PCC 4 Exam 1
beta blockers - ✔️✔️what is the second class med for tachydysrhythmias?
potassium channel blockers - ✔️✔️what is the third class med for tachydysrhythmias?
calcium channel blockers - ✔️✔️what is the 4th class med for tachydysrhythmias?
sodium channel blockers - ✔️✔️-class used for tachydysrhythmias
-prolong depolarization time
-slow conduction in atria, ventricles, purkinje fibers
-used for supra ventricular and ventricular tachycardias
beta blockers - ✔️✔️-class used for tachydysrhythmias
-decrease SA node firing
-slow conduction through AV node
-decrease myocardial contractility
-used for supraventricular and ventricular tachycardias
-decreases sympathetic stimulation
depolarization - ✔️✔️inside of cell positive compared to outside
dysrhythmia - ✔️✔️disturbance of heart rhythm
ectopic pacemaker - ✔️✔️pacemaker that bypasses the normal conduction pathway
such as non pacemaker cardiac cells in the atria, AV junction, or ventricles
cardiac output - ✔️✔️a dysrhythmia is a problem when it impacts _________.
48 hours - ✔️✔️how long does someone need to have afib before needing an
anticoagulant?
vagal maneuver, medication, shock - ✔️✔️what are the methods used when HR is too
fast?
vagal maneuver - ✔️✔️method for slowing heart
-patient stable
-hold breath and try to blow it out with mouth and nose closed
-bear down
contractile cells - ✔️✔️working cells responsible for mechanical muscle action of heart
, pacemaker cells - ✔️✔️cells in electrical conduction system responsible for generating
electrical impulses
excitability - ✔️✔️ability to respond to an electrical impulse
conductivity - ✔️✔️ability to conduct an electrical impulse
SA node - ✔️✔️what is the primary pacemaker of the heart?
resting state - ✔️✔️outside of cell positive compared to inside
atropine, escalate meds (epinephrine/dopamine), pacemaker - ✔️✔️what are the
methods used for speeding up the heart?
prodysrhythmic action - ✔️✔️all drugs that treat dysrhythmias can cause dysrhythmias
sodium channel blocker - ✔️✔️what is the first class med for tachydysrhythmias?
potassium channel blockers - ✔️✔️-class used for tachydysrhythmias
-prolong repolarization
-used for supra ventricular and ventricular tachycardias
-possible prolonged QT
-can turn into Torsades de pointes and progress to V fib
calcium channel blocker - ✔️✔️-class used for tachydysrhythmias
-decrease SA node firing
-slow conduction through AV node
-decrease myocardial contractility
-used for supraventricular tachycardias
-only 2
adenosine - ✔️✔️-used for tachydysrhythmias
-decreases automaticity of SA node and slows conduction through AV node
-6mg over 1-3 sec followed by rapid, forceful 20 cc bolus NS
-may repeat with 12mg (repeat to total dose of 30 if no effect)
-supraventricular tachycardia ONLY
digoxin - ✔️✔️-used for tachydysrhythmias
-decreases automaticity of SA node and slows conduction through AV node
-first dose larger, followed by maintenance dose
-can cause cardio toxicity
-used only for supraventricular rhythms
beta blockers - ✔️✔️what is the second class med for tachydysrhythmias?
potassium channel blockers - ✔️✔️what is the third class med for tachydysrhythmias?
calcium channel blockers - ✔️✔️what is the 4th class med for tachydysrhythmias?
sodium channel blockers - ✔️✔️-class used for tachydysrhythmias
-prolong depolarization time
-slow conduction in atria, ventricles, purkinje fibers
-used for supra ventricular and ventricular tachycardias
beta blockers - ✔️✔️-class used for tachydysrhythmias
-decrease SA node firing
-slow conduction through AV node
-decrease myocardial contractility
-used for supraventricular and ventricular tachycardias
-decreases sympathetic stimulation
depolarization - ✔️✔️inside of cell positive compared to outside
dysrhythmia - ✔️✔️disturbance of heart rhythm
ectopic pacemaker - ✔️✔️pacemaker that bypasses the normal conduction pathway
such as non pacemaker cardiac cells in the atria, AV junction, or ventricles
cardiac output - ✔️✔️a dysrhythmia is a problem when it impacts _________.
48 hours - ✔️✔️how long does someone need to have afib before needing an
anticoagulant?
vagal maneuver, medication, shock - ✔️✔️what are the methods used when HR is too
fast?
vagal maneuver - ✔️✔️method for slowing heart
-patient stable
-hold breath and try to blow it out with mouth and nose closed
-bear down
contractile cells - ✔️✔️working cells responsible for mechanical muscle action of heart
, pacemaker cells - ✔️✔️cells in electrical conduction system responsible for generating
electrical impulses
excitability - ✔️✔️ability to respond to an electrical impulse
conductivity - ✔️✔️ability to conduct an electrical impulse
SA node - ✔️✔️what is the primary pacemaker of the heart?
resting state - ✔️✔️outside of cell positive compared to inside
atropine, escalate meds (epinephrine/dopamine), pacemaker - ✔️✔️what are the
methods used for speeding up the heart?
prodysrhythmic action - ✔️✔️all drugs that treat dysrhythmias can cause dysrhythmias
sodium channel blocker - ✔️✔️what is the first class med for tachydysrhythmias?
potassium channel blockers - ✔️✔️-class used for tachydysrhythmias
-prolong repolarization
-used for supra ventricular and ventricular tachycardias
-possible prolonged QT
-can turn into Torsades de pointes and progress to V fib
calcium channel blocker - ✔️✔️-class used for tachydysrhythmias
-decrease SA node firing
-slow conduction through AV node
-decrease myocardial contractility
-used for supraventricular tachycardias
-only 2
adenosine - ✔️✔️-used for tachydysrhythmias
-decreases automaticity of SA node and slows conduction through AV node
-6mg over 1-3 sec followed by rapid, forceful 20 cc bolus NS
-may repeat with 12mg (repeat to total dose of 30 if no effect)
-supraventricular tachycardia ONLY
digoxin - ✔️✔️-used for tachydysrhythmias
-decreases automaticity of SA node and slows conduction through AV node
-first dose larger, followed by maintenance dose
-can cause cardio toxicity
-used only for supraventricular rhythms
