acomplete
thesentence
a acylcnainsarecarriedby AKAB
acidoxidation
intat.ly oxidationcoenzymes.co
b acylcnainsarecarriedby intattyaciasyntne.is carrier
acyl proteins
ii iiiiiiiii.itiiii iit i ia i i ii ii mmmmmmines
Initialteam
Iii iiinohgi.in
Ii
iiit.si ii it
iiai mT r ne
gg
r
Seventually
will
É
switchplaseswl
HsAcpion
synthase
fi
maionynon.catmzedbyatransacylase
a whereinmesellwouldacetyrcoacarboxylaseoccuriorfasynmesi.in general
ofanabolicactivities
inthecytosolalot
takepartinmecytoso o.Howaidacetyl
con
getintothecytosol.my're
in mitochondria
located the
loncanget.inocytosolascitrateviathecitratetransport system
acetyl
waeE.fi tiiiii
Fincetate
f
i matatgnppb.it
mrnuate ipyrnate G.thp.io
Hey
p
mitochondria cytosol
aAfter viaasetyl.comcarboxylasewhat
malonylloaismadewlnsetylcon next
happens
i 2transacylation
unoccurs mine
iii iii iii
ii iinii.fi t.fi i iiitii
Iiii.isiiiiti
acpis
Q.Afteracetoacetyl formedwhat next
happens
ofBketogroupwNADPH
ooo
reduction
at
mail.fiaanaifftwnetf EYRE
a ini i
for bond
dehydration double formation
oooo
fi t.it µ
of
reduction double
bond NADPH w Mumm
HERE
Mfff
fi.fiianifi
FA
mm mm
extended transferred
to
mceatalytic
cysteine
,asoifyouputallthestepstogether.wnatwouldfat.ly
Acid like
look
synthesis
iacetyl
ioaundergoestransacetylationtobeaddedtocatalyticcystei.n t
it i.F Mmmmm
1
ioa
2malonyl undergoes tobeaddedtoAC
transacetylation
ion
malonyl
si iii ti
acp
malonyl
s .fi deaf
or
3the
m alonyl acpattacksmemoleculeonactivecysteinetoregenitbacitoits.gs
AI
Mo Tu
itif.tini itiii m
fffmit
spfp .it
4 reductionofpretogroupwinana
iii anti
painaincombe
extended'onserman
for
5dehydration double
bond
formation
mononaria
a.milffaipjjt
f additional
enzymeslikedesatura.es willprocessthe
no
IT nn.fi Twaip
add
Facnain.desamratingbiomolec.to bonds
d ouble
7extendedFashaindisplacedto
catalytic
cysteine nowmecyaecanrepeatwlmaionylto.net
addedtomeacpmencnainextendedi.me maxlengtnincytosolwouldbe161
sooncef y.fi
ggspygppgggpyi
fit
synmesisconcludesathiolasewillhydrolyzeme
as.ysps p s
p
gagging
FA the
from
chain c atalytic
cysteine
beingaadedinounsaturatedbotsietygp.iq
pipinated
cbingefrat.li'sisa
carboxulatio o.Howarelipidsdesaturated occursacarbonistostasco
are are
NADPH biomolecules
required 450 required
for enzymes
c ofactors desamrating
2H 2cytie FAD NADPH
Ilya
NADP unsTahon
cytes t.am
a onmetopicofpyso.whycanatolerancetobarbituratearna.ieadtooverdosing
the will
liver make enzyme p450
containing when e thanol to
orbarbituratesareingestedinordertobreakmem.itsomeonewere
takenlotofthosedrugsmeliverwouldmakemoreof.me
p450containingenzymesbut initial
the dose
might.be
oomuc formebraintohandlewhichcouldleadtotheperson
overdosing
doesitmean
awhat for
cellular tovary
localization across
kingdoms
meenanimaisormantsmeseroeationswinaitter
iiiiii.iniainiIiiii'iiiits is
plantsioccurs
t inperoxisomes
FAbiosynthesis inanimalswilloccurinmecytosol.but.in
plantsit
occursin
the chloroplast
, awhat
arethe
different is in
NADPH produced a nimals
ways about
what plants
animals iPentosephosphatepathwayppp
thinkin.tninidethiYeiiifah9nnarogenase
i malicenzymeincitrate
transport
system
malate
warmigituate
topowerNADPHsynthesis
used
plantslight
H2ONADP 202
NADPH 99951
gs
hlhnhYelaphpfbstem a.wnyaomese
pathways
regulation sense
make
anabundant
having amountofcystoliccitratemeansthatmecellissatiateda.es
