FNP 2 Exam Prep Test Questions with Complete
Solutions| Verified Answers
BP Med Choices w/ Comorbidities - ANSWER Osteoporosis: thiazide
Diabetes: ACE/ARB
CKD: ACE/ARB
CHF: ACE/ARB + BB + diuretic
Post MI/CAD: ACE/ARB + BB
Recurrent stroke: ACE, diuretic
BPH: alpha-1 antagonist
Pregnancy: labetalol (first line), nifedipine, methyldopa
4 categories of HTN in Pregnancy - ANSWER 1. Chronic HTN
2. Gestation HTN
3. Preeclampsia
4. Eclampsia (status epilepticus)
*Collectively, HTN is the most common medical condition in pregnancy
Management of HTN in preggo - ANSWER -Tx if maternal BP is >160/105
-1st line is CCB (per Buttaro) w/ BB or methyldopa 2nd line
-Refer if BP not responsive
-Daily kick counts
-US to asses growth and amniotic fluid q3wks
-BP assess 2x/wk,
-cbc and liver enzyme/creat levels 1x/wk
, -Low dose ASA (75mg/day) for women at high risk of developing preeclampsia as well as
calcium supplementation
Symptoms of Severe Preeclampsia - ANSWER Severe HA not resolved w/ rest, Tylenol,
fluids or food, RUQ px, visual changes, SOB
-These patients should deliver at 34 wks and receive mag sulfate for seizure prevention
RF HTN - ANSWER Genetics, obesity, metabolic syndrome, high dietary fat intake,
excessive sodium ingestion, physical inactivity, OSA, excess ETOH, smoking, stress
**possible: glucose intolerance and low intake mag and postassium
Causes of secondary HTN - ANSWER -Renal Artery Stenosis (RAS):70-80% blockage of a
renal artery that activates the renin-angiotensin system
-Pheochromocytoma: catecholamine-producing tumor of the adrenal glands, 5H's (htn,
ha, hyperhidrosis, hypermatabolic, hyperglycemia), labile htn
-Primary Hyperaldosteronism: unprovoked hypokalemia
-Coarctation of the Aorta: usually youth, claudication, delayed femoral pulses, notching
ribs on cxr
-Cushing Syndrome: hypersecretion glucocorticoid
-Meds: NSAID, SSRI, SNRI, decongestants, illicits
-OSA: hypoxia-driven SNS discharge
-Renal Parenchymal Disease: glomerular dz i.e. SLE
-Genetics: genetic link to secondary causes
Symptoms indicative of target organ damage - ANSWER -Neurovascular (transient
weakness, blindness, visual acuity change, HA, confusion, lethargy, seizure)
Solutions| Verified Answers
BP Med Choices w/ Comorbidities - ANSWER Osteoporosis: thiazide
Diabetes: ACE/ARB
CKD: ACE/ARB
CHF: ACE/ARB + BB + diuretic
Post MI/CAD: ACE/ARB + BB
Recurrent stroke: ACE, diuretic
BPH: alpha-1 antagonist
Pregnancy: labetalol (first line), nifedipine, methyldopa
4 categories of HTN in Pregnancy - ANSWER 1. Chronic HTN
2. Gestation HTN
3. Preeclampsia
4. Eclampsia (status epilepticus)
*Collectively, HTN is the most common medical condition in pregnancy
Management of HTN in preggo - ANSWER -Tx if maternal BP is >160/105
-1st line is CCB (per Buttaro) w/ BB or methyldopa 2nd line
-Refer if BP not responsive
-Daily kick counts
-US to asses growth and amniotic fluid q3wks
-BP assess 2x/wk,
-cbc and liver enzyme/creat levels 1x/wk
, -Low dose ASA (75mg/day) for women at high risk of developing preeclampsia as well as
calcium supplementation
Symptoms of Severe Preeclampsia - ANSWER Severe HA not resolved w/ rest, Tylenol,
fluids or food, RUQ px, visual changes, SOB
-These patients should deliver at 34 wks and receive mag sulfate for seizure prevention
RF HTN - ANSWER Genetics, obesity, metabolic syndrome, high dietary fat intake,
excessive sodium ingestion, physical inactivity, OSA, excess ETOH, smoking, stress
**possible: glucose intolerance and low intake mag and postassium
Causes of secondary HTN - ANSWER -Renal Artery Stenosis (RAS):70-80% blockage of a
renal artery that activates the renin-angiotensin system
-Pheochromocytoma: catecholamine-producing tumor of the adrenal glands, 5H's (htn,
ha, hyperhidrosis, hypermatabolic, hyperglycemia), labile htn
-Primary Hyperaldosteronism: unprovoked hypokalemia
-Coarctation of the Aorta: usually youth, claudication, delayed femoral pulses, notching
ribs on cxr
-Cushing Syndrome: hypersecretion glucocorticoid
-Meds: NSAID, SSRI, SNRI, decongestants, illicits
-OSA: hypoxia-driven SNS discharge
-Renal Parenchymal Disease: glomerular dz i.e. SLE
-Genetics: genetic link to secondary causes
Symptoms indicative of target organ damage - ANSWER -Neurovascular (transient
weakness, blindness, visual acuity change, HA, confusion, lethargy, seizure)
