ARDS - nursing Questions with Correct Answers
Question 1:
ARDS - acute respiratory distress syndrome
sudden progressive form of acute respiratorty failure
alveolar capillary interface becomes damaged and more permeable to intravascular fluid alveoli
fill with fluid
hypoxia that persists even when oxygen is administered at 100% decreased pulmonary
compliance
dyspnea
non-cardiac associated bilateral pulmonary edema dense pulmonary infiltrates seen on xray
Answer:
sudden progressive form of acute respiratorty failure
Question 2:
diagnostic assessment of ARDS - lower PaO2 value on ABG poor response to refractory
hypoxemia
ground-glass appearance to chest xray no cardiac involvement on ECG
low to normal PCWP
Answer:
poor response to refractory hypoxemia
Question 3:
Interventions for ARDS - endotracheal intubation and mechanical ventilation with positive
end-expiratory pressure or continuous positive airway pressure
drug therapy nutrition therapy fluid therapy
case management
Answer:
drug therapy
Question 4:
Chest trauma includes pulmonary contusion rib fractureflail chest pneumothoraxtension
pneumothorax hemothorax tracheobronchial trauma
Answer:
pulmonary contusion
, Question 5:
Chest trauma includes pulmonary contusion rib fracture
flail chest pneumothorax
tension pneumothorax hemothorax tracheobronchial trauma
Answer:
pulmonary contusion
Question 6:
S+S of ARDS - severe dyspnea hypoxia
decreased lung compliance diffuse pulmonary infiltrates
Answer:
hypoxia
Question 7:
exudative phase of ARDS - 1-7 days after direct lung injury or host insult
neutrophils adhere to pulmonary microcirculation and damages vascular endothelium
engorgement of peribronchial interstitial space
fluid crosses into alveolar space surfactant dysfunction causes atelactasis
severe V/Q mismath and shunting of pulmonary capillary blood results in refractory hypoxemia
lungs become less compliant
increased WOB, increased RR, decreased tidal volume, decreased CO2 and tissue perfusion
Answer:
neutrophils adhere to pulmonary microcirculation and damages vascular endothelium
Question 8:
proliferative phase of ARDS - 1-2 weeks after initial lung injury lung becomes dense and fibrous
lung compliance continues to decrease hypoxemia worsens
if phase persists- fibrosis results if phase ends- lesions resolve
Answer:
lung becomes dense and fibrousif phase persists- fibrosis results if phase ends- lesions resolve
lung becomes dense and fibrous
if phase persists- fibrosis results if phase ends- lesions resolve
lung becomes dense and fibrous
if phase persists- fibrosis results if phase ends- lesions resolve
lung becomes dense and fibrous
Question 1:
ARDS - acute respiratory distress syndrome
sudden progressive form of acute respiratorty failure
alveolar capillary interface becomes damaged and more permeable to intravascular fluid alveoli
fill with fluid
hypoxia that persists even when oxygen is administered at 100% decreased pulmonary
compliance
dyspnea
non-cardiac associated bilateral pulmonary edema dense pulmonary infiltrates seen on xray
Answer:
sudden progressive form of acute respiratorty failure
Question 2:
diagnostic assessment of ARDS - lower PaO2 value on ABG poor response to refractory
hypoxemia
ground-glass appearance to chest xray no cardiac involvement on ECG
low to normal PCWP
Answer:
poor response to refractory hypoxemia
Question 3:
Interventions for ARDS - endotracheal intubation and mechanical ventilation with positive
end-expiratory pressure or continuous positive airway pressure
drug therapy nutrition therapy fluid therapy
case management
Answer:
drug therapy
Question 4:
Chest trauma includes pulmonary contusion rib fractureflail chest pneumothoraxtension
pneumothorax hemothorax tracheobronchial trauma
Answer:
pulmonary contusion
, Question 5:
Chest trauma includes pulmonary contusion rib fracture
flail chest pneumothorax
tension pneumothorax hemothorax tracheobronchial trauma
Answer:
pulmonary contusion
Question 6:
S+S of ARDS - severe dyspnea hypoxia
decreased lung compliance diffuse pulmonary infiltrates
Answer:
hypoxia
Question 7:
exudative phase of ARDS - 1-7 days after direct lung injury or host insult
neutrophils adhere to pulmonary microcirculation and damages vascular endothelium
engorgement of peribronchial interstitial space
fluid crosses into alveolar space surfactant dysfunction causes atelactasis
severe V/Q mismath and shunting of pulmonary capillary blood results in refractory hypoxemia
lungs become less compliant
increased WOB, increased RR, decreased tidal volume, decreased CO2 and tissue perfusion
Answer:
neutrophils adhere to pulmonary microcirculation and damages vascular endothelium
Question 8:
proliferative phase of ARDS - 1-2 weeks after initial lung injury lung becomes dense and fibrous
lung compliance continues to decrease hypoxemia worsens
if phase persists- fibrosis results if phase ends- lesions resolve
Answer:
lung becomes dense and fibrousif phase persists- fibrosis results if phase ends- lesions resolve
lung becomes dense and fibrous
if phase persists- fibrosis results if phase ends- lesions resolve
lung becomes dense and fibrous
if phase persists- fibrosis results if phase ends- lesions resolve
lung becomes dense and fibrous
