Nurs 615 Exam IV Verified Exam Questions and
Answers 2026/2027
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Terms in this set (81)
How will you prescribe lipase, Patients with cystic fibrosis are often prescribed
protease, and amylase components? enzyme replacement for pancreatic secretions each
replacement drug has amylase, lipase and protease
components, however the drug is prescribed in units
of lipase
,What is the medication of choice for Surgical resection of the tumor is the first treatment of
hypertensive crisis with choice either my open laparotomy or laparoscopy
pheochromocytoma? either surgical option requires prior treatment of
nonspecific irreversible adrenergic adraonoreceptor
blocker phenoxybenzamine or a shorter acting alpha
antagonists, prazosin, terazosin, and doxazosin.
Mainly use phenozibenamine in practice. Doing so
promotes the surgery to proceed while minimizing the
likelihood of severe intraoperative hypertension which
is likely when the tumor is manipulated.
What is the onset of action, peak of (Intermediate Acting) NPH
action, and duration of action of each Onset-60-90 min after administration,
insulin preparation? Peak 48 hrs
Duration 10-18 hrs.
(Short Acting) Regular Onset 30-60 min
Peak 2-4 hrs
Duration 6-10 hrs
(Long Acting) Aspart, Lispro, Glulisine
Onset less than 15 min
Peak 1-2 hrs
Duration 3-6 hrs
(Long Acting) Glargine, Detemir
Onset 1-2 hrs
Peak NO PEAK
Duration 24 hrs
, Identify the symptoms of Hypoglycemia- dizziness, confusion, diaphoresis,
hypoglycemia, hyperglycemia, and tachycardia
ketoacidosis. Hyperglycemia- polyphagia, polydipsia, polyuria,
blurred vision, and fatigue
Ketoacidosis- hallmark symptoms include acetone
breath like nail polish remover or fruity breath. Also
abdominal pain, nausea, vomiting and sob.
When changing from NPH to glargine The initial dose of glargine is reduced by 20% to
insulin, how will you adjust the patient's prevent hypoglycemia.
dose?
How does metformin work? Decreases hyperglycemia by decreasing hepatic
glucose production called hepatic gluconeogenesis.
The average person with type 2 diabetes has three
times the rate of gluconeogenesis, metformin
treatment reduces this by over 1/3rd. The molecular
mechanism of metformin isn't completely understood.
In addition to suppressing hepatic glucose
production, metformin increases insulin sensitivity,
enhances peripheral glucose uptake by inducing the
phosphorilization of glu4 enhancer factor, decreases
insulin induced suppression of fatty acid oxidation,
and decreases absorption of glucose from the GI
tract. Also of note* Metformin helps reduce LDL
cholesterol and triglyceride levels and is not
associated with weight gain, in some people it helps
promote weight loss*
What diagnostic testing is required Metformin is not metabolized, it is cleared from the
before and throughout therapy with body by tubular secretion and is secreted unchanged
metformin? in the urine. Metformin is undetectable in blood
plasma within 24 hrs of a single oral dose the average
elimination half-life in plasma is 6.2 hrs as it is secreted
in the urine you should check a serum crt to assess
renal function.
Answers 2026/2027
Play your way to mastery with fun games
Match Blocks Charms NEW
Terms in this set (81)
How will you prescribe lipase, Patients with cystic fibrosis are often prescribed
protease, and amylase components? enzyme replacement for pancreatic secretions each
replacement drug has amylase, lipase and protease
components, however the drug is prescribed in units
of lipase
,What is the medication of choice for Surgical resection of the tumor is the first treatment of
hypertensive crisis with choice either my open laparotomy or laparoscopy
pheochromocytoma? either surgical option requires prior treatment of
nonspecific irreversible adrenergic adraonoreceptor
blocker phenoxybenzamine or a shorter acting alpha
antagonists, prazosin, terazosin, and doxazosin.
Mainly use phenozibenamine in practice. Doing so
promotes the surgery to proceed while minimizing the
likelihood of severe intraoperative hypertension which
is likely when the tumor is manipulated.
What is the onset of action, peak of (Intermediate Acting) NPH
action, and duration of action of each Onset-60-90 min after administration,
insulin preparation? Peak 48 hrs
Duration 10-18 hrs.
(Short Acting) Regular Onset 30-60 min
Peak 2-4 hrs
Duration 6-10 hrs
(Long Acting) Aspart, Lispro, Glulisine
Onset less than 15 min
Peak 1-2 hrs
Duration 3-6 hrs
(Long Acting) Glargine, Detemir
Onset 1-2 hrs
Peak NO PEAK
Duration 24 hrs
, Identify the symptoms of Hypoglycemia- dizziness, confusion, diaphoresis,
hypoglycemia, hyperglycemia, and tachycardia
ketoacidosis. Hyperglycemia- polyphagia, polydipsia, polyuria,
blurred vision, and fatigue
Ketoacidosis- hallmark symptoms include acetone
breath like nail polish remover or fruity breath. Also
abdominal pain, nausea, vomiting and sob.
When changing from NPH to glargine The initial dose of glargine is reduced by 20% to
insulin, how will you adjust the patient's prevent hypoglycemia.
dose?
How does metformin work? Decreases hyperglycemia by decreasing hepatic
glucose production called hepatic gluconeogenesis.
The average person with type 2 diabetes has three
times the rate of gluconeogenesis, metformin
treatment reduces this by over 1/3rd. The molecular
mechanism of metformin isn't completely understood.
In addition to suppressing hepatic glucose
production, metformin increases insulin sensitivity,
enhances peripheral glucose uptake by inducing the
phosphorilization of glu4 enhancer factor, decreases
insulin induced suppression of fatty acid oxidation,
and decreases absorption of glucose from the GI
tract. Also of note* Metformin helps reduce LDL
cholesterol and triglyceride levels and is not
associated with weight gain, in some people it helps
promote weight loss*
What diagnostic testing is required Metformin is not metabolized, it is cleared from the
before and throughout therapy with body by tubular secretion and is secreted unchanged
metformin? in the urine. Metformin is undetectable in blood
plasma within 24 hrs of a single oral dose the average
elimination half-life in plasma is 6.2 hrs as it is secreted
in the urine you should check a serum crt to assess
renal function.
