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1:58:15
1:58:15AMNSG4330
AMNSG4330
AMNSG4330---L2
L2
L2---ACS(*((&#*!(*#*($$(*($(*$Y(&*&$(*)&*)((&$*^$&*&$
ACS(*((&#*!(*#*($$(*($(*$Y(&*&$(*)&*)((&$*^$&*&$
ACS(*((&#*!(*#*($$(*($(*$Y(&*&$(*)&*)((&$*^$&*&$(&*$
(&*$
(&*$ **F
**F
**FVJyvhjbnoP+_IREOLKDJ(UWRORWUOI
VJyvhjbnoP+_IREOLKDJ(UWRORWUOI
VJyvhjbnoP+_IREOLKDJ(UWRORWUOI
NSG4330 - L2 - ACS
NSG4330 - L2 - ACS
Study online at https://quizlet.com/_fpjeoh
1. What is the epicardium? visceral layer of the heart, coronary arteries sit on top of this
2. what is the myocardi- the muscular tissue of the heart.
um? damage occurs here during an MI
3. what is the endocardi- inner lining of the heart that is continuous with all of the great vessels
um?
4. where is the heart locat- behind the sternum (mediastinum) above the diaphragm
ed?
5. what is the structure of Relatively thin walls, collect blood returning to the heart
atria? less pressure allowing for passive filling
6. what is the structure of ventricles are muscular, and are the mass of the myocardium
ventricles?
7. Where is the tricuspid between right atrium and right ventricle
valve located? Atrioventricular (AV)
8. where is the pulmonic right ventricle and pulmonary artery
valve located?
Semilunar
9. where is the mitral valve left atrium and left ventricle
located? AV
10. where is the aortic valve left ventricle and aorta
located
SL
11. what pneumonic is Tissue Paper My Assets
used to remember
valve locations?
Page
Page
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of
of 1610/13/2026,
1610/13/2026,
1 of 1610/13/2026,
1:58:15
1:58:151:58:15
AMNSG4330
AMNSG4330
AMNSG4330
-- L2
L2 -- ACS(*((&#*!(*#*($$(*($(*$Y(&*&$(*)&*)((&$*^$&*&$
ACS(*((&#*!(*#*($$(*($(*$Y(&*&$(*)&*)((&$*^$&*&$
- L2 - ACS(*((&#*!(*#*($$(*($(*$Y(&*&$(*)&*)((&$*^$&*&$
(&*$
(&*$ **F
**F
(&*$
VJyvhjbnoP+_IREOLKDJ(UWRORWUOI
VJyvhjbnoP+_IREOLKDJ(UWRORWUOI
**F VJyvhjbnoP+_IREOLKDJ(UWRORWUOI
, Page
Page
Page222of
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1610/13/2026,
1610/13/2026,1:58:15
1:58:15
1:58:15AMNSG4330
AMNSG4330
AMNSG4330---L2
L2
L2---ACS(*((&#*!(*#*($$(*($(*$Y(&*&$(*)&*)((&$*^$&*&$
ACS(*((&#*!(*#*($$(*($(*$Y(&*&$(*)&*)((&$*^$&*&$
ACS(*((&#*!(*#*($$(*($(*$Y(&*&$(*)&*)((&$*^$&*&$(&*$
(&*$
(&*$ **F
**F
**FVJyvhjbnoP+_IREOLKDJ(UWRORWUOI
VJyvhjbnoP+_IREOLKDJ(UWRORWUOI
VJyvhjbnoP+_IREOLKDJ(UWRORWUOI
NSG4330 - L2 - ACS
NSG4330 - L2 - ACS
Study online at https://quizlet.com/_fpjeoh
12. When do AV valves open during ventricular diastole (filling), closed during systole to pre-
open? vent back flow (pumping)
13. when do SL valves open during systole (pumping) to go to the body
open?
14. what is coronary artery blockage of arteries/vessels by plaques due to chronic endothelial
disease? injury causing fatty streaks, fibrous plaques that eventually develop
occlusion
15. what is the major cause atherosclerosis
of CAD?
16. what causes chronic en- hypertension, tobacco use, hyperhomocysteinemia, diabetes, infec-
dothelial injury? tions, toxins
17. what medication is statins (ie. rosuvostatin)
used to manage dyslipi-
demia?
