507 Final / Review of Key Quizzes, and Practice
Questions for Guaranteed Success
Terms in this set (352)
The posterior lobe of the False.
pituitary produces its own
hormones
The posterior pituitary receives its hormones (oxytocin and
ADH) from the hypothalamus. The anterior pituitary
produces its own hormones.
Aldosterone promotes This statement is true.
sodium reabsorption and
potassium excretion in the
kidneys.
Hyperthyroidism Elevated Thyroid Hormone
Suppressed TSH
Enlarged liver
Hand tremors
Hypothyroidism Decreased thyroid hormone and
Elevated TSH
Fatigue
Diminished deep tendon reflexes
, Onset 1<10-20 years
Associated with diabetic ketoacidosis
Symptoms: polyuria, polyphagia, polydipsia
Autoimmune:Genetic and environmental factors,
Type 1 DM resulting in gradual process of autoimmune
destruction in genetically susceptible individuals
Nonautoimmune:Unknown
Strong association with HLA-DQA and HLA-DQB genes
acute complications: Diabetic ketoacidosis
,The only way for a hormone False.
to work is through the blood
stream.
The endocrine cell can also secrete hormones that target
cells directly close it. When this occurs, this type of
secretion is known as paracrine signaling.
Lipid-derived hormones True.
cross the cell membrane
and bind to receptors in
This statement is true. Lipid-derived hormones cross the cell
the cell's cytoplasm
membrane and bind to receptors in the cell's cytoplasm.
The anterior lobe of the True.
pituitary produces its own
hormones
This statement is true. The anterior lobe of the pituitary
produces its own hormones
To be considered a lipid False.
hormone, it must be bound
to a protein and it must bind
It must be bound to a protein and it bind to receptors on the
to receptors inside the cell
cell membrane rather than inside the cell membrane.
membrane
True.
The pineal gland is
responsible for regulating
the sleep-wake cycle
, Type 2 DM Usually > 40 years of age
Associated with hyperosmolar nonketotic coma
Symptoms: weakness, weight loss, infections
Results from genetic susceptibility (polygenic) combined
with environmental determinants and other risk factors
Inherited defects in beta-cell mass and function
combined with peripheral tissue insulin resistance
Associated with long-duration obesity
strong genetic association
Acute complications: Hyperosmolar nonketotic coma
alpha cells responsible for secreting glucagon
beta cells responsible for secreting insulin and amylin inhibits
glucagon secretion
delta cells responsible for secreting gastrin and somatostatin
secrete pancreatic polypeptide that stimulates gastric
F (PP) Cells
secretions and antagonizes cholecystokinin.