NR 571 Final exam with verified answers
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Esophageal |rupture***, |pancreatic |and |malignant |effusions |are |indicated |when |PF |shows |what? |- |
ANSWER-Amylase
PF |triglyceride |level |>100 |mg/dL |suggests |a |___________ |- |ANSWER-Chylothorax
Hemolysis
Gilbert's |syndrome
Drugs
Hepatitis
Chemotherapy
Neonatal
Obstructive |cholecystokinin |
Cirrhosis |with |liver |failure |
Are |causes |of |what? |- |ANSWER-Hyperbilirubinemia
Alk |Phos |can |be |elevated |with |pregnancy, |bone |disease |or |growing |children |
T/f |- |ANSWER-True
True |test |of |liver |function |is... |- |ANSWER-Total |protein/albumin
PT/INR
AST |is |not |very |specific |for |liver |disease |except |in... |- |ANSWER-ETOH
, To |determine |if |the |source |of |high |Alk |Phos |is |from |the |liver, |you |would |draw |a |_________ |- |
ANSWER-GGTP
*if |this |is |normal, |the |source |is |not |due |to |the |liver
Causes |of |high |indirect |bilirubin |are |due |to |the |liver
T/f |- |ANSWER-False. |Indirect |bilirubin |is |unconjugated. |Sources |of |elevated |indirect |bili |are |due |to |
RBC |destruction.
Causes |of |elevated |direct |bilirubin |levels |are |due |to |the |liver
T/f |- |ANSWER-True. |Direct |bilirubin |is |conjugated |by |the |liver |having |been |bound |with |glucoronic |acid
|and |made |water |soluble |for |excretion
In |alcoholics, |you |can |expect |an |AST:ALT |ratio |of |____ |- |ANSWER->2
If |the |AST:ALT |ratio |is |>5, |the |NP |expects |there |are |extra-hepatic |sources |of |AST |
T/f |- |ANSWER-True
In |most |chronic |liver |disease |not |caused |by |ETOH, |which |is |most |elevated?
AST |or |ALT? |- |ANSWER-ALT. |This |is |found |primarily |in |hepatocytes |and |released |when |cells |are |
injured.
Alk |phos |will |be |severely |elevated |in |hepatobiliary |disease |
T/f |- |ANSWER-False. |Although |ALP |will |be |elevated |in |hepatobiliary |disease, |it |is |severely |elevated |in |
biliary |disease |only.
Hepatitis |A |is |spread |via |- |ANSWER-Fecal-oral. |90% |of |children |acquire |hep |a |in |developing |countries
| | | | | |
Esophageal |rupture***, |pancreatic |and |malignant |effusions |are |indicated |when |PF |shows |what? |- |
ANSWER-Amylase
PF |triglyceride |level |>100 |mg/dL |suggests |a |___________ |- |ANSWER-Chylothorax
Hemolysis
Gilbert's |syndrome
Drugs
Hepatitis
Chemotherapy
Neonatal
Obstructive |cholecystokinin |
Cirrhosis |with |liver |failure |
Are |causes |of |what? |- |ANSWER-Hyperbilirubinemia
Alk |Phos |can |be |elevated |with |pregnancy, |bone |disease |or |growing |children |
T/f |- |ANSWER-True
True |test |of |liver |function |is... |- |ANSWER-Total |protein/albumin
PT/INR
AST |is |not |very |specific |for |liver |disease |except |in... |- |ANSWER-ETOH
, To |determine |if |the |source |of |high |Alk |Phos |is |from |the |liver, |you |would |draw |a |_________ |- |
ANSWER-GGTP
*if |this |is |normal, |the |source |is |not |due |to |the |liver
Causes |of |high |indirect |bilirubin |are |due |to |the |liver
T/f |- |ANSWER-False. |Indirect |bilirubin |is |unconjugated. |Sources |of |elevated |indirect |bili |are |due |to |
RBC |destruction.
Causes |of |elevated |direct |bilirubin |levels |are |due |to |the |liver
T/f |- |ANSWER-True. |Direct |bilirubin |is |conjugated |by |the |liver |having |been |bound |with |glucoronic |acid
|and |made |water |soluble |for |excretion
In |alcoholics, |you |can |expect |an |AST:ALT |ratio |of |____ |- |ANSWER->2
If |the |AST:ALT |ratio |is |>5, |the |NP |expects |there |are |extra-hepatic |sources |of |AST |
T/f |- |ANSWER-True
In |most |chronic |liver |disease |not |caused |by |ETOH, |which |is |most |elevated?
AST |or |ALT? |- |ANSWER-ALT. |This |is |found |primarily |in |hepatocytes |and |released |when |cells |are |
injured.
Alk |phos |will |be |severely |elevated |in |hepatobiliary |disease |
T/f |- |ANSWER-False. |Although |ALP |will |be |elevated |in |hepatobiliary |disease, |it |is |severely |elevated |in |
biliary |disease |only.
Hepatitis |A |is |spread |via |- |ANSWER-Fecal-oral. |90% |of |children |acquire |hep |a |in |developing |countries
