TEST BANK FOR RUBIN’S PATHOLOGY CLINICOPATHOLOGIC FOUNDATIONS OF MEDICINE 7TH EDITION ( WITH ANSWER KEY) WITH ALL CHAPTERS 100% COMPLETE

TEST BANK
D

,ChapterD1:DCellDAdaptation,DInjuryDandDDeath
IschemiaDandDotherDtoxicDinjuriesDincreaseDtheDaccumulationDofDintracellularDcalciumDasDaDresult
1. Dof:



A) releaseDofDstoredDcalciumDfromDtheDmitochondria.
B) improvedDintracellularDvolumeDregulation.
C) decreasedDinfluxDacrossDtheDcellDmembrane.
D) attractionDofDcalciumDtoDfattyDinfiltrates.
TheDpatientDisDfoundDtoDhaveDliverDdisease,DresultingDinDtheDremovalDofDaDlobeDofDhisDliver.DA
2. daptationDtoDtheDreducedDsizeDofDtheDliverDleadsDtoD
ofDtheDremainingDliverDcells
.
A) metaplasia
B) organDatrophy
C) compensatoryDhyperplasia
D D D) physiologicDhypertrophy
ADpersonDeatingDpeanutsDstartsDchokingDandDcollapses.DHisDairwayDobstructionDisDpartiall
yDcleared,DbutDheDremainsDhypoxicDuntilDheDreachesDtheDhospital.DTheDprolongedDcellDhyp
3. oxiaDcausedDaDcerebralDinfarctionDandDresultingD inDtheDbrain.
A) caspaseDactivation
B) coagulationDnecrosis
C) rapidDphagocytosis
D) proteinDp53Ddeficiency
BacteriaDandDvirusesDcauseDcellDdamageDbyD
4. ,DwhichDisDuniqueDfromDtheDintracellula
rDdamageDcausedDbyDotherDinjuriousDagents.
A) disruptingDtheDsodium/potassiumDATPaseDpump
B) interruptingDoxidativeDmetabolismDprocesses
D D C) replicatingDandDproducingDcontinuedDinjury
D) decreasingDproteinDsynthesisDandDfunction
TheDpatientDhasDaDprolongedDinterruptionDinDarterialDbloodDflowDtoDhisDleftDkidney,DcausingDhypo
5. xicDcellDinjuryDandDtheDreleaseDofDfreeDradicals.DFreeDradicalsDdamageDcellsDby:
A) destroyingDphospholipidsDinDtheDcellDmembrane.
B) alteringDtheDimmuneDresponseDofDtheDcell.
C) disruptingDcalciumDstorageDinDtheDcell.
D) inactivationDofDenzymesDandDmitochondria.

, 6. InjuredDcellsDhaveDimpairedDflowDofDsubstancesDthroughDtheDcellDmembraneDasDaDresultDof:
A) increasedDfatDload.
B) alteredDpermeability.
C) alteredDglucoseDutilization.
D) increasedDsurfaceDreceptors.
7. ReversibleDadaptiveDintracellularDresponsesDareDinitiatedDby:
A) stimulusDoverload.
B) geneticDmutations.
C) chemicalDmessengers.
D) mitochondrialDDNA.
8. InjuredDcellsDbecomeDveryDswollenDasDaDresultDof:
A) increasedDcellDproteinDsynthesis.
B) alteredDcellDvolumeDregulation.
C) passiveDentryDofDpotassiumDintoDtheDcell.
D) blebDformationDinDtheDplasmaDmembrane.
ADdiabeticDpatientDhasDimpairedDsensation,Dcirculation,DandDoxygenationDofDhisDfeet.DHeDstepsDo
nDaDpieceDofDglass,DtheDwoundDdoesDnotDheal,DandDtheDareaDtissueDbecomesDnecrotic.DTheDnecro
9. ticDcellDdeathDisDcharacterizedDby:
A) rapidDapoptosis.
B) cellularDrupture.
C) shrinkageDandDcollapse.
D) chronicDinflammation.
AD99-year-
10. oldDwomanDhasDexperiencedDtheDdeclineDofDcellDfunctionDassociatedDwithDage.DADgroupDofDthe
oriesDofDcellularDagingDfocusDonDprogrammed:
A) changesDwithDgeneticDinfluences.
B) eliminationDofDcellDreceptorDsites.
C) insufficientDtelomeraseDenzyme.
D D D) DNADmutationDorDfaultyDrepair.
AnD89-year-
oldDfemaleDpatientDhasDexperiencedDsignificantDdecreasesDinDherDmobilityDandDstaminaDduring
DaD3-weekDhospitalDstayDforDtheDtreatmentDofDaDfemoralDheadDfracture.DWhichDof

