TEST BANK for RUBIN'SPATHOLOGY: CLINICOPATHOLOGIC FOUNDATIONSOFMEDICINE 7th Edition by Strayer and Rubin All chapters 1-34 covered

TEST BANK
RUBIN'S PATHOLOGY: CLINICOPATHOLOGIC
FOUNDATIONS OF MEDICINE
7th Edition by Strayer and Rubin
All chapters 1-34 covered

,TestHBankHRubin'sHPathology:HClinicopathologicHFoundationsHofHMedicineH7thH Edition

TableHofHContents:
ChapterH1:HCellHAdaptation,HInjuryHandHDeathH
ChapterH2:HInflammation
ChapterH3:HRepair,HRegenerationHandHFibrosisH
ChapterH4:HImmunopathology
ChapterH5:HNeoplasia
ChapterH6:HDevelopmentalHandHGeneticHDiseas
esH ChapterH7:HHemodynamicHDisorders
ChapterH8:HEnvironmentalHandHNutritionalHPathologyH Chap
terH9:HInfectiousHandHParasiticHDiseases
SectionHII:HPathogenesisHofHSystemicHConditionsHExpandableHsectio
nH ChapterH10:HAging
ChapterH11:HSystemicHAutoimmuneHDiseasesH
ChapterH12:HSepsis
ChapterH13:HObesityHandHDiabetesHMellitusH Ch
apterH14:HTheHPathologyHofHPregnancyH Chapter
H15:HTheHAmyloidoses

SectionHIII:HDiseasesHofHIndividualHOrganHSystemsExpandableHsectio
nH ChapterH16:HBloodHVessels
ChapterH17:HTheHHeart
ChapterH18:HTheHRespiratoryHSystemH Chap
terH19:HTheHGastrointestinalHTractH Chapter
H20:HTheHLiverHandHBiliaryHSystemH Chapter

H21:HTheHPancreas

ChapterH22:HTheHKidney
ChapterH23:HTheHLowerHUrinaryHTractHandHMaleHReproductiveHSyste
mH ChapterH24:HTheHFemaleHReproductiveHSystemHandHPeritoneumH
ChapterH25:HTheHBreast
ChapterH26:HHematopathologyH Chap
terH27:HTheHEndocrineHSystemH Chap
terH28:HTheHSkin
ChapterH29:HTheHHeadHandHNeck
ChapterH30:HBones,HJointsHandHSoftHTissue
ChapterH31:HSkeletalHMuscleHandHPeripheralHNervousHSystemH Ch
apterH32:HTheHCentralHNervousHSystem
ChapterH33:HTheHEye
ChapterH34:HForensicHPathology

,Rubin'sHPathology:HClinicopathologicHFoundationsHofHMedicineH Chapt
erH1:HCellHAdaptation,HInjuryHandHDeath

IschemiaHandHotherHtoxicHinjuriesHincreaseHtheHaccumulationHofHintracellularHcalciumHasHaHresu
1. of:

A) releaseHofHstoredHcalciumHfromHtheHmitochondria.
B) improvedHintracellularHvolumeHregulation.
C) decreasedHinfluxHacrossHtheHcellHmembrane.
D) attractionHofHcalciumHtoHfattyHinfiltrates.
TheHpatientHisHfoundHtoHhaveHliverHdisease,HresultingHinHtheHremovalHofHaHlobeHofHhisHliver.
2. AdaptationHtoHtheHreducedHsizeHofHtheHliverHleadsHtoH_ ofHtheHremainingHliverHcells.
A) metaplasia
B) organHatrophy
C) compensatoryHhyperplasia
D) physiologicHhypertrophy
AHpersonHeatingHpeanutsHstartsHchokingHandHcollapses.HHisHairwayHobstructionHisHpartiallyH cl
eared,HbutHheHremainsHhypoxicHuntilHheHreachesHtheHhospital.HTheHprolongedHcellHhypoxia
3. causedHaHcerebralHinfarctionHandHresulting _HinHtheHbrain.
A) caspaseHactivation
B) coagulationHnecrosis
C) rapidHphagocytosis
D) proteinHp53Hdeficiency
BacteriaHandHvirusesHcauseHcellHdamageHby ,HwhichHisHuniqueHfromHtheHintracellular
4. damageHcausedHbyHotherHinjuriousHagents.
A) disruptingHtheHsodium/potassiumHATPaseHpump
B) interruptingHoxidativeHmetabolismHprocesses
C) replicatingHandHproducingHcontinuedHinjury
D) decreasingHproteinHsynthesisHandHfunction
TheHpatientHhasHaHprolongedHinterruptionHinHarterialHbloodHflowHtoHhisHleftHkidney,Hcausing
5. hypoxicHcellHinjuryHandHtheHreleaseHofHfreeHradicals.HFreeHradicalsHdamageHcellsHby:
A) destroyingHphospholipidsHinHtheHcellHmembrane.
B) alteringHtheHimmuneHresponseHofHtheHcell.
C) disruptingHcalciumHstorageHinHtheHcell.
D) inactivationHofHenzymesHandHmitochondria.

, 6. InjuredHcellsHhaveHimpairedHflowHofHsubstancesHthroughHtheHcellHmembraneHasHaHresultHof:
A) increasedHfatHload.
B) alteredHpermeability.
C) alteredHglucoseHutilization.
D) increasedHsurfaceHreceptors.
7. ReversibleHadaptiveHintracellularHresponsesHareHinitiatedHby:
A) stimulusHoverload.
B) geneticHmutations.
C) chemicalHmessengers.
D) mitochondrialHDNA.
8. InjuredHcellsHbecomeHveryHswollenHasHaHresultHof:
A) increasedHcellHproteinHsynthesis.
B) alteredHcellHvolumeHregulation.
C) passiveHentryHofHpotassiumHintoHtheHcell.
D) blebHformationHinHtheHplasmaHmembrane.
AHdiabeticHpatientHhasHimpairedHsensation,Hcirculation,HandHoxygenationHofHhisHfeet.HHeHstep
sHonH aHpieceHofHglass,HtheHwoundHdoesHnotHheal,HandHtheHareaHtissueHbecomesHnecrotic.HTh
eHnecrotic
9. cellHdeathHisHcharacterizedHby:
A) rapidHapoptosis.
B) cellularHrupture.
C) shrinkageHandHcollapse.
D) chronicHinflammation.
AH99-year-oldHwomanHhasHexperiencedHtheHdeclineHofHcellHfunctionHassociatedHwithHage.HA
10 groupHofHtheoriesHofHcellularHagingHfocusHonHprogrammed:
.
A) changesHwithHgeneticHinfluences.
B) eliminationHofHcellHreceptorHsites.
C) insufficientHtelomeraseHenzyme.
D) DNAHmutationHorHfaultyHrepair.
AnH89-year-
oldHfemaleHpatientHhasHexperiencedHsignificantHdecreasesHinHherHmobilityHandH staminaHduri
ngHaH3-
weekHhospitalHstayHforHtheHtreatmentHofHaHfemoralHheadHfracture.HWhichHofH theHfollowingHp
henomenaHmostHlikelyHaccountsHforHtheHpatientsHdecreaseHinHmuscleHfunction
11 thatHunderliesHherHreducedHmobility?
.