NUR2392 MULTIDIMENSIONAL CARE II
EXAM QUESTIONS AND DETAILED
ANSWERS. EXPERT VERIFIED FOR
GUARANTEED PASS.
Uncompensated - ANS pH and one other value is abnormal
Partial compensation - ANS pH, CO2, and HCO3 are all off
Full compensation - ANS pH is normal
Acidosis - ANS reduces the excitability of cardiovascular muscle, neurons, skeletal muscle,
and smooth muscle.
Alkalosis - ANS increases the sensitivity of excitable tissues allowing them to OVERRESPOND
without stimulation
ABG considerations - ANS - Assess cardiovascular w/ acidosis (cardiac arrest from
hyperkalemia)
- Assess neuro status
- Fall precautions
1 @COPYRIGHT 2025/2026 ALLRIGHTS RESERVED
, Bicarb HC03 - ANS kidney compensation (slow and powerful)
Respiratory PaC02 - ANS Respiratory compensation (fast but limited)
Metabolic acidosis causes - ANS DKA, Starvation, diarrhea, kidney failure, dehydration, liver
failure, pancreatitis, heavy exercise, seizure activity, fever, hypoxia, ischemia,
ethanol/methanol intoxication
Metabolic acidosis signs and symptoms - ANS bradycardia, hypotension, thready pulse, CNS
depression, hyporeflexia, kussmal resp (with resp compensation), warm, flushed, dry skin.
Metabolic acidosis treatment - ANS hydration and medication to treat underlying problems
(DKA - give insulin).
Metabolic alkalosis causes - ANS antacids, blood transfusion, sodium bicarbonate, total
parenteral nutrition (TPN), prolonged vomiting, nasogastric suctioning, hypercortisolism,
hyperaldosteronism, Loop/Thiazide diuretics.
Metabolic Alkalosis s/s - ANS anxiety, irritability, tetany, seizures, POSITIVE CHVOSTEK,
POSITIVE TROUSSEAU, parathesis, hyperreflexia, muscle cramping/twitching, skeletal muscle
weakness, Tachycardia, norm/low BP, increased Digoxin toxicity, decreased respiratory effort
(muscle weakness).
metabolic alkalosis treatment - ANS restore fluid/electrolyte imbalances
Respiratory acidosis cause - ANS Opioids, anesthetics, electrolyte imbalance, inadequate
chest expansion, muscle weakness, airway obstruction, alveolar-capillary block.
2 @COPYRIGHT 2025/2026 ALLRIGHTS RESERVED
EXAM QUESTIONS AND DETAILED
ANSWERS. EXPERT VERIFIED FOR
GUARANTEED PASS.
Uncompensated - ANS pH and one other value is abnormal
Partial compensation - ANS pH, CO2, and HCO3 are all off
Full compensation - ANS pH is normal
Acidosis - ANS reduces the excitability of cardiovascular muscle, neurons, skeletal muscle,
and smooth muscle.
Alkalosis - ANS increases the sensitivity of excitable tissues allowing them to OVERRESPOND
without stimulation
ABG considerations - ANS - Assess cardiovascular w/ acidosis (cardiac arrest from
hyperkalemia)
- Assess neuro status
- Fall precautions
1 @COPYRIGHT 2025/2026 ALLRIGHTS RESERVED
, Bicarb HC03 - ANS kidney compensation (slow and powerful)
Respiratory PaC02 - ANS Respiratory compensation (fast but limited)
Metabolic acidosis causes - ANS DKA, Starvation, diarrhea, kidney failure, dehydration, liver
failure, pancreatitis, heavy exercise, seizure activity, fever, hypoxia, ischemia,
ethanol/methanol intoxication
Metabolic acidosis signs and symptoms - ANS bradycardia, hypotension, thready pulse, CNS
depression, hyporeflexia, kussmal resp (with resp compensation), warm, flushed, dry skin.
Metabolic acidosis treatment - ANS hydration and medication to treat underlying problems
(DKA - give insulin).
Metabolic alkalosis causes - ANS antacids, blood transfusion, sodium bicarbonate, total
parenteral nutrition (TPN), prolonged vomiting, nasogastric suctioning, hypercortisolism,
hyperaldosteronism, Loop/Thiazide diuretics.
Metabolic Alkalosis s/s - ANS anxiety, irritability, tetany, seizures, POSITIVE CHVOSTEK,
POSITIVE TROUSSEAU, parathesis, hyperreflexia, muscle cramping/twitching, skeletal muscle
weakness, Tachycardia, norm/low BP, increased Digoxin toxicity, decreased respiratory effort
(muscle weakness).
metabolic alkalosis treatment - ANS restore fluid/electrolyte imbalances
Respiratory acidosis cause - ANS Opioids, anesthetics, electrolyte imbalance, inadequate
chest expansion, muscle weakness, airway obstruction, alveolar-capillary block.
2 @COPYRIGHT 2025/2026 ALLRIGHTS RESERVED