11. theDfollowingDphenomenaDmostDlikelyDaccountsDforDtheDpatientsDdecreaseDinDmuscleDfunctionDt
hatDunderliesDherDreducedDmobility?
A) ImpairedDmuscleDcellDmetabolismDresultingDfromDmetaplasia

, B) DysplasiaDasDaDconsequenceDofDinflammationDduringDboneDremodeling
C) DisuseDatrophyDofDmuscleDcellsDduringDaDprolongedDperiodDofDimmobility
D) IschemicDatrophyDresultingDfromDvascularDchangesDwhileDonDbedrest
AD20-year-
oldDcollegeDstudentDhasDpresentedDtoDherDcampusDmedicalDclinicDforDaDscheduledDPapanicolaouD
12. (Pap)Dsmear.DTheDclinicianDwhoDwillDinterpretDtheDsmearDwillDexamineDcellDsamplesDforDeviden
ceDof:
A) changesDinDcellDshape,Dsize,DandDorganization.
B) theDpresenceDofDunexpectedDcellDtypes.
D D C) ischemicDchangesDinDcellDsamples.
D) abnormallyDhighDnumbersDofDcellsDinDaDspecifiedDfield.
WhichDofDtheDfollowingDpathophysiologicDprocessesDisDmostDlikelyDtoDresultDinDmetastaticDcalcif
D D 13. ication?
A) BenignDprostaticDhyperplasia
B) LiverDcirrhosis
C) ImpairedDglycogenDmetabolism
D) Hyperparathyroidism
DespiteDtheDlowDlevelsDofDradiationDusedDinDcontemporaryDradiologicDimaging,DaDradiologyDtec
hnicianDisDawareDofDtheDneedDtoDminimizeDherDexposureDtoDionizingDradiation.DWhatDisDtheDprim
14. aryDrationaleDforDtheDtechniciansDprecautions?
A) RadiationDstimulatesDpathologicDcellDhypertrophyDandDhyperplasia.
B) RadiationDresultsDinDtheDaccumulationDofDendogenousDwasteDproductsDinDtheDcytoplasm.
C) RadiationDinterferesDwithDDNADsynthesisDandDmitosis.
D) RadiationDdecreasesDtheDactionDpotentialDofDrapidlyDdividingDcells.
TheDparentsDofDaD4-year-
oldDgirlDhaveDsoughtDcareDbecauseDtheirDdaughterDhasDadmittedDtoDchewingDandDswallowingDim
15. portedDtoyDfigurinesDthatDhaveDbeenDdeterminedDtoDbeDmadeDofDlead.DWhichDofDtheDfollowingDbl
oodDtestsDshouldDtheDcareDteamDprioritize?
D D A) WhiteDbloodDcellDlevelsDwithDdifferential
B) RedDbloodDcellDlevelsDandDmorphology
C) UreaDandDcreatinineDlevels
D) LiverDfunctionDpanel
AD70-year-oldDmaleDpatientDhasDbeenDadmittedDtoDaDhospitalDforDtheDtreatmentDofDaDrecent
hemorrhagicDstrokeDthatDhasDleftDhimDwithDnumerousDmotorDandDsensoryDdeficits.DTheseDdeficit
16. sDareDmostDlikelyDtheDresultDofDwhichDofDtheDfollowingDmechanismsDofDcellDinjury?
A) FreeDradicalDinjury