CCRN REVIEW

SIADH - ANStoo much ADH low Na, hypo-osmolar, low UOP ADH - ANSmade in hypothalamus stored in pituitary released to kidney and makes kidney hold onto h20 serum osmolarity - ANSNa X2 275-295 low=fluid overloaded high=concentrated SIADH CAUSES - ANSoat cell carcinoma (bronchogenic CA)-makes its own ADH Viral PNA Head Problem inc. serum osmo, anesthesia, analgesics, stress COMPLICATIONS OF SIADH - ANSSz's TREATMENT of SIADH - ANSget rid of causes fluid restrictions hypertonic solutions (3%, D5NS, D51/2NS) give 25-50cc/hr d/t CHF DI - ANSNo ADH (no h20 at kidney) inc Na+ levels, inc osmolarity, inc. UOP (spec grav 1.001-1.005) DI Causes - ANShead problems dilantin DI Treatment - ANSGive ADH (PItressin or vasopressin) Give fluids to increase Intravascular volume monitor UOP *monitor fo ischemia Hypoglycemia - ANSCVS s/s tachycardia, palpitations, diaphoresis, irritable, restlessness CNS s/s confusion, lethargy, slurred speech, sz, coma hypoglycemia pathophys - ANSlow glucose-adrenal medulla knows and releases adrenaline-liver releases glycogen which is converted into glucose to increase BGL if block in adrenaline or liver cant convert glycogen into glucose (AKA BETA BLOCKERS) then CVS s/s wont occur DKA s/s - ANSonly in insulin dependent diabetics