MSN 611 Midterm Practice Questions With Correct Answers

MSN 611 Midterm Practice Questions With Correct
Answers


Eosinophils, |neutrophils, |and |T |cells |contain |the |type |of |histamine |receptors |referred |to |as:



a. |H1

b. |H2

c. |H3

d. |H4 |- |CORRECT |ANSWER✔✔-d. |H4



Some |1st |generation |antihistamines |are |used |to |treat



a. |as |stimulants

b. |to |prevent |motion |sickness

c. |as |anticonvulsants

d. |as |laxatives |- |CORRECT |ANSWER✔✔-b. |to |prevent |motion |sickness



2nd |generation |antihistamines |are |preferred |for |treating |which |patient |category?



a. |children

b. |women

c. |patients |with |hypertension |

d. |patients |with |insomnia |- |CORRECT |ANSWER✔✔-a. |children



Compared |to |1st |generation |antihistamines, |2nd |generation |antihistamines:

,a. |are |less |able |to |penetrate |the |blood |brain |barrier

b. |have |fewer |muscarinic |effects

c. |have |greater |anticholinergic |effects

d. |are |more |likely |to |cause |drowsiness |- |CORRECT |ANSWER✔✔-a. |are |less |able |to |penetrate |
the |blood |brain |barrier



Which |types |of |antihistamines |are |used |to |inhibit |the |secretion |of |gastric |acid |in |patients |with |
gastrointestinal |disorders?



a. |H1

b. |H2

c. |H3

d. |H4 |- |CORRECT |ANSWER✔✔-b. |H2



Under |what |two |circumstances |may |2nd |generation |antihistamines |cause |drowsiness?



a. |if |used |concurrently |with |medications |that |cause |CNS |depression |or |with |alcohol

b. |if |used |concurrently |with |medications |that |cause |CNS |depression

c. |if |they |are |taken |with |OTC |medications

d. |if |they |are |taken |with |alcohol |- |CORRECT |ANSWER✔✔-a. |if |used |concurrently |with |
medications |that |cause |CNS |depression |or |with |alcohol



Why |is |it |important |for |antiviral |compounds |to |be |able |to |discriminate |between |host |and |viral |
functions |with |a |high |degree |of |specificity?



a. |available |antiviral |agents |mainly |target |stages |in |the |viral |life |cycle

,b. |to |avoid |attacking |host |cells, |since |viruses |replicate |intracellularly |and |often |use |host |cell |
enzymes, |macromolecules, |and |organelles |to |make |their |own |particles |

c. |in |recent |years |the |demand |for |new |antiviral |strategies |has |increased |marketably |- |CORRECT
|ANSWER✔✔-b. |to |avoid |attacking |host |cells, |since |viruses |replicate |intracellularly |and |often |


use |host |cell |enzymes, |macromolecules, |and |organelles |to |make |their |own |particles



Which |of |the |following |statements |pertains |to |antimicrobial |therapy?



a. |lower |dosage |levels |of |antimicrobial |therapy |can |help |prevent |allergic |responses

b. |a |differential |diagnosis |should |indicate |whether |an |infection |is |self-limiting |

c. |self-limiting |infections |often |require |antimicrobial |therapy

d. |in |cases |of |sepsis, |laboratory |specimens |are |not |required |- |CORRECT |ANSWER✔✔-b. |a |
differential |diagnosis |should |indicate |whether |an |infection |is |self-limiting



Chose |the |correct |statement |describing |antibacterial |drugs:



a. |antibiotics |are |developed |to |have |similar |required |exposure |times |across |drug |classes

b. |Penicillinase-resistant |penicillins |are |the |agents |of |choice |for |most |staphylococcal |disease

c. |narrow |therapeutic |agents |have |a |higher |safety |tolerance |for |large |fluctuations |of |drug |in |
the |blood

d. |therapeutic |levels |of |antibacterial |agents |are |established |during |tissue |culture |studies |- |
CORRECT |ANSWER✔✔-b. |Penicillinase-resistant |penicillins |are |the |agents |of |choice |for |most |
staphylococcal |disease



The |available |modes |of |action |of |antihelmintic |drugs |is/are:



a. |excrete |in |the |intestine

b. |locally |within |the |gut |lumen |to |cause |expulsion |of |worms |from |the |GI |tract |and |systemically
|against |helminths |residing |outside |the |GI |tract

, c. |is |likely |present |in |virtually |every |mammal |residing |in |a |"natural" |habitat

d. |systemically |against |helminths |residing |outside |the |GI |tract |- |CORRECT |ANSWER✔✔-b. |
locally |within |the |gut |lumen |to |cause |expulsion |of |worms |from |the |GI |tract |and |systemically |
against |helminths |residing |outside |the |GI |tract



The |desired |response |to |antimicrobial |therapy |within |the |first |24 |to |48 |hours |includes:



a. |increased |thirst

b. |increased |sputum |production

c. |white |blood |cell |count |begins |to |normalize

d. |spike |in |temperature |- |CORRECT |ANSWER✔✔-c. |white |blood |cell |count |begins |to |normalize



Most |systemic |fungal |infections |are |acquired |via:



a. |oral-fecal |transmission

b. |inhalation

c. |direct |contact |

d. |nosocomial |transmission |- |CORRECT |ANSWER✔✔-b. |inhalation



The |ability |of |an |agent |to |continually |suppress |growth |of |an |organism |after |concentrations |
have |decreased |to |below |the |minimal |inhibitory |concentration |is |known |as:



a. |probiotic |effect

b. |postantibiotic |effect

c. |alternative |effect

d. |residual |effect |- |CORRECT |ANSWER✔✔-b. |postantibiotic |effect