NSG 5003- Advanced
Pathophysiology-Wk 1 (Ch 1-3) Exam
Questions and Answers 100% PASS
ATP - CORRECT ANSWER-Functions as energy-transferring molecule
Oxidative phosphorylation - CORRECT ANSWER-occurs in mitochondria;
mechanism by which energy is produced from carbs, fats, & proteins
Atrophy - CORRECT ANSWER-decrease in cellular size
Hypertophy - CORRECT ANSWER-increase in cellular size by increased work
demand or hormonal situation
Hyperplasia - CORRECT ANSWER-increase cellular number by increased cellular
division
Dysplasia - CORRECT ANSWER-abnormal change in size, shape, & organization
of mature tissue cells
Metaplasia - CORRECT ANSWER-reversible replacement of one mature cell type
by another less mature cell type
Cell Injury - CORRECT ANSWER-caused by hypoxia
, Free Radicals - CORRECT ANSWER-can cause lipid peroxidation or destruction
of unsaturated fatty acids, alterations of proteins, & alterations in DNA
Apoptosis - CORRECT ANSWER-cell death from programmed, selective self
destruction
Necrosis - CORRECT ANSWER-sum of changes after local cell death & includes
inflammation & cell lysis
PTH - CORRECT ANSWER-parathyroid hormone
Buffers - CORRECT ANSWER-substances that can absorb excess acid/base to
minimize fluctuations in pH
Somatic Death - CORRECT ANSWER-no respirations, no heart beat
Autophagy - CORRECT ANSWER-degradation process where ell debris
encapsulated with in vesicle that reacts with lysosomes
Cellular Metabolism - CORRECT ANSWER-chemical task of maintaining cellular
function
Anabolism - CORRECT ANSWER-energy-using process
Catabolism - CORRECT ANSWER-energy-releasing process
Osmotic Pressure - CORRECT ANSWER-amount of hydrostatic pressure
required to oppose osmotic pressure
© 2026 Copyright. All Rights Reserved. This document is
protected by copyright law
Pathophysiology-Wk 1 (Ch 1-3) Exam
Questions and Answers 100% PASS
ATP - CORRECT ANSWER-Functions as energy-transferring molecule
Oxidative phosphorylation - CORRECT ANSWER-occurs in mitochondria;
mechanism by which energy is produced from carbs, fats, & proteins
Atrophy - CORRECT ANSWER-decrease in cellular size
Hypertophy - CORRECT ANSWER-increase in cellular size by increased work
demand or hormonal situation
Hyperplasia - CORRECT ANSWER-increase cellular number by increased cellular
division
Dysplasia - CORRECT ANSWER-abnormal change in size, shape, & organization
of mature tissue cells
Metaplasia - CORRECT ANSWER-reversible replacement of one mature cell type
by another less mature cell type
Cell Injury - CORRECT ANSWER-caused by hypoxia
, Free Radicals - CORRECT ANSWER-can cause lipid peroxidation or destruction
of unsaturated fatty acids, alterations of proteins, & alterations in DNA
Apoptosis - CORRECT ANSWER-cell death from programmed, selective self
destruction
Necrosis - CORRECT ANSWER-sum of changes after local cell death & includes
inflammation & cell lysis
PTH - CORRECT ANSWER-parathyroid hormone
Buffers - CORRECT ANSWER-substances that can absorb excess acid/base to
minimize fluctuations in pH
Somatic Death - CORRECT ANSWER-no respirations, no heart beat
Autophagy - CORRECT ANSWER-degradation process where ell debris
encapsulated with in vesicle that reacts with lysosomes
Cellular Metabolism - CORRECT ANSWER-chemical task of maintaining cellular
function
Anabolism - CORRECT ANSWER-energy-using process
Catabolism - CORRECT ANSWER-energy-releasing process
Osmotic Pressure - CORRECT ANSWER-amount of hydrostatic pressure
required to oppose osmotic pressure
© 2026 Copyright. All Rights Reserved. This document is
protected by copyright law
