HBS Case Studies Review Practice Test.
ALS (amyotrophic lateral sclerosis) - CORRECT ANSWER A nervous system disease that weakens muscles
and impacts physical function. It affects nerve cells in the brain & spinal cord, while the nerve cells die.
ALS symptoms - CORRECT ANSWER -trouble with walking
-weak muscles
-slurred speaking
-cannot project voice
ALS regions in the brain - CORRECT ANSWER Frontal- motor actions, language, impusle controls, and
initation
Temporal- auditory perception
ALS Prognosis - CORRECT ANSWER -no cure
-muscles and bones weaken rapidly
-difficulty with everyday tasks
-symptoms worsen quickly
ALS error in communication - CORRECT ANSWER Affected motor neurons (which provide voluntary
muscle control and movement)
Huntington's disease - CORRECT ANSWER inherited condition in which nerve cells in the brain break
down over time; rare and incurable
Huntington's disease effect on the brain - CORRECT ANSWER HTT mutation, which causes CAG
trinucleotide repeat, producing abnormally long Huntington protein. This larger protein is cut into small,
toxic fragments that bind and accumulate in neurons (which disrupts the function)
ALS (amyotrophic lateral sclerosis) - CORRECT ANSWER A nervous system disease that weakens muscles
and impacts physical function. It affects nerve cells in the brain & spinal cord, while the nerve cells die.
ALS symptoms - CORRECT ANSWER -trouble with walking
-weak muscles
-slurred speaking
-cannot project voice
ALS regions in the brain - CORRECT ANSWER Frontal- motor actions, language, impusle controls, and
initation
Temporal- auditory perception
ALS Prognosis - CORRECT ANSWER -no cure
-muscles and bones weaken rapidly
-difficulty with everyday tasks
-symptoms worsen quickly
ALS error in communication - CORRECT ANSWER Affected motor neurons (which provide voluntary
muscle control and movement)
Huntington's disease - CORRECT ANSWER inherited condition in which nerve cells in the brain break
down over time; rare and incurable
Huntington's disease effect on the brain - CORRECT ANSWER HTT mutation, which causes CAG
trinucleotide repeat, producing abnormally long Huntington protein. This larger protein is cut into small,
toxic fragments that bind and accumulate in neurons (which disrupts the function)
