Test bank for Rubin s pathology clinicopathologic foundations of medicine 7th edition by David S. Strayer, Emanuel Rubin Course

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RUBIN'SPATHOLOGY:CLINICOPATHOLOGICFOUNDATIONSOF MEDICINE
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7TH EDITION
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DAVIDS.STRAYER,EMANUELRUBIN
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,Test Bank Rubin's Pathology: Clinicopathologic Foundations of Medicine 7th Edition
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Table of Contents:
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Chapter1: Cell Adaptation, Injury and Death
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Chapter2: Inflammation
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Chapter3: Repair, Regeneration and Fibrosis
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Chapter4: Immunopathology
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Chapter 5: Neoplasia
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Chapter 6: Developmental and Genetic Diseases
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Chapter 7: Hemodynamic Disorders
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Chapter 8: Environmental and Nutritional Pathology
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Chapter9: Infectious and Parasitic Diseases
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Section II: Pathogenesis of Systemic Conditions Expandable section
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Chapter 10: Aging
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Chapter 11: Systemic Autoimmune Diseases
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Chapter 12: Sepsis
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Chapter 13: Obesity and Diabetes Mellitus
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Chapter 14: The Pathology of Pregnancy
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Chapter 15: The Amyloidoses
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Section III: Diseases of Individual Organ SystemsExpandable section
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Chapter 16: Blood Vessels
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Chapter 17: The Heart
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Chapter 18: The Respiratory System
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Chapter 19: The Gastrointestinal Tract
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Chapter 20: The Liver and Biliary System
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Chapter 21: The Pancreas
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Chapter 22: The Kidney
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Chapter 23: The Lower Urinary Tract and Male Reproductive System
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Chapter 24: The Female Reproductive System and Peritoneum
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Chapter 25: The Breast
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Chapter 26: Hematopathology
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Chapter 27: The Endocrine System
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Chapter 28: The Skin
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Chapter 29: The Head and Neck
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Chapter 30: Bones, Joints and Soft Tissue
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Chapter31: Skeletal Muscle and Peripheral Nervous System
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Chapter32: The Central Nervous System
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Chapter 33: The Eye
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Chapter 34: Forensic Pathology
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,Rubin's Pathology: Clinicopathologic Foundations of
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MedicineChapter1:Cell Adaptation, Injuryand Death
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Ischemiaand other toxic injuries increase theaccumulation ofintracellular calcium as aresult
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1. of:
A) release ofstored calcium from the mitochondria. l l l l l l




B) improved intracellular volumeregulation. l l l




C) decreased influx acrossthecell membrane. l l l l l




D) attraction of calcium tofatty infiltrates. l l l l l




The patient is found to have liver disease, resulting in the removal of a lobe of his liver.
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2. Adaptation tothe reduced size ofthe liver leads to_ l ofthe remaining liver cells.
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A) metaplasia
B) organ atrophy l




C) compensatoryhyperplasia l




D) physiologic hypertrophy l




A person eating peanuts starts choking and collapses. His airway obstruction is partially cleared, but
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he remains hypoxic until hereaches the hospital. The prolonged cell hypoxia
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3. caused acerebral infarction and resulting
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A) caspaseactivation l




B) coagulation necrosis l




C) rapidphagocytosis l




D) protein p53 deficiency l l




Bacteriaand viruses cause cell damageby
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4. damage caused by other injurious agents.
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A) disruptingthe sodium/potassium ATPase pump l l l l




B) interrupting oxidativemetabolism processes l l l




C) replicating and producing continued injury l l l l




D) decreasing protein synthesis andfunction l l l l




Thepatient has aprolonged interruption inarterial blood flowto his left kidney, causing
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5. hypoxiccell injuryand therelease of freeradicals. Free radicals damage cells by:
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A) destroying phospholipids inthecell membrane. l l l l l




B) alteringthe immune response of the cell. l l l l l l




C) disrupting calcium storage in thecell. l l l l l




D) inactivation ofenzymes and mitochondria. l l l l

, 6. Injured cells have impaired flow ofsubstances throughthecell membrane as aresult of:
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A) increased fat load. l l




B) alteredpermeability. l




C) altered glucoseutilization. l l




D) increased surfacereceptors. l l




7. Reversible adaptive intracellular responses areinitiated by: l l l l l l




A) stimulus overload. l




B) geneticmutations. l




C) chemicalmessengers. l




D) mitochondrialDNA. l




8. Injured cells become veryswollen as a result of:
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A) increased cell proteinsynthesis. l l l




B) altered cell volumeregulation. l l l




C) passive entry ofpotassium into the cell. l l l l l l




D) bleb formation in theplasmamembrane.
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Adiabeticpatient has impaired sensation, circulation, and oxygenation of his feet. Hesteps on a piece
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of glass, the wound does not heal, and the area tissue becomes necrotic. The necrotic
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9. celldeath is characterized by:
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A) rapidapoptosis. l




B) cellularrupture. l




C) shrinkageand collapse. l l




D) chronicinflammation. l




A 99-year-old woman has experienced the decline of cell function associated with age. A
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10. group oftheories ofcellular aging focus on programmed:
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A) changes withgenetic influences. l l l




B) elimination ofcell receptor sites. l l l l




C) insufficient telomeraseenzyme. l l




D) DNAmutation or faultyrepair. l l l l




An 89-year-old female patient has experienced significant decreases in her mobility and stamina
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during a3-week hospital stay forthetreatment of a femoral head fracture. Which of the following
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phenomena most likely accounts for the patients decrease in muscle function
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11. thatunderlies her reduced mobility?
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A) Impaired muscle cell metabolism resulting from metaplasia l l l l l l




B) Dysplasia as a consequence of inflammation during boneremodeling l l l l l l l l




C) Disuseatrophy ofmuscle cells during aprolonged period of immobility
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