the is
mitochondriabacked
upnot mitochondrial
using fast
citrate so.me
enough
iiverwillnowtocusonstoragesuchasfasynmes.is
xsFAwouldleadtoanegativefeedbackloopofasetyt.iocarboxylase asit
naiiiaiiiiiaaiiim.int
maonymoameansmatmecenissatiated.es
themitochondviaisnotusingciat
for
ibutratnerexportingit.thi's signals biomolecules
like
FAtonotbreakdownasmeliverdoesntneedtomakeatern li
veronly
intheliverglucagonepinephrinewanistoebloodsugar.sothdoesmis.by
activatinga mat
cascade
signaling pkavia
activates camp bindingwhichthen
theF26Bpaseefkacomplex.ptczisinactivatedwhichallows
phosphorylates
forgluconeogenesistobefavoredthis
also
meansother metabolicpathways
likefasynmesiswinbehaltedinfavorof.br breakdownexporting
omolecule
thesellcanttocusonstoringxsmaterialsinmeformof.in when
mose
resources
be by
can used starved
cellsGlycolysis favoredmake
usually a cetylirator to
would
epinephrine
hYaFzed
befold't's FAsyntgy.snYf9ulagon
liveronly
inthe
liver
insulin
signals
m atbloodsugaristoonighithatmusaecellsaresatiated.no
liver
can
theliverwillfocusongimoseuptakeistorage.misalsomean.me resources
allocate
toFAsynmesistoo.matway.escarbohydratesamino
acids be
can for term
stored long
mmmm
liveronly
aHowcanbeingd iabetic FAsynthesis
i nterrupt
inanormalpersoninsulinwould leadthe
to liver
into
switching u ptakeconservation For
mode those
arediabetic.melivercant
who toinsulinthis
respond
that
means me liver know
cannot when are
cells
already is
satiated always
i nanonconservationmode
thatiswhy inthepresenceofinsulindiabetic
even
will to
blood
high
persons continuehave as
sugar they
areusingresourcesfor
ketone orgiulose
body exporting
purposesNot storage
longterm
thesentence
a acylcnainsarecarriedby AKAB
acidoxidation
intat.ly oxidationcoenzymes.co
b acylcnainsarecarriedby intattyaciasyntne.is carrier
acyl proteins
ii iiiiiiiii.itiiii iit i ia i i ii ii mmmmmmines
Initialteam
Iii iiinohgi.in
Ii
iiit.si ii it
iiai mT r ne
gg
r
Seventually
will
É
switchplaseswl
HsAcpion
synthase
fi
maionynon.catmzedbyatransacylase
a whereinmesellwouldacetyrcoacarboxylaseoccuriorfasynmesi.in general
ofanabolicactivities
inthecytosolalot
takepartinmecytoso o.Howaidacetyl
con
getintothecytosol.my're
in mitochondria
located the
loncanget.inocytosolascitrateviathecitratetransport system
acetyl
waeE.fi tiiiii
Fincetate
f
i matatgnppb.it
mrnuate ipyrnate G.thp.io
Hey
p
mitochondria cytosol
aAfter viaasetyl.comcarboxylasewhat
malonylloaismadewlnsetylcon next
happens
i 2transacylation
unoccurs mine
iii iii iii
ii iinii.fi t.fi i iiitii
Iiii.isiiiiti
acpis
Q.Afteracetoacetyl formedwhat next
happens
ofBketogroupwNADPH
ooo
reduction
at
mail.fiaanaifftwnetf EYRE
a ini i
for bond
dehydration double formation
oooo
fi t.it µ
of
reduction double
bond NADPH w Mumm
HERE
Mfff
fi.fiianifi
FA
mm mm
extended transferred
to
mceatalytic
cysteine
,asoifyouputallthestepstogether.wnatwouldfat.ly
Acid like
look
synthesis
iacetyl
ioaundergoestransacetylationtobeaddedtocatalyticcystei.n t
it i.F Mmmmm
1
ioa
2malonyl undergoes tobeaddedtoAC
transacetylation
ion
malonyl
si iii ti
acp
malonyl
s .fi deaf
or
3the
m alonyl acpattacksmemoleculeonactivecysteinetoregenitbacitoits.gs
AI
Mo Tu
itif.tini itiii m
fffmit
spfp .it
4 reductionofpretogroupwinana
iii anti
painaincombe
extended'onserman
for
5dehydration double
bond
formation
mononaria
a.milffaipjjt
