HCR 240 Module 6 Learning Guide| Questions and answers | Updated RATED A+ | NEW EDITION| 100% CORRECT| Arizona College

HCR 240 Module 6 Learning Guide Ii Ii Ii Ii Ii Ii




MODULE REFLECTION – Ii Ii Ii




In the box below, reflect on the course material & activities for this module. If you did not complete all the activities, did you have any questions or comments relating
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to this? What content did you find the most interesting and why? This should be a 4-5 sentence paragraph.
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Most interesting thing I found out about this module was the different types of Diabetes from DI to DM and DM type 1 and type 2. I always knew what diabetes was
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but it was very interesting to learn more in depth about the different types of diabetes. I never knew that there were more than one type.
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MODULE 6 DISORDERS OR DISEASES Ii Ii Ii Ii Ii




Diseases to know for Module 6, include etiology, typical signs and symptoms, diagnostic testing, and general treatment (or prognosis). Use your textbook and/or
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supplemental PowerPoint for the completion of this table. Complete sentences are NOT required. It is important to be succinct in your analysis of each disease and
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choose the most important components to look for in a clinical setting. For this reason, do not include more than 5 different components in each box.
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Signs & Symptoms of Ii Ii Ii




Disease or Disorder Ii Ii Ii Diagnosis / Treatment Ii Ii




Etiology (cause) Ii Ii Pathophysiology Disease (Clinical Ii




(Chapter) (General)
Presentation) / Risk Factors Ii Ii Ii




Posterior Ii Central DI Lack ADH from the ‒ Ii Ii Ii Ii Ii Ii Frequent urination, thirst, Ii Ii Ii ADH administration posterior pituitary• Ii Ii Ii




Ii Nephrogenic dehydration, disorientation, Ii Ii Ii




Diabetes insipidus Ii Ii pituitary Ii DI Kidney fails to respond to ‒
Ii Ii Ii Ii Ii Ii seizures Blood test will show high Ii Ii Ii Ii Ii Ii (if central DI)Ii Ii




(Ch. 24) Ii ADH Distinguish by administering‒
Ii Ii Ii osmolarity and hypernatremia Ii Ii Ii




ADH to see if kidneys can Urine osmolarity and specific gravity
Ii Ii Ii Ii Ii Ii Ii Ii Ii Ii Ii hypopituitarism respond, if so, Ii Ii




Ii central DI Ii will be low Ii Ii




Causes fluid retention Ii Ii Thyroid disorder more common in Ii Ii Ii Ii Fluid restriction• Ii




Syndrome of Ii Ii Excessive ADH Ii •Concentrated urine, dilute Ii Ii Ii women Primary thyroid Ii Ii Slow correction of Ii Ii Ii




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, HCR 240 Module 6 Learning Guide
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Inappropriate Antidiuretic Ii •Causes: brain injury or Ii Ii Ii Ii plasma, hypervolemi Ii disorders most Ii Ii hyponatremia
Hormone (Ch. 24) Ii Ii neurosurgery common Enlarged thyroid can Ii Ii Ii •ADH receptor antagonists
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•Paraneoplastic disorder Ii indicate hypo- or hyperfunction Ii Ii Ii may be used
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Hashimoto’s Ii Primary: high TSH, low free Ii Ii Ii Ii Ii Drugs•Genetics•Thyroiditis Ii Replacement hormone: thyroiditis•Autoimmune Ii Ii Ii T3,
Ii low free T4
Ii Ii (postpartum period especially Ii Ii Ii levothyroxine Ii




Hypothyroidism Ii disorder•Anti-thyroglobulin Ii Secondary: low TSH, low Ii Ii Ii Ii high incidence)•Congenital
Ii Ii Surgical intervention if
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(Ch. 24)Ii antibody and anti- Ii Ii free T3 and T4 Ii Ii Ii hypothyroidism: cretinism Ii necessary
thyroperoxidase antibody Ii Myxedema coma Ii




Elevated free T3 and free Ii Ii Ii Ii Ii Thyroid-stimulating Ii Primary: low TSH, high free T3 Ii Ii Ii Ii Ii Ii Antithyroid hormone Ii Ii




Hyperthyroidism Ii T4 Graves’ disease•Most Ii Ii antibodies•Bind to thyrotropin Ii Ii and T4 Secondary: high TSH,
Ii Ii Ii Ii medication propylthiouracil Ii Ii




(Ch. 24) Ii common cause•Autoimmune receptors •Gland
Ii Ii Ii Ii high free T3 and T Ii Ii Ii Ii (PTU) Radioactive iodine stimulation of the thyroid Ii Ii Ii Ii Ii Ii




enlargement•Continual treatment Surgery Ii




Signs & Symptoms of Ii Ii Ii




Disease or Disorder Ii Ii Ii Diagnosis / Treatment Ii Ii




Etiology (cause) Ii Ii Pathophysiology Disease (Clinical Ii




(Chapter) (General)
Presentation) / Risk Factors Ii Ii Ii




gland synthesis thyroid hormones Ii Ii




Secondary•Decreased With prolonged glucocorticoid Diagnosis•Rapid ACTH test‒ Treatment•Daily replacement Ii Ii Ii Ii Ii Ii Ii Ii




ACTH Primary•AKA: use, CRF-ACTH signals to With ACTH administration, of glucocorticoid and Addison’s adrenal cortex Ii Ii Ii Ii Ii Ii Ii Ii Ii Ii Ii Ii Ii Ii




cortisol should rise within 30 mineralocorticoid•Parenteral disease•Autoimmune suppressed•Adrenal gland
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minutes‒No cortisol rise: steroid coverage in times of Ii Ii Ii Ii Ii Ii Ii Ii




Adrenal Insufficiency destruction adrenal cortex‒ down regulates adrenal cortex insufficiency
Ii Ii major stress, trauma, surgery
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