18. What is dyslipidemia? abnormal amount of lipids in the blood
more LDL than HDL
19. what is ACS? acute coronary syndrome
20. what are the symptoms - asymptomatic
of CAD? OR - chronic stable angina
21. what is included in ACS? unstable angina, NSTEMI, STEMI
Page
Page
Page222Page
of
of 1610/13/2026,
1610/13/2026,
2 of 1610/13/2026,
1:58:15
1:58:151:58:15
AMNSG4330
AMNSG4330
AMNSG4330
-- L2
L2 -- ACS(*((&#*!(*#*($$(*($(*$Y(&*&$(*)&*)((&$*^$&*&$
ACS(*((&#*!(*#*($$(*($(*$Y(&*&$(*)&*)((&$*^$&*&$
- L2 - ACS(*((&#*!(*#*($$(*($(*$Y(&*&$(*)&*)((&$*^$&*&$
(&*$
(&*$ **F
**F
(&*$
VJyvhjbnoP+_IREOLKDJ(UWRORWUOI
VJyvhjbnoP+_IREOLKDJ(UWRORWUOI
**F VJyvhjbnoP+_IREOLKDJ(UWRORWUOI
, Page
Page
Page333of
of
of1610/13/2026,
1610/13/2026,
1610/13/2026,1:58:15
1:58:15
1:58:15AMNSG4330
AMNSG4330
AMNSG4330---L2
L2
L2---ACS(*((&#*!(*#*($$(*($(*$Y(&*&$(*)&*)((&$*^$&*&$
ACS(*((&#*!(*#*($$(*($(*$Y(&*&$(*)&*)((&$*^$&*&$
ACS(*((&#*!(*#*($$(*($(*$Y(&*&$(*)&*)((&$*^$&*&$(&*$
(&*$
(&*$ **F
**F
**FVJyvhjbnoP+_IREOLKDJ(UWRORWUOI
VJyvhjbnoP+_IREOLKDJ(UWRORWUOI
VJyvhjbnoP+_IREOLKDJ(UWRORWUOI
NSG4330 - L2 - ACS
NSG4330 - L2 - ACS
Study online at https://quizlet.com/_fpjeoh
22. what is collateral circu- The development of new, smaller branches of coronary arteries that
lation? grow in response to a vessel that has slowly become occluded or nearly
occluded over time.
23. what factors contribute - inherited predisposition for angiogenesis
to the growth of collat- - chronic ischemia episodes (CAD or vasospasm) leading to diminished
eral circulation? coronary artery blood flow results in more severe ischemia or infarction
24. what are non-modifi- - increasing age
able risk factors for - being male
CAD? - being black
- genetics
- FamHx heart disease
25. major modifiable risk - alter serum lipids (more LDL, less HDL)
factors for CAD - hypertension
- tobacco use
- physical inactivity
- obesity
26. contributing modifiable diabetes
risk factors for CAD elevated fasting BG
psychosocial risk factors (stress)
elevated homocysteine levels
substance use
27. how can health promo- - identifying high risk people
tion affect CAD? - managing those at high risk
if you identify those who are high risk and alter those modifiable and
contributing risk factors before they become a problem, then you are
able to decrease risk of CAD
Page
Page
Page333Page
of
of 1610/13/2026,
1610/13/2026,
3 of 1610/13/2026,
1:58:15
1:58:151:58:15
AMNSG4330
AMNSG4330
AMNSG4330
-- L2
L2 -- ACS(*((&#*!(*#*($$(*($(*$Y(&*&$(*)&*)((&$*^$&*&$
ACS(*((&#*!(*#*($$(*($(*$Y(&*&$(*)&*)((&$*^$&*&$
- L2 - ACS(*((&#*!(*#*($$(*($(*$Y(&*&$(*)&*)((&$*^$&*&$
(&*$
(&*$ **F
**F
(&*$
VJyvhjbnoP+_IREOLKDJ(UWRORWUOI
VJyvhjbnoP+_IREOLKDJ(UWRORWUOI
**F VJyvhjbnoP+_IREOLKDJ(UWRORWUOI
Page
Page111of
of
of1610/13/2026,
1610/13/2026,
1610/13/2026,1:58:15
1:58:15
1:58:15AMNSG4330
AMNSG4330
AMNSG4330---L2
L2
L2---ACS(*((&#*!(*#*($$(*($(*$Y(&*&$(*)&*)((&$*^$&*&$
ACS(*((&#*!(*#*($$(*($(*$Y(&*&$(*)&*)((&$*^$&*&$
ACS(*((&#*!(*#*($$(*($(*$Y(&*&$(*)&*)((&$*^$&*&$(&*$
(&*$
(&*$ **F
**F
**FVJyvhjbnoP+_IREOLKDJ(UWRORWUOI
VJyvhjbnoP+_IREOLKDJ(UWRORWUOI
VJyvhjbnoP+_IREOLKDJ(UWRORWUOI
NSG4330 - L2 - ACS
NSG4330 - L2 - ACS
Study online at https://quizlet.com/_fpjeoh
1. What is the epicardium? visceral layer of the heart, coronary arteries sit on top of this
2. what is the myocardi- the muscular tissue of the heart.