f additional
enzymeslikedesatura.es willprocessthe
no
IT nn.fi Twaip
add
Facnain.desamratingbiomolec.to bonds
d ouble
7extendedFashaindisplacedto
catalytic
cysteine nowmecyaecanrepeatwlmaionylto.net
addedtomeacpmencnainextendedi.me maxlengtnincytosolwouldbe161
sooncef y.fi
ggspygppgggpyi
fit
synmesisconcludesathiolasewillhydrolyzeme
as.ysps p s
p
gagging
FA the
from
chain c atalytic
cysteine
beingaadedinounsaturatedbotsietygp.iq
pipinated
cbingefrat.li'sisa
carboxulatio o.Howarelipidsdesaturated occursacarbonistostasco
are are
NADPH biomolecules
required 450 required
for enzymes
c ofactors desamrating
2H 2cytie FAD NADPH
Ilya
NADP unsTahon
cytes t.am
a onmetopicofpyso.whycanatolerancetobarbituratearna.ieadtooverdosing
the will
liver make enzyme p450
containing when e thanol to
orbarbituratesareingestedinordertobreakmem.itsomeonewere
takenlotofthosedrugsmeliverwouldmakemoreof.me
p450containingenzymesbut initial
the dose
might.be
oomuc formebraintohandlewhichcouldleadtotheperson
overdosing
doesitmean
awhat for
cellular tovary
localization across
kingdoms
meenanimaisormantsmeseroeationswinaitter
iiiiii.iniainiIiiii'iiiits is
plantsioccurs
t inperoxisomes
FAbiosynthesis inanimalswilloccurinmecytosol.but.in
plantsit
occursin
the chloroplast
, awhat
arethe
different is in
NADPH produced a nimals
ways about
what plants
animals iPentosephosphatepathwayppp
thinkin.tninidethiYeiiifah9nnarogenase
i malicenzymeincitrate
transport
system
malate
warmigituate
topowerNADPHsynthesis
used
plantslight
H2ONADP 202
NADPH 99951
gs
hlhnhYelaphpfbstem a.wnyaomese
pathways
regulation sense
make
anabundant
having amountofcystoliccitratemeansthatmecellissatiateda.es
the is
mitochondriabacked
upnot mitochondrial
using fast
citrate so.me
enough
iiverwillnowtocusonstoragesuchasfasynmes.is
xsFAwouldleadtoanegativefeedbackloopofasetyt.iocarboxylase asit
naiiiaiiiiiaaiiim.int
maonymoameansmatmecenissatiated.es
themitochondviaisnotusingciat
for
ibutratnerexportingit.thi's signals biomolecules
like
FAtonotbreakdownasmeliverdoesntneedtomakeatern li
veronly
intheliverglucagonepinephrinewanistoebloodsugar.sothdoesmis.by
activatinga mat
cascade
signaling pkavia
activates camp bindingwhichthen
theF26Bpaseefkacomplex.ptczisinactivatedwhichallows
phosphorylates
forgluconeogenesistobefavoredthis
also
meansother metabolicpathways
likefasynmesiswinbehaltedinfavorof.br breakdownexporting
omolecule
thesellcanttocusonstoringxsmaterialsinmeformof.in when
mose
resources
be by
can used starved
cellsGlycolysis favoredmake
usually a cetylirator to
would
epinephrine
hYaFzed
befold't's FAsyntgy.snYf9ulagon
liveronly
inthe
liver
insulin
signals
m atbloodsugaristoonighithatmusaecellsaresatiated.no
liver
can
theliverwillfocusongimoseuptakeistorage.misalsomean.me resources
allocate
toFAsynmesistoo.matway.escarbohydratesamino
acids be
can for term
stored long
mmmm
liveronly
aHowcanbeingd iabetic FAsynthesis
i nterrupt
inanormalpersoninsulinwould leadthe
to liver
into
switching u ptakeconservation For
mode those
arediabetic.melivercant
who toinsulinthis
respond
that
means me liver know
cannot when are
cells
already is
satiated always
i nanonconservationmode
thatiswhy inthepresenceofinsulindiabetic
even
will to
blood
high
persons continuehave as
sugar they
areusingresourcesfor
ketone orgiulose
body exporting
purposesNot storage
longterm
