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NR565 Pharm Htn/Cardiac/Arrthymias – Questions & Solutions

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NR565 Pharm Htn/Cardiac/Arrthymias – Questions & Solutions

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NR565 Pharm Htn/Cardiac/Arrthymias – Questions
& Solutions

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Practice questions for this set


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reduce BP, HR, or SVR



Choose an answer



1 htn med classes (7) 2 arbs contraindications



3 ACE inhibitors contraindications 4 what does ranolazine NOT do



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Terms in this set (75)



htn med classes (7) -angiotensin-converting enzyme (ACE) inhibitors
-angiotensin receptor blockers (ARBs)
- beta blockers
- calcium channel blockers
- loop diuretics
- thiazide diuretic
- potassium sparing diuretics

, angtiotensin converting enzyme - targets RAAS
MOA, electrolytes -inhibit angiotensin 1 from converting to angiotensin
II - causes vasodilation, reduced aldosterone,
potassium retention
- increase in bradykinin: cough, angioedema


ace inhibitors indicated for HTN, post MI, reduce CV risk, HF, diabetic
nephropathy


ACE inhibitors affect CO how increased CO by decreasing afterload; makes it
easier to pump and increases stroke volume


ACE inhibitors contraindications pregnancy, poor renal function


ace inhibitors special cough from increase in bradykinin, can cause first
dose hypotension, hyperK risk


ace inhibitors electrolytes hyperK


ARBs MOA block angiotensin II at receptor level, cannot bind
- decreased aldosterone --> vasodilation, lower BP


ARBs indication HTN (when ace is not tolerated)
HF
diabetic nepropathy
post MI


ARBS effect on CO improve CO by decreasing SVR


ARBs electrolytes hyperK


arbs adverse effects hyperK
angioedema from increased cap perm
renal issues

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