treat low CVP? patient needs to fluids of some kind to increase preload - can
replace volume with fluids, blood, albumin, etc, vasopressors IF needed
recovering heart i am going to exhaust all of my fluid options first to correct my
hypovolemia, then pressors if needed
which port can you draw a CVP from? the blue port on the PA catheter
can you give meds through the blue port? only IV push
afterload resistance ventricles must overcome
what is SVR? systemic vascular resistance
SVR normal values 800-1200
what of the heart does SVR deal with? let sided afterload, this is your systemic
circulation
Graded A+
SVO2 gives us info about balance between oxygen delivery and oxygen consumption in
the body
SVO2 normal values 60-80%
SVO2 <60% considered low
SVO2 >80% considered high
SVO2 <60% decreased oxygen delivery (oxygen in body is low) increased oxygen
consumption
reasons for decreased O2 delivery (when SVO2 is low) -- anemia, hemorrhage (pt could
be bleeding), hypoxia, heart failure (decreased CO)
reasons for increased O2 consumption (when SVO2 is low) -- agitation, pain, fever
(infection), shivering, respiratory failure, stress/anxiety, bleeding --taking all that it can to
survive
reasons for increased O2 delivery (when SVO2) is high) -- too much oxygen delivery,
blood transfusions, IV fluids, inotropics, increased CO
reasons for decreased O2 consumption (when SVO2 is high) -- sedation/anesthesia,
analgesia (pain medicine), hypothermia, mechanical ventilation, tissues aren't using enough of
the over oxygenation they are getting
SVO2 significance least reliable to be accurate, if all over numbers are WDL except SVO2,
PA/surgeon may be less concerned about it
,mixed venous blood gas this would be drawn from the yellow port on the swan, this is
the only time you would access this port
mixed venous blood gas measures SVO2, respiratory runs this after you draw it from
YELLOW port
preload the amount of blood that fills the ventricle during diastole
CVP central venous pressure
deals with right sided preload CVP
good indicator of right heart function and right sided preload CVP
if the right ventricle is failing, what will happen to CVP? it will rise
normal CVP values 2-6 mmHg
high CVP d/t over-hydration (^ venous return), cardiac tamponade, <3 failure, pulmonary
stenosis (limits venous outflow and leads to venous congestion) positive pressure breathing,
straining, RV failure
what is the goal with high CVP? decreasing patient's preload
How do we treat high CVP? pt's need diuresis, vasodilation (widens vessels), or
treatment for tamponade
, high CVP hypervolemia, RV failure, tamponade, tricuspid insufficiency/stenosis, PE - goal
is to reduce preload
low CVP d/t hypovolemic shock from bleed/hemorrhage, fluid shift, dehydration (dry
patient), negative pressure breathing from when pt demonstrates retractions or mechanical
negative pressure - sometimes used for high spinal cord injury
what is our goal with low CVP? to increase pt's preload
how do we treat low CVP? patient needs to fluids of some kind to increase preload - can
replace volume with fluids, blood, albumin, etc, vasopressors IF needed
recovering heart i am going to exhaust all of my fluid options first to correct my
hypovolemia, then pressors if needed
which port can you draw a CVP from? the blue port on the PA catheter
can you give meds through the blue port? only IV push
afterload resistance ventricles must overcome
what is SVR? systemic vascular resistance
SVR normal values 800-1200
what of the heart does SVR deal with? let sided afterload, this is your systemic
circulation
