Test bank for Rubin s pathology clinicopathologic foundations of medicine 7th edition by David S. Strayer, Emanuel Rubin Course

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RUBIN'S PATHOLOGY: CLINICOPATHOLOGIC FOUNDATIONS OF MEDICIN
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E 7TH EDITION
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DAVID S. STRAYER, EMANUEL RUBIN
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,TestvBankvRubin'svPathology:vClinicopathologicvFoundationsvofvMedicinev7thvEdition

TablevofvContents:
Chapterv1:vCellvAdaptation,vInjuryvandvDeath
Chapterv2:vInflammation
Chapterv3:vRepair,vRegenerationvandvFibrosis
Chapterv4:vImmunopathology
Chapterv5:vNeoplasia
Chapterv6:vDevelopmentalvandvGeneticvDiseases
Chapterv7:vHemodynamicvDisorders
Chapterv8:vEnvironmentalvandvNutritionalvPathology
Chapterv9:vInfectiousvandvParasiticvDiseases
SectionvII:vPathogenesisvofvSystemicvConditionsvExpandablevsection
Chapterv10:vAging
Chapterv11:vSystemicvAutoimmunevDiseases
Chapterv12:vSepsis
Chapterv13:vObesityvandvDiabetesvMellitus
Chapterv14:vThevPathologyvofvPregnancy
Chapterv15:vThevAmyloidoses
SectionvIII:vDiseasesvofvIndividualvOrganvSystemsExpandablevsection
Chapterv16:vBloodvVessels
Chapterv17:vThevHeart
Chapterv18:vThevRespiratoryvSystem
Chapterv19:vThevGastrointestinalvTract
Chapterv20:vThevLivervandvBiliaryvSystem
Chapterv21:vThevPancreas
Chapterv22:vThevKidney
Chapterv23:vThevLowervUrinaryvTractvandvMalevReproductivevSystem
Chapterv24:vThevFemalevReproductivevSystemvandvPeritoneum
Chapterv25:vThevBreast
Chapterv26:vHematopathology
Chapterv27:vThevEndocrinevSystem
Chapterv28:vThevSkin
Chapterv29:vThevHeadvandvNeck
Chapterv30:vBones,vJointsvandvSoftvTissue
Chapterv31:vSkeletalvMusclevandvPeripheralvNervousvSystem
Chapterv32:vThevCentralvNervousvSystem
Chapterv33:vThevEye
Chapterv34:vForensicvPathology

,Rubin'svPathology:vClinicopathologicvFoundationsvofvM
edicineChapterv1:vCellvAdaptation,vInjuryvandvDeath

Ischemiavandvothervtoxicvinjuriesvincreasevthevaccumulationvofvintracellularvcalciumvasvavresult
1. of:
A) releasevofvstoredvcalciumvfromvthevmitochondria.
B) improvedvintracellularvvolumevregulation.
C) decreasedvinfluxvacrossvthevcellvmembrane.
D) attractionvofvcalciumvtovfattyvinfiltrates.
Thevpatientvisvfoundvtovhavevlivervdisease,vresultingvinvthevremovalvofvavlobevofvhisvliver.
2. Adaptationvtovthevreducedvsizevofvthevlivervleadsvtov_ ofvthevremainingvlivervcells.
A) metaplasia
B) organvatrophy
C) compensatoryvhyperplasia
D) physiologicvhypertrophy
Avpersonveatingvpeanutsvstartsvchokingvandvcollapses.vHisvairwayvobstructionvisvpartiallyvcleared
,vbutvhevremainsvhypoxicvuntilvhevreachesvthevhospital.vThevprolongedvcellvhypoxia
3. causedvavcerebralvinfarctionvandvresulting _vinvthevbrain.
A) caspasevactivation
B) coagulationvnecrosis
C) rapidvphagocytosis
D) proteinvp53vdeficiency
Bacteriavandvvirusesvcausevcellvdamagevby ,vwhichvisvuniquevfromvthevintracellular
4. damagevcausedvbyvothervinjuriousvagents.
A) disruptingvthevsodium/potassiumvATPasevpump
B) interruptingvoxidativevmetabolismvprocesses
C) replicatingvandvproducingvcontinuedvinjury
D) decreasingvproteinvsynthesisvandvfunction
Thevpatientvhasvavprolongedvinterruptionvinvarterialvbloodvflowvtovhisvleftvkidney,vcausing
5. hypoxicvcellvinjuryvandvthevreleasevofvfreevradicals.vFreevradicalsvdamagevcellsvby:
A) destroyingvphospholipidsvinvthevcellvmembrane.
B) alteringvthevimmunevresponsevofvthevcell.
C) disruptingvcalciumvstoragevinvthevcell.
D) inactivationvofvenzymesvandvmitochondria.

, 6. Injuredvcellsvhavevimpairedvflowvofvsubstancesvthroughvthevcellvmembranevasvavresultvof:
A) increasedvfatvload.
B) alteredvpermeability.
C) alteredvglucosevutilization.
D) increasedvsurfacevreceptors.
7. Reversiblevadaptivevintracellularvresponsesvarevinitiatedvby:
A) stimulusvoverload.
B) geneticvmutations.
C) chemicalvmessengers.
D) mitochondrialvDNA.
8. Injuredvcellsvbecomevveryvswollenvasvavresultvof:
A) increasedvcellvproteinvsynthesis.
B) alteredvcellvvolumevregulation.
C) passiveventryvofvpotassiumvintovthevcell.
D) blebvformationvinvthevplasmavmembrane.
Avdiabeticvpatientvhasvimpairedvsensation,vcirculation,vandvoxygenationvofvhisvfeet.vHevstepsvonvav
piecevofvglass,vthevwoundvdoesvnotvheal,vandvthevareavtissuevbecomesvnecrotic.vThevnecrotic
9. cellvdeathvisv characterizedvby:
A) rapidvapoptosis.
B) cellularvrupture.
C) shrinkagevandvcollapse.
D) chronicvinflammation.
Av99-year-oldvwomanvhasvexperiencedvthevdeclinevofvcellvfunctionvassociatedvwithvage.vA
10. groupvofvtheoriesvofvcellularvagingvfocusvonvprogrammed:
A) changesvwithvgeneticvinfluences.
B) eliminationvofvcellvreceptorvsites.
C) insufficientvtelomerasevenzyme.
D) DNAvmutationvorvfaultyvrepair.
Anv89-year-
oldvfemalevpatientvhasvexperiencedvsignificantvdecreasesvinvhervmobilityvandvstaminavduringvav3
-
weekvhospitalvstayvforvthevtreatmentvofvavfemoralvheadvfracture.vWhichvofvthevfollowingvphenom
enavmostvlikelyvaccountsvforvthevpatientsvdecreasevinvmusclevfunction
11. thatvunderliesvhervreducedvmobility?
A) Impairedvmusclevcellvmetabolismvresultingvfromvmetaplasia
B) Dysplasiavasvavconsequencevofvinflammationvduringvbonevremodeling
C) Disusevatrophyvofvmusclev cellsvduringvavprolongedvperiodvofvimmobility