um? damage occurs here during an MI
3. what is the endocardi- inner lining of the heart that is continuous with all of the great vessels
um?
4. where is the heart locat- behind the sternum (mediastinum) above the diaphragm
ed?
5. what is the structure of Relatively thin walls, collect blood returning to the heart
atria? less pressure allowing for passive filling
6. what is the structure of ventricles are muscular, and are the mass of the myocardium
ventricles?
7. Where is the tricuspid between right atrium and right ventricle
valve located? Atrioventricular (AV)
8. where is the pulmonic right ventricle and pulmonary artery
valve located?
Semilunar
9. where is the mitral valve left atrium and left ventricle
located? AV
10. where is the aortic valve left ventricle and aorta
located
SL
11. what pneumonic is Tissue Paper My Assets
used to remember
valve locations?
Page
Page
Page111Page
of
of 1610/13/2026,
1610/13/2026,
1 of 1610/13/2026,
1:58:15
1:58:151:58:15
AMNSG4330
AMNSG4330
AMNSG4330
-- L2
L2 -- ACS(*((&#*!(*#*($$(*($(*$Y(&*&$(*)&*)((&$*^$&*&$
ACS(*((&#*!(*#*($$(*($(*$Y(&*&$(*)&*)((&$*^$&*&$
- L2 - ACS(*((&#*!(*#*($$(*($(*$Y(&*&$(*)&*)((&$*^$&*&$
(&*$
(&*$ **F
**F
(&*$
VJyvhjbnoP+_IREOLKDJ(UWRORWUOI
VJyvhjbnoP+_IREOLKDJ(UWRORWUOI
**F VJyvhjbnoP+_IREOLKDJ(UWRORWUOI
, Page
Page
Page222of
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of1610/13/2026,
1610/13/2026,
1610/13/2026,1:58:15
1:58:15
1:58:15AMNSG4330
AMNSG4330
AMNSG4330---L2
L2
L2---ACS(*((&#*!(*#*($$(*($(*$Y(&*&$(*)&*)((&$*^$&*&$
ACS(*((&#*!(*#*($$(*($(*$Y(&*&$(*)&*)((&$*^$&*&$
ACS(*((&#*!(*#*($$(*($(*$Y(&*&$(*)&*)((&$*^$&*&$(&*$
(&*$
(&*$ **F
**F
**FVJyvhjbnoP+_IREOLKDJ(UWRORWUOI
VJyvhjbnoP+_IREOLKDJ(UWRORWUOI
VJyvhjbnoP+_IREOLKDJ(UWRORWUOI
NSG4330 - L2 - ACS
NSG4330 - L2 - ACS
Study online at https://quizlet.com/_fpjeoh
12. When do AV valves open during ventricular diastole (filling), closed during systole to pre-
open? vent back flow (pumping)
13. when do SL valves open during systole (pumping) to go to the body
open?
14. what is coronary artery blockage of arteries/vessels by plaques due to chronic endothelial
disease? injury causing fatty streaks, fibrous plaques that eventually develop
occlusion
15. what is the major cause atherosclerosis
of CAD?
16. what causes chronic en- hypertension, tobacco use, hyperhomocysteinemia, diabetes, infec-
dothelial injury? tions, toxins
17. what medication is statins (ie. rosuvostatin)
used to manage dyslipi-
demia?
18. What is dyslipidemia? abnormal amount of lipids in the blood
more LDL than HDL
19. what is ACS? acute coronary syndrome
20. what are the symptoms - asymptomatic
of CAD? OR - chronic stable angina
21. what is included in ACS? unstable angina, NSTEMI, STEMI
Page
Page
Page222Page
of
of 1610/13/2026,
1610/13/2026,
2 of 1610/13/2026,
1:58:15
1:58:151:58:15
AMNSG4330
AMNSG4330
AMNSG4330
-- L2
L2 -- ACS(*((&#*!(*#*($$(*($(*$Y(&*&$(*)&*)((&$*^$&*&$
ACS(*((&#*!(*#*($$(*($(*$Y(&*&$(*)&*)((&$*^$&*&$
- L2 - ACS(*((&#*!(*#*($$(*($(*$Y(&*&$(*)&*)((&$*^$&*&$
(&*$
(&*$ **F
**F
(&*$
VJyvhjbnoP+_IREOLKDJ(UWRORWUOI
VJyvhjbnoP+_IREOLKDJ(UWRORWUOI
**F VJyvhjbnoP+_IREOLKDJ(UWRORWUOI
, Page
Page
Page333of
of
of1610/13/2026,
1610/13/2026,
1610/13/2026,1:58:15
1:58:15
1:58:15AMNSG4330
AMNSG4330
AMNSG4330---L2
L2
L2---ACS(*((&#*!(*#*($$(*($(*$Y(&*&$(*)&*)((&$*^$&*&$
ACS(*((&#*!(*#*($$(*($(*$Y(&*&$(*)&*)((&$*^$&*&$
ACS(*((&#*!(*#*($$(*($(*$Y(&*&$(*)&*)((&$*^$&*&$(&*$
(&*$
(&*$ **F
**F
**FVJyvhjbnoP+_IREOLKDJ(UWRORWUOI
VJyvhjbnoP+_IREOLKDJ(UWRORWUOI
VJyvhjbnoP+_IREOLKDJ(UWRORWUOI
NSG4330 - L2 - ACS
NSG4330 - L2 - ACS
Study online at https://quizlet.com/_fpjeoh
22. what is collateral circu- The development of new, smaller branches of coronary arteries that
lation? grow in response to a vessel that has slowly become occluded or nearly
occluded over time.
23. what factors contribute - inherited predisposition for angiogenesis
to the growth of collat- - chronic ischemia episodes (CAD or vasospasm) leading to diminished
eral circulation? coronary artery blood flow results in more severe ischemia or infarction
24. what are non-modifi- - increasing age
able risk factors for - being male
CAD? - being black
- genetics
- FamHx heart disease
25. major modifiable risk - alter serum lipids (more LDL, less HDL)
factors for CAD - hypertension
- tobacco use
- physical inactivity
- obesity
26. contributing modifiable diabetes
risk factors for CAD elevated fasting BG
psychosocial risk factors (stress)
elevated homocysteine levels
substance use
27. how can health promo- - identifying high risk people
tion affect CAD? - managing those at high risk
if you identify those who are high risk and alter those modifiable and
contributing risk factors before they become a problem, then you are
able to decrease risk of CAD
Page
Page
Page333Page
of
of 1610/13/2026,
1610/13/2026,
3 of 1610/13/2026,
1:58:15
1:58:151:58:15
AMNSG4330
AMNSG4330
AMNSG4330
-- L2
L2 -- ACS(*((&#*!(*#*($$(*($(*$Y(&*&$(*)&*)((&$*^$&*&$
ACS(*((&#*!(*#*($$(*($(*$Y(&*&$(*)&*)((&$*^$&*&$
- L2 - ACS(*((&#*!(*#*($$(*($(*$Y(&*&$(*)&*)((&$*^$&*&$
(&*$
(&*$ **F
**F
(&*$
VJyvhjbnoP+_IREOLKDJ(UWRORWUOI
VJyvhjbnoP+_IREOLKDJ(UWRORWUOI
**F VJyvhjbnoP+_IREOLKDJ(UWRORWUOI