Advance Pathophysiology
mccance ch 32 Questions And
Answers
Varicose veins
✓~ pooling of blood, usually in saphenous vein, altered connective tissue proteins
and increased growth of proteolyic enzyme activity and decreased growth of vein
walls.
Chronic venous insufficiency
✓~ inadequate venous return over a long period as a result of of varicose veins,
valves in veins become useless
DVT
✓~ thrombus or clot,
Superior vena cava syndrome
, ✓~ progressive occlusion of the SVC that leads to venous distention in the upper
extremities and and head
isolated systolic hypertension
✓~ elevated systolic pressure accompanied by normal diastolic blood pressure
Primary HTN
✓~ mixture of genetics and environmental factors.
Secondary HTN
✓~ caused by altered characteristics in blood from an underlying disease or drugs
systolic HTN
✓~ most significant factor in causing target organ damage.
HTN
✓~ kidney disease increases risk of MI due to vasoconstriction and ischemia in
renal tubules
mccance ch 32 Questions And
Answers
Varicose veins
✓~ pooling of blood, usually in saphenous vein, altered connective tissue proteins
and increased growth of proteolyic enzyme activity and decreased growth of vein
walls.
Chronic venous insufficiency
✓~ inadequate venous return over a long period as a result of of varicose veins,
valves in veins become useless
DVT
✓~ thrombus or clot,
Superior vena cava syndrome
, ✓~ progressive occlusion of the SVC that leads to venous distention in the upper
extremities and and head
isolated systolic hypertension
✓~ elevated systolic pressure accompanied by normal diastolic blood pressure
Primary HTN
✓~ mixture of genetics and environmental factors.
Secondary HTN
✓~ caused by altered characteristics in blood from an underlying disease or drugs
systolic HTN
✓~ most significant factor in causing target organ damage.
HTN
✓~ kidney disease increases risk of MI due to vasoconstriction and ischemia in
renal